metadata
language:
- en
license: cc-by-4.0
library_name: span-marker
tags:
- span-marker
- token-classification
- ner
- named-entity-recognition
- generated_from_span_marker_trainer
datasets:
- EMBO/SourceData
metrics:
- precision
- recall
- f1
widget:
- text: >-
Comparison of ENCC-derived neurospheres treated with intestinal extract
from hypoganglionosis rats, hypoganglionosis treated with Fecal microbiota
transplantation (FMT) sham rat. Comparison of neuronal markers. (J)
Immunofluorescence stain number of PGP9.5+. Nuclei were stained blue with
DAPI; Triangles indicate PGP9.5+.
- text: >-
Histochemical (H & E) immunostaining (red) show T (CD3+) neutrophil
(Ly6b+) infiltration in skin of mice in (A). Scale bar, 100 μm. (of CD3
Ly6b immunostaining from CsA treated mice represent seperate analyses
performed on serial thin sections.) of epidermal thickness, T (CD3+)
neutrophil (Ly6b+) infiltration (red) in skin thin sections from (C), (n =
6). Data information: Data represent mean ± SD. * P < 0.05, * * P < 0.01
by two -Mann-Whitney; two independent experiments.
- text: >-
C African green monkey kidney epithelial (Vero) were transfected with NC,
siMLKL, or miR-324-5p for 48 h. qPCR for expression of MLKL. Data
information: data are represented as means ± SD of three biological
replicates. Statistical analyses were performed using unpaired Student ' s
t -. experiments were performed at least three times, representative data
are shown.
- text: >-
(F) Binding between FTCD p47 between p47 p97 is necessary for mitochondria
aggregation mediated by FTCDwt-HA-MAO. HeLa Tet-off inducibly expressing
FTCDwt-HA-MAO were transfected with mammalian expression constructs of
siRNA-insensitive Flag-tagged p47wt / mutants at same time as treatment of
p47 siRNA, cultured for 24 hrs. were further cultured in DOX-free medium
for 48 hrs for induction of FTCD-HA-MAO. After fixation, were visualized
with a monoclonal antibody to mitochondria polyclonal antibodies to HA
Flag. Panels a-l display representative. Scale bar = 10 μm. (G) Binding
between FTCD p97 is necessary for mitochondria aggregation mediated by
FTCDwt-HA-MAO. HeLa Tet-off inducibly expressing FTCDwt-HA-MAO were
transfected with mammalian expression construct of siRNA-insensitive
Flag-tagged p97wt / mutant at same time as treatment with p97 siRNA.
following procedures were same as in (F). Panels a-i display
representative. Scale bar = 10 μm. (H) results of of (F) (G). Results are
shown as mean ± SD of five sets of independent experiments, with 100
counted in each group in each independent experiment. Asterisks indicate a
significant difference at P < 0.01 compared with siRNA treatment alone
('none') compared with mutant expression (Bonferroni method).
- text: >-
(b) Parkin is recruited selectively to depolarized mitochondria directs
mitophagy. HeLa transfected with HA-Parkin were treated with CCCP for
indicated times. Mitochondria were stained by anti-TOM20 (pseudo coloured;
blue) a ΔΨm dependent MitoTracker (red). Parkin was stained with anti-HA
(green). Without treatment, mitochondria are intact stained by both
mitochondrial markers, whereas Parkin is equally distributed in cytoplasm.
After 2 h of CCCP treatment, mitochondria are depolarized as shown by loss
of MitoTracker. Parkin completely translocates to mitochondria clustering
at perinuclear regions. After 24h of CCCP treatment, massive loss of
mitochondria is observed as shown by disappearance of mitochondrial
marker. Only Parkin-positive show mitochondrial clustering clearance, in
contrast to adjacent untransfected. Scale bars, 10 μm.
pipeline_tag: token-classification
base_model: bert-base-uncased
model-index:
- name: SpanMarker with bert-base-uncased on SourceData
results:
- task:
type: token-classification
name: Named Entity Recognition
dataset:
name: SourceData
type: EMBO/SourceData
split: test
metrics:
- type: f1
value: 0.8336481983993405
name: F1
- type: precision
value: 0.8345368269032392
name: Precision
- type: recall
value: 0.8327614603348888
name: Recall
You can finetune this model on your own dataset.
