What is atheromatous aorta?
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Basically, it's plaque buildup in your aorta.  Atheromas can cause embolisms, stroke, etc.
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Vena cava and Aorta are parallel near to the heart but, they change courses soon.
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As you have mentioned in your question, different sources will show different ranges for hypertension. However, almost all of the studies and medical text books, agree on that the blood pressure above 140 mmHG systolic and 90 mmHg diastolic is a risk factor for atherosclerotic diseases.
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cardiovascular system
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Blood pressure is the pressure of the blood against the walls of the arteries.  It results from two forces, one is created when the heart pumps blood into the arteries and through the circulatory system.  The other is the arteries resisting the flow of blood.\nThe higher (systolic) represents the heart contracting and the lower (diastolic)is the heart relaxing between beats.  A blood pressure of 120/80 is considering optimal for adults over the age of 18.  A blood pressure of 140 over 90 is considered hypertension.  Yours of 150/70 is considered mild high and many factors affect blood pressure, one of which is weight.  Others are stress, salt intake, exercise, race, age and alcohol consumption among others.  150/70 is not terribly high but then again the higher the number the greater the risk to good health.  Not being a physician, my advice would be assess your lifestyle and have it checked again.  Don't stress yourself out before visiting your clinic and ask questions that concern you, they are the best resource since they have your health history.  You can also visit www.americanheart.org, which is considered one of the top online resources for a healthy heart.
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A healthy diet is important to keep atherosclerosis from getting worse.\n\nA program of daily walking for short periods, and stopping for pain or cramping, may help improve function. You MUST stop smoking.\n\nAvoid placing hot or cold items on legs. Avoid tight shoes.
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To the aorta and then out to the rest of the body.
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Bas - The carotid pulse is visible is most patients, although it does vary depending on the patient's body habitus.  Carotid pulses in obese patients may be very difficult to see, whereas they can be quite obvious in very thin patients.  However, there are also cases where the carotid pulsations can be more pronounced than normal such as in severe aortic valve regurgitation.  If you have any concerns about this, you should consult with a physician who can do a complete physical exam and tell you if there is anything you need to be worried about.
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Clogged arteries which feed the heart muscle.  Usually caused by poor diet and sedentary life style.
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An aneurysm is a abnormal localized sac or a irreversible dilation caused by weakness (decreased elastin) of arterial wall. Aneurysms are classified  as either fusiform or saccular. In a saccular aneurysm  one side of the artery wall is outputted. \nIn the fusiform the entire circumference of the artery is outputted. The abdominal aorta is the most common site for an aneurysm to develop. The exact cause of abdominal aortic aneurysms (AAA) is unknown. However, risks associated with AAA include atherosclerosis , hypertension and smoking. Other risks include trauma to the arterial wall, infection and congenital defects of the artery wall. Most AAA occur below the level of the renal artery and involve the bifurcation of the aorta as well as the proximal ends of the iliac arteries. Stasis of blood can lead to thrombus formation along arterial wall. Peripheral emboli can develop causing arterial insufficiency. Once an aneurysm forms it often increase in size and consequently the chances of rupture also increases. Aneurysm rupture can lead to hemorrhage and death.\n\nRod.
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Cutting the aorta will kill you in seconds, but it's very hard to get to (thank goodness!)\n\nOh and by the way, check these out:
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http://en.wikipedia.org/wiki/Blood_vessel
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Are you talking about atherosclerotic vascular disease? If so I beleive it means the clogging and hardening of the arteries. Sclerosis means scarring and Athero has to do with the arteries vascular just means anything that is a "tube" in your body (veins, vas deferens, etc"
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You're thinking cholesterol from arteries.
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atherosclerosis, is when u have rigid arteries; They have become hardened with time for lot of causes.(Most of the times the age <50-80> is the comun cause). Resistance to the sanguineous fluid increases and fluid haveto generate a higher sanguineos pressure. So it doesnt takes so long to notice cause u will start with high arterial pressure. So ur dr will notice it. Or u can notice it by being tired, cause ur heart is working harder. U can have headaches, inflamation in low extremities. The consecuences r: heart attack, cerebral isquemia (not only u have high blood presure.. u can get mad and increase that presure too 300 for ex, so u will develop a cerebral spill). So when y get the symptoms i told u before, u should go to the dr.
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The systolic pressure is defined as the peak pressure in the arteries during the cardiac cycle; the diastolic pressure is the lowest pressure (at the resting phase of the cardiac cycle). The mean arterial pressure and pulse pressure are other important quantities.
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Larry - I'm somewhat appalled that people are chiming in with answers to your health care questions when it is clear that they don't know what you're talking about.  A person's health is not something one should be so cavalier about.  To clarify this for others, coarctation of the aorta is a condition in which there is underdevelopment of a segment of the aorta, generally at about the level of the left subclavian artery.  This can range from a mild narrowing to complete interruption of the aortic arch.  Many surgical procedures have been used to repair coarctations including: resection of the tight segment followed by end-to-end anastamosis; resection followed by placement of a prosthetic (or artificial) graft; patch augmentation of the tight segment, augmentation of the tight segment using a portion of the left subclavian artery; and ascending-to-descending aortic bypass in which a prosthetic (artificial) graft is used to create a detour around the narrow segment.\n\nIt sounds like you underwent the last procedure which was first performed, I believe, at the Mayo Clinic.  One of the first papers on this procedure by Heidi Connolly, MD, described 18 patients who underwent the same procedure between 1985 and 2000.  In that cohort, all patients were followed for an average of 4 years at which time all the grafts had remained open.  More recently, a group from Russia reported on 52 patients who underwent the procedure since 1987.  Patients were followed for an average of about 7 years (ranging from 2 years to 11 years) at which time all grafts had remained open.\n\nThe bypass procedure that we use for patients with coarctation is VERY DIFFERENT from the more common bypass surgery that is done for coronary artery disease!  It is important that you do not get the two confused as Samantha and foni.toni have done.  Given the size of the bypass needed to accomodate the large amount of blood flow, ascending-to-descending aortic bypass is always done with a prosthetic graft.  The veins from your legs that are used for coronary bypass are much too small.  At the same time, because of the high flow of blood through an aortic bypass graft, occlusion of these grafts is VERY RARE as opposed to the 50% loss at 10 years that we see in coronary bypass grafts that are taken from the leg.  Keep in mind, though, that we have only been doing aortic bypass grafts for about 20 years and in MUCH smaller numbers than for coronary bypass surgery, so we don't have as much experience with them to be able to say if they will stay open forever.\n\nIt is very important for you to continue to follow up with a cardiologist who is specially trained in congenital heart disease!  It is appropriate to get routine imaging to make sure that the graft is still open and that no leaks or aneurysms have developed.  If you have further questions on this, please don't hesitate to contact me, but again, I urge you to maintain regular follow-up with your cardiologist!  Good luck!