which could result in hemolytic disease of the newborn?
816957
What causes hemolytic disease of the newborn (HDN)?\n\n\nIt's basically an incompatible blood of mother and baby, due to an Rh - mom and Rh + dad.  Baby has Rh + and so mother's immune system (antibodies) starts attacking baby's "foreign cells".  For the second pregancy, the mom will be "Rh sensitized" --remembers, so it may most likely not occur again.\n\n"HDN most frequently occurs when an Rh negative mother has a baby with an Rh positive father. When the baby's Rh factor is positive, like the father's, problems can develop if the baby's red blood cells cross to the Rh negative mother. "
326493
Sure, there's no problem. I think that you are confusing this with rh problems that a developing baby can have, but its unrelated to this.
252783
Believe it or not, the children could have any type of blood, as long as it is positive. (A+, B+, AB+, O+)\n\nThere's a blood typing calculator on the following site:\n\nhttp://www.biology.arizona.edu/human_bio/problem_sets/blood_types/inherited.html
231409
Birth is a natural thing. there is little chances of complications with birth, but they still occur. Some of the more common are: bleeding, placenta previa, abruptio placenta, genetic anomalies, Diabetes, Pregnancy induced hypertension (PIH), and anemia. There is higer chances of havin a normal baby than having complications as long as you visit your doctor and have good prenatal care.
448941
No, sickle cell is a disease the people are born with.  It only occurs in African Americans or people of African American decent.\n\nHere is a page that makes it easy to understand...\n\nhttp://kidshealth.org/teen/diseases_conditions/blood/sickle_cell_anemia.html
855874
Yes.\nAny infection is dengerous to anyone who has an immune-supressing condition ie cancer. The fever will be even more dangerous if the child is recieveing chemotherapy as this attacks the cells which fight off infections. \nWow I've learnt something after all of these months in treatment!
799128
No. The mother needs to be RH- and the baby needs to be RH+ in order for the baby to have erythroblastosis fetalis, and it will happen only after the first child.
202335
The reaction apears differently on the majority of strips.  Hemolyzed blood is suggestive of renal sources of blood, while non-hemolyzed suggests urinary tract bleeding.  Some say Nephrologic vs Urologic causes of blood in the urine.  the strip is really a screening tool though and either way blood shows up - a follow up evaluation is needed.
479845
In their first few days of life, more than half of all full-term babies and as many as four out of five premature infants who are otherwise healthy develop jaundice. Although some babies are jaundiced at birth, most develop infant jaundice during their second or third day of life. This would usually disappear in 2 weeks.  Jaundice that develops or lasts more than 2 weeks can be a sign of a liver malfunction, a severe infection, an enzyme deficiency, or an abnormality of your baby's red blood cells.  Your doctor should do further testing to determine what the exact cause is.
1151508
There was an interesting article a few years ago in the New England Journal of Medicine regarding fetal cells and scleroderma.  Scleroderma is a rare disease that usually affects females who have had kids.  The disease is characterized by thickening and hardening of the skin in its most benign form.  The theory suggest that during birth some fetal cells are released into the mother's blood and get embedded into their face or fingers.  As time goes on, the mother's immune system attacks these fetal cells and as the theory suggest a chronic host vs graft reaction occurs.  I thought it was sort of interesting.
174184
yes they can, the Rh is made up of two alleles and the baby receives one from each parent. If both parents are heterozygous for the d gene then the baby has a one in foud chance of being RhD negative
511231
They're not far enough out of range to worry about. I have 3 dfferent books with different sets of ranges. If the WBC was really high, that would indicate some type of infection. Platelets are a blood clotting factor. Bilirubin is a reference to liver function. High levels indicate that the old bloodcells are not being broken down and 'recycled' by the liver. In adults this shows up as hemolytic anemia and in newborns as jaundice.
346724
if mother catch some infectious disease then the bacterias or virus could transfer to womb and make some problem for fetus and even abortion.\nlike Brucellas,and the virus of Measles or Rubella
944725
if der's little blood,did u jst hav an abortion. i hear it's one of d negative effects
118740
The child can have all possible blood types. \n\nA + \nB + \nAB + \nO + \n\nIt would be extremely rare, but the children can also end up with - blood if the parents both have the recessive gene for the Rh factor.
961267
Hepatits E in pregnancy usually leads to encephalopathy, liver failure and death. It is not transmitted to the newborn.
1102675
Rh negative is NOT a blood type like A or B or AB or O. You can have A or B or any of those and you are still ALSO either Rh positive or Rh negative.\n\nEveryone has an Rh factor, it's either negative or positive. \nI found out I am Rh negative when I was pregnant because women have to be tested for it. It has something to do with your antibodies attacking the blood cells of a fetus that could potentially have Rh positive blood. I had to get a shot to prevent it from happening.\n\nThe Rh in Rh factor stands for rhesus, so named because the Rh factor is an antigen that was first discovered in the blood of rhesus monkeys. The Rh antigen is called D. Most people produce the D antigen, but a few people are missing functional copies of the RHD gene, and they produce no D antigen. These are the people who have Rh negative blood types, and the non- functional version (allele) of the RHD gene is called the RHD silent allele. \n\n\nHere is something wikipedia says about the Rh negative aspect of pregnancy:\n\nRhD Hemolytic disease of the newborn is often called Rhesus disease or Rh disease. Sensitization to Rh D antigens (usually by feto-maternal transfusion during pregnancy) may lead to the production IgG anti-RhD antibodies which can pass through the placenta. This is of particular importance to RhD negative females of or below childbearing age, because any subsequent pregnancy may be affected by the Rhesus D hemolytic disease of the newborn if the baby is Rh D positive. The vast majority of Rh disease is preventable in modern antenatal care by injections of IgG anti-D antibodies (Rho(D) Immune Globulin)\n.