Can inhaling alcohol fumes invade the blood stream?
613283
Yes. Alcohol is more volitile than water. It evaporates faster. So if you inhale it it goes directly into your blood stream. The concentration is no so great as drinking it but it does enter the blood faster--instantly. Sort of like somking pot or cigarettes.
1005339
Yes it can.  Being in a pressurized area for a prolonged period of time causes it.  I have a girlfriend who is a stewardess, she talks about it, and drinking anything carbonated does it to while on the plane.
358046
As a result of peripheral vasodilation (widening of small blood vessels) the face becomes flushed and the drinker will feel warm, although in fact a greater amount of body heat is lost.\n\nThis is really dangerous in cold weather situations, where people think alcohol will keep them warm.  Yes, they will have an immediate sensation of being warm but it actually lowers the body's core temperature and can lead to hypothermia.
165396
Probably because alcohol with evaporate
1054984
I think as long as you're not bleeding, you should be ok.  (Alcohol thins your blood, so don't get drunk if you're bleeding or you'll bleed more.)
102303
yes if you have inhaled the smoke you have chemical trce in your system
145209
alcohol thins your blood.  The same with tattoos and piercings.
1051991
More than 90% of the ethyl alcohol that enters the body is completely oxidized to acetic acid. This process occurs primarily in the liver. The remainder of the alcohol is not metabolized and is excreted either in the sweat, urine, or given off in one’s breath. The latter provides the basis of the breathalizer test used in law enforcement and is the reason one can smell alcohol on the breath of someone who has been drinking recently. \nThere are several routes of metabolism of ethyl alcohol in the body. The major pathways involve the liver and in particular the oxidation of ethyl alcohol by alcohol dehydrogenase (ADH).\n\nPlaying a role, particularly at higher alcohol concentrations is the oxidation of alcohol by the microsomal (small spherical vesicles) - cytochrome P450 system (MEOS) system. In addition to these routes, there is catalase-dependent oxidation of ethyl alcohol and oxidation of it by the stomach when it is first ingested. These latter two routes of metabolism are minor in comparison to the ADH and MEOS systems.\n  \n\nAs mentioned above perhaps the major route of metabolism of ethyl alcohol is its oxidation in the liver catalyzed by the cytosolic enzyme alcohol dehydrogenase (ADH). It catalyzes the following reaction:\n\nCH3CH2OH + NAD+ -> CH3CHO + NADH + H+.\n\nThis reaction produces acetaldehyde, a highly toxic substance. ADH has broad specificity, catalyzing various alcohols and steroids and catalyzing the oxidation of fatty acids. It also is not a solitary enzyme in that there are five different ADH genes, two of which ADH2 and ADH3 shown polymorphism (variations). Of importance is the fact that the ability of people to oxidize ethyl alcohol is dependent upon the genetic makeup of the individual. People with alleles (types) of ADH2 and ADH3 may protect those having these genes from developing alcoholism. These genes are common in the Asian population and convert alcohol to acetaldehyde more rapidly than normal. Because of this increased production of acetaldehyde, this toxic compound builds up and makes people who drink too much uncomfortable and ill. Therefore, these carries are discouraged from consuming large amount of alcohol.\n\nA similar situation is found in the second step of ethanol metabolism, which is catalyzed by acetaldehyde dehydrogenase. This enzyme converts acetaldehyde to acetic acid, which is a normal metabolite in humans and hence is non toxic. The past use of Antabuse as a possible deterrent to drinking was based on the ability of Antabuse to inhibit the action of acetaldehyde dehydrogenase, thus slow down or stop the destruction of acetaldehyde. If one drank after taking Antabuse, the person got very ill. It turns out that certain individuals, again common in Asians, have a defective aldehyde dehydrogenase gene, ALDH2, in that it doesn’t metabolize acetaldehyde as rapidly as normal. Thus, a person who drinks too much builds up acetaldehyde in their system and feels bad or is sick. This manifests in Asians with the defected ALDH gene as a facial flush as they drink. These responses discourage drinking, thus preventing the development of alcohol abuse, dependence, and alcoholism.\n\nAnother system in the liver which oxidizes ethanol via the enzyme cytochrome P450IIE1 (CYP2E1) is called the MEOS system. The reaction catalyzed by MEOS is:\n\nCH3CH2OH + NADPH + O2 -> CH3CHO + NADP+ + H2O.\n\nThough of minor significance in comparison to ADH metabolism of ethanol, the MEOS system seems to play an increasingly important role at higher concentrations of ethanol. It is not surprising that there are variations in the P450E1 enzyme which lead to differences in the rate of ethanol metabolism. This may have implications for tissue damage from ethanol, particular in the liver.
834429
Yes, when drunk excessively.\n\nIt causes little crystals to form in the blood, a little like glass fragments. They then try and pass through joints like your knees, fingers and toes... when it reaches a joint where they cant pass through they get blocked up and create gout!\n\nI'll have a pint if your off to the bar!
1058861
This happened in 2005 to a young man who was the victim of hazing in Chico, CA.\n\n"Forced water consumption and heavy exercise are known to dilute the salt content of blood to the point where it interferes with brain, heart and muscle function. Without enough sodium, the brain swells and victims can suffer fatal comas.\n\nCarrington's (the fraternity pledge)  friend who survived the hazing told police that he and Carrington each were forced to drink about five gallons of water. They were kept up all night in the fraternity's basement doing rigorous calisthenics as fraternity members splashed them with ice water and turned fans in their direction."\n\nhttp://sfgate.com/cgi-bin/article.cgi?file=/c/a/2005/02/04/BAGNSB576121.DTL
758326
Yes, I agree with the no straw thing - you definitely don't want to disturb the clot. The only thing about the alcohol is that it thins the blood, so that could also disrupt the clotting and healing. You don't want to get dry socket.
529505
A lot of questions. First cigarette smoke contains nicotine and Carbon monoxide and tear and further for the body toxic ingredients, which have following effect. Nicotine increases the vascular tension of small arteries, which causes the bloodpressure to rise. There may ba also a transient increase in respiratory rate through a centrally stimulating effect. If a patient has a rapid respiratory rate, it can be caused by insufficient gas exchange through in the lung. The effectiveness of the lungs gas exchange gets worse, through longterm smoking exposure. tear and toxic ingredients seize the function of the microvilli, which are little mobile hairlike formations on the surface of bronchial cells. Their job is to transport little foreign bodies and mucus etc. out of the lung. Small areas of the lung are being plugged up and are getting inflamed. The person developes chronic bronchitis with a decrease of normally functioning lung tissue. Also Atelectasis may develop. \nThere are many reasons, why bloddpressure rises. Smoking is one of them, often it is not clear if smoking or other causes i.e. genetic predeterminatiion are contributing to high blood pressure.\nSecond the blood pressure measured is the pressure created by the left heart. If right heart pressure rises, that means thge left heart is unable to transport enough blood away form the right heart outflow, which can cause eventually pulmonary hypertension or high bloodpressure in the pulmonary arteries (the one going away from the right heart). That happens, when the left heart does not work right.\nFor smokers the blood pressure is normally only high in the left heart outflow system.\nPlasma flow is widely independent from blood systolic  pressure alone. To assess plasma flow, the osmotic venous pressures also need to be known.\nThe body usually tries to maintain homeostasis.\nThat means acutely (when the bloodpressure goes up suddenly) ANP counteracts that development and ANP goes up. Chronically elevated BP would probably not change the ANP Level. Accordingly with Aldosterone. That means the Aldosterone level would go down with sudden elevated BP
779060
They are probably reffering to breath smelling like the sauce, "alcohol"
929045
Signs of alcohol poisoning include:\n\nVomiting. An unconscious person could choke and suffocate from vomiting.\nConfusion.\nPassing out, coma, or inability to awaken the person.\nSeizures.\nSlow breathing—fewer than 8 breaths per minute.\nIrregular breathing—10 seconds or more between breaths.\nLow body temperature (hypothermia), bluish skin, and paleness
1005886
Alcohol is one of the top ingredients in disinfectants.
119349
some of the methanol will soak thru the skin and enter the bloodstream(how much I couldn't say). Once in the body it will become oxidized to formaldehyde which is very bad for the optic nerve in particular. \nit is possible to go permanently blind by this. people that consume too much methanol can and do die from methanol poisoning.\nif you say only a small amount was spilled on you you may be ok but wouldn't you rather know for sure and talk to a doctor?
875205
I can drink in front of my child without abusing him... However, I cannot in any way smoke in front of my child without having the toxins enter into his lungs...\nSure, some people get wild on alcohol, but a lot don't.  The fact of the matter is that ALL people who smoke in front of children will be sharing their terrible smoke.\nGood Question!!!