Is a blood pressure medication like Lopressor,a BETA BLOCKER slow down the thryoid function?
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Lopressor \n  \nWhat is Lopressor?  \nLopressor is a type of medication known as a beta blocker and is used in the treatment of high blood pressure, angina pectoris, and heart attack. \n \n Are there any side effects associated with Lopressor?  \nLopressor side effects may include depression, diarrhea, dizziness, itching, rash, shortness of breath, slow heartbeat, and tiredness. If the side effects continue or are bothersome, check with your doctor. Less common or rare Lopressor side effects may include blurred vision, cold hands and feet, confusion, congestive heart failure, constipation, difficult or labored breathing, dry eyes, dry mouth, gas, hair loss, headache, heart attack, heartburn, low blood pressure, muscle pain, nausea, nightmares, rapid heartbeat, ringing in the ears, short-term memory loss, stomach pain, swelling due to fluid retention, trouble sleeping, wheezing, and worsening of heart irregularities. \n  \nHow is Lopressor taken?  \nFollow the directions for taking Lopressor provided by your doctor. Lopressor should be taken with food or immediately after you have eaten. Take Lopressor exactly as prescribed, even if your symptoms have disappeared. Try not to miss any doses. If this medication is not taken regularly, your condition may worsen. If you miss a dose and it is within 4 hours of your next dose, skip the one you missed and go back to your regular schedule. Do not take two doses at once. \n  \nAre there any effects of taking Lopressor with other medications?  \nSome medicines may interact with Lopressor. Inform your doctor of all prescription and over-the-counter medicine that you are taking. It is especially important to check with your doctor before combining Lopressor with certain high blood pressure drugs. Other medications that might interact with Lopressor include albuterol, amiodarone, barbiturates, calcium channel blockers, cimetidine, ciprofloxacin, clonidine, epinephrine, fluoxetine, hydralazine, insulin, nonsteroidal anti-inflammatory drugs, oral diabetes drugs, paroxetine, prazosin, propafenone, quinidine, ranitidine, or rifampin. \n  \nHow should I store Lopressor?  \nStore Lopressor at room temperature in a tightly closed container, away from heat and light.
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"I have been on Toprol XL (metaprolol succinate), a beta blocker, for almost three years. Like many others I was having trouble maintaining erections, when finally last year I got up the nerve to mention it to one of my Drs. He acted like he had been expecting me to bring it up at some point, and gave me samples of Viagra to take. My only regret is that I did not mention it to him sooner. I feel like I'm thirty again.\nI am very happy now with the results I receive from both of these drugs. Talk to your Dr about it. I'm positive he is aware of problems caused by beta blockers, and should work with you to eliminate those problems.", Mat from NY.\n\nRead about Viagra, Cialis, Levitra - treatment for Erectile Dysfunction Treatment http://www.edpills.net/article_viagra_cialis_levitra.php\n\nA wide variety of options exist for treating erectile dysfunction. They include everything from medications and simple mechanical devices to surgery and psychological counseling. The cause and severity of your condition are important factors in determining the best treatment or combination of treatments for you. You and your doctor may also want to consider how much money you're willing to spend and the personal preferences of you and your partner. If erectile dysfunction is the result of a medical condition, the cost of treatment may be covered by insurance.\n\nOral medications\nThree oral medications are available to treat ED. These include:\n\n* Sildenafil (Viagra)\n* Tadalafil (Cialis)\n* Vardenafil (Levitra)\n\nThe Food and Drug Administration (FDA) approved Viagra in 1998, and it became the first oral medication for erectile dysfunction on the market. Since then, doctors have written millions of prescriptions for the blue, diamond-shaped tablets. But Viagra is no longer the only pill that treats this condition. Levitra and Cialis are two other options.\n\nViagra, Levitra and Cialis work in much the same way. Chemically known as phosphodiesterase inhibitors, these drugs enhance the effects of nitric oxide, a chemical messenger that relaxes smooth muscles in the penis. This increases the amount of blood and allows a natural sequence to occur — an erection in response to sexual stimulation. These medications don't automatically produce an erection. Instead they allow an erection to occur after physical and psychological stimulation. Many men experience improvement in erectile function after taking these medications regardless of the cause of their impotence.\n\nThese medications share many similarities, but they have differences as well. They vary in dosage, duration of effectiveness and possible side effects. Other distinctions — for example, which drug is best for certain types of men — aren't yet known. No study has directly compared these three medications.\n\nAlthough these medications can help many people, not all men can or should take them to treat erectile dysfunction. If you've had a heart attack, stroke or life-threatening heart rhythm during the last six months, don't take these medications. If you've been told that sexual activity could trigger a cardiac event, discuss other options with your doctor. In addition, don't take Viagra, Levitra or Cialis with nitrate medications, such as the heart drug nitroglycerin. The combination of these medications, which work to widen (dilate) blood vessels, can cause dizziness, low blood pressure, and circulation and heart problems.\n\nDon't expect these medications to fix your impotence immediately, because that's not always the case. Dosages may need adjusting. Or you may need to alter when you take the medication. Before taking any medication, make sure to discuss with your doctor its potential benefits and side effects.\n\n\nNon-surgical ED Treatment Treatment Sex Therapy\n...
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This is the purpose of this medication.  YOu obviously have a tachy (fast) arrhythmia, likely atrial fibrilation.  If this is the case then you need a beta-blocker(like sotalol).  Understand, the goal of your treatment is controlling your heart rate which is what this drug does.  If you are unhappy with this drug in particular talk with your cardiologist about another beta-blocker (but understand the side-effects are the same) OR consider a cardioversion or EP study.  Stop taking these medications can be life-threatning and set you up for increased risk of stroke
574560
yes hypotension can be treated decrease your caffeine intake and increase your water ,orange squash (not sugar free) and isotonic drinks. slightly increase your salt intake . if you are experiencing dizziness due to your hypotension you should contact your g p for a full examination
740789
Per the Phycian's Desk Reference Pocket Guide, copyright 2002 (I got this at a local drugstore 1-4-02. I'm NOT a doctor). This is what the book says, regarding your question: ""Inderal" is the brand name for "propranolol". Overdosage: No specific information on Inderal overdosage is available;however, overdose symptoms with other beta blockers (what this drug is--a beta blocker helps block certain chemicals in the brain to keep them from over-producing and causing pain. I have a history of migraines, and take a prevenative that's a beta blocker. I researched beta blockers down...that's how I got my info. about them.)include: Extremely slow heartbeat, irregular heartbeat, low blood pressure, severe congestive heart failure,seizures, wheezing." As for affects later in life...check with her doc. FYI: My dad took this for a short time after a car wreck. Under a doc's guidance, if the patient is ever taken off of this medication, it must be done slowly. If you wish to know more about this issue, talk to her doc...they can tell you more.
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This is a beta blocker and helps maintain hypertension (high blood presser) it can also help control angina pain which is well the only way that I can explain it is a very bad heart burn type of pain that hurts really bad. I really would as the doctor what it is for because some medications are used by doctors to achieve different things then what they were designed for if you ever need information on a drug your local pharmacy can print of information for you as well all you need to do is ask for it that is what they are there for.
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I have low thyroid. caused from taking Radio Active Iodine for over active thyroid. My doctor per scribed Synthroid. which must be taken for life. Some blood pressure medicines interact with the Synthroid as well as Cholesterol Medication. If you have low thyroid you should be under the care of a doctor.
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have ur thyroid tested.hypothyroid disease can make u lightheaded,exhausted,and can cause a shoulder problem called tendonitis.its a simple blood test,insist on it.also,have a diabetes test,its an illness practically epidemic in this country,and can cause tiredness,weakness,blurred vision and paraesthesias(pins and needles,creepy crawly feelings).ur hypertension meds may contain a diuretic,causinfg frequent urination-but DONT stop taking it!!!!!good luck,feel better.ps-it isnt usually a mental problem,its usually physical.ps-dont panic,but check for ms (multiple sclerosis) as well-symtoms-dizziness,lack of co-ordination,fatigue,blurred vision,loss of bladder control,paraesthesias in limbs.
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actually you can have a blockage and your cholestorol be very low.  Some people have clotting factor issues with their blood and it causes blockages very easy.  I would definately talk to you doctor and see if there are ways to help control this.  33 years old is too young to be having these kinds of issues.
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There are medications called "beta blockers" that act against adrenaline.  There are many available out there; talk to your physician.\n\nThey work by blocking one kind of "receptor" that adrenaline molecules normally plug into.  When adrenaline plugs into these receptors, this is when you get the effects that you don't enjoy - racing heart, sweating, nausea, etc.\n\nSide effects are obviously the opposite of having too much adrenaline; they include sleepiness and hypotension (low blood pressure).  The key is for you and your physician to find the right dose, if you choose this method.\n\nAs far as herbal remedies are concerned, there are a few out there, but there is no way to control exactly how much of the herb (in terms of potency) that you're taking, and the herb might also contain other ingredients that could be detrimental to your health.\n\nOne of the herbal remedies that has enjoyed popularity recently is valerian root, available in powder/gelcap and tea form.  Most nutrition stores stock the gelcap, the tea form is much harder to find.  Some people find that only the tea form is effective.  Most people find valerian to be pretty safe, but too much can make you drowsy.\n\nPreliminary research has shown that the active ingredients in valerian act very similarly to some anti-epilepsy medications (such as Neurontin) and in slight similarity to the  "benzodiazepines" (such as Valium).  Again, remember that valerian, just like any pill your doctor prescribes you, is in fact a drug.
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As long as you have a doctor prescribing Levitra you should be fine.  As you know, Levitra can cause your blood vessels to enlarge and your blood pressure can fall.  This is a big problem for people on nitrogycerin pills.  I would start on the lowest dose of Levitra and see how it goes.  You should be fine.
37652
Get some Suedafed, as long as you dont have blood pressure problems.  That'll help.  Just ask your Pharmacist.
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Bambuterol is a Sympathomimetic drug. These stimulate a branch of the autonomic nervous system that controls heart rate, they may sometimes cause palpitations and trembling.As such they need to be prescribed with caution for those with heart problems, high blood pressure or an overactive thyroid gland.\nSeek medical advice if you experience ANY problems.
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I've cut and pasted this information from the link below.  If you want to see the tables, follow the link.  For more information, try a google search and you'll get tons of responses.\n\nSerotonin syndrome is described in the literature as a potentially serious drug-related condition characterized by a number of mental, autonomic and neuromuscular changes.1 Although serotonin syndrome can cause death, the condition is mild in most persons, and with supportive care alone they tend to recover completely. The syndrome, first described in animal models in the 1950s, was referred to as the "serotonin behavioral" or "hyperactivity syndrome."1 Reports of serotonin syndrome in humans followed, and have become increasingly frequent since the 1960s. The earliest reports involved persons who were taking monoamine oxidase inhibitors (MAOIs). Some of the early reports included patients who were also taking tryptophan, a serotonin precursor.1,2\n\nSerotonin syndrome is most often reported in patients taking two or more medications that increase CNS serotonin levels by different mechanisms. The most common drug combinations associated with serotonin syndrome involve the MAOIs, selective serotonin reuptake inhibitors (SSRIs), and the tricyclic antidepressants.3 Because of the dramatic rise in the use of SSRIs, it is predicted that emergency room physicians are going to encounter the serotonin syndrome more frequently than in the past.1\n\nSerotonin Syndrome Risk Factors\n\nRisk factors for the development of serotonin syndrome are unclear, but some trends are becoming apparent as more cases appear in the literature. Some researchers have suggested that peripheral vascular disease and atherosclerosis may lead to severe vasospasm and hypertension in the presence of elevated serotonin levels. This seems paradoxical, since in peripheral areas of the body serotonin usually causes vasodilation. However, in patients with vascular disease serotonin can lead to profound vasoconstriction.6 \n\nAnother risk factor relates to drug metabolism rate. Slow metabolizers of SSRIs (approximately 7% of the population) may produce higher than normal levels of these antidepressants in the blood. A slow metabolizer receiving an SSRI in combination with another agent that raises serotonin levels is therefore at increased risk of developing serotonin syndrome.6\n\nClinical Features and Differential Diagnosis\n\nMental status changes are the most frequently reported symptoms associated with serotonin syndrome.2 Other commonly reported features include motor abnormalities, cardiovascular changes, gastrointestinal problems and miscellaneous changes such as diaphoresis and fever (TABLE 1).2,4 Martin refers to three categories of symptoms that are present: altered mental status, autonomic dysfunction and neuromuscular abnormalities.1 Sporer indicates that at least three of the following must be present for a diagnosis: mental status changes, agitation, myoclonus, hyperreflexia, fever (hyperpyrexia), shivering, diaphoresis, ataxia and diarrhea in the setting of a recent addition or increase in dose of an agent that raises serotonin levels. Sporer also points out that there should be no other obvious causes of the confusion and/or fever, and that no antipsychotic medications have been used or increased in dose prior to the onset of symptoms.3\n\nPoisonings and other diseases, such as neuroleptic malignant syndrome (NMS), can cause symptoms that are very similar to serotonin syndrome (TABLE 3). Martin offers suggestions that may be useful in differentiating serotonin syndrome from these conditions. One is an observation that bromocriptine has been proposed as a treatment for NMS and a cause or precipitant of serotonin syndrome. Another is that NMS occurs from prolonged exposure to neuroleptic agents or withdrawal of dopamine agonists, and there is lead-pipe rigidity with NMS, in contrast to myoclonus or hyperreflexia seen in persons with serotonin
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It's effects!\n\nBeta blockers block the action of endogenous catecholamines, epinephrine (adrenaline) and norepinephrine (noradrenaline) in particular, on β-adrenergic receptors, part of the sympathetic nervous system which mediates the "fight or flight" response.\n\nThere are three known types of beta receptor, designated β1, β2 and β3. β1-Adrenergic receptors are located mainly in the heart, kidney, and adipose tissue. β2-Adrenergic receptors are located mainly in the heart, lung, GI tract, liver, pancreas, and skeletal muscle. The role and location of β3-receptors is less well-defined.\n\n\n β-Receptor antagonism\nStimulation of β1 receptors by epinephrine induces a positive chronotropic and inotropic effect on the heart and increases cardiac conduction velocity and automaticity. Stimulation of β2 receptors induces smooth muscle relaxation (resulting in vasodilation and bronchodilation amongst other actions), induces tremor in skeletal muscle, increases glycogenolysis in the liver and skeletal muscle.\n\nBeta blockers inhibit these normal epinephrine-mediated sympathetic actions, but have minimal effect on resting subjects. That is, they reduce the effect of excitement/physical exertion on heart rate and force of contraction, dilation of blood vessels, opening of bronchi, reduce tremor, and breakdown of glycogen.\n\nIt is therefore somewhat unexpected that non-selective beta blockers have an antihypertensive effect, since they appear to cause vasoconstriction. The antihypertensive mechanism appears to involve: reduction in cardiac output (due to negative chronotropic and inotropic effects), reduction in renin release from the kidneys, and a central nervous system effect to reduce sympathetic activity.\n\nAntianginal effects result from negative chronotropic and inotropic effects, which decrease cardiac workload and oxygen demand.\n\nThe antiarrhythmic effects of beta blockers arise from sympathetic nervous system blockade – resulting in depression of sinus node function and atrioventricular node conduction, and prolonged atrial refractory periods. Sotalol, in particular, has additional antiarrhythmic properties and prolongs action potential duration through potassium channel blockade.\n\n\nIntrinsic sympathomimetic activity\nSome beta blockers (e.g. oxprenolol and pindolol) exhibit intrinsic sympathomimetic activity (ISA). These agents are capable of exerting low level agonist activity at the β-adrenergic receptor while simultaneously acting as a receptor site antagonist. These agents, therefore, may be useful in individuals exhibiting excessive bradycardia with sustained beta blocker therapy.\n\nAgents with ISA are not used post-myocardial infarction as they have not been demonstrated to be beneficial. They may also be less effective than other beta blockers in the management of angina and tachyarrhythmia (Rossi, 2006).\n\n\nα1-Receptor antagonism\nSome beta blockers (e.g. labetalol and carvedilol) exhibit mixed antagonism of both β- and α1-adrenergic receptors, which provides additional arteriolar vasodilating action.\n\n\n Other effects\nBeta blockers decrease nocturnal melatonin release, perhaps partly accounting for sleep disturbance caused by some agents (Stoschitzky et al., 1999).
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To be honest the correct answer is yes and no. Let me explain. If you are taking to proper amount that is perscribed then a slower resting heart rate is a side effect. If you take too much, (I know this is going to sound strange) the effect is actually the opposite. It will speed up your heart rate. And this is very bad!!!!
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Beta blockers (like Lopressor) commonly cause people to feel tired and can precipitate shortness of breath.  \n\nIt sounds like you are very focused on your B/P which in some respects is great but you can over kill it too.  Sometimes just stepping back and relaxing a little can be useful.  If you are healthy enough, adding vigorous physical exercise like swimming laps or playing tennis can be helpful in reducing you blood pressure.  \n\nUnless you have some dangerous condition, like an aneurysm, then taking a few months to get the B/P under control is okay.  Most people require two and often three medications to adequately treat the condition.  You are probably saying "Three medicines???"  Taking modest doses of two or three medicines can often produce great results with no side effects.  ACE inhibitors (or ARB's are a newer generation but are more expensive), diuretics (HCTZ) and B-blocker or Calcium Channel Blockers as a third adjunct...these choices can be influenced, however, by other conditions that you are at risk for that might benefit from these medications too.\n\nContinue to work with your medical provider and you will achieve your goal...good luck.