what is metabolic syndrome and how it can be cured ?
489334
Signs: increased blood pressure, excess body fat around waist and abdominal area, and/or elevated insulin levels.\n\nThreat: greatly increased chance of developing diabetes, heart disease and/or stroke\n\nCure: Exercise, quitting smoking, losing weight and changing diet. In all, it involves an aggressive lifestyle change.
384361
G6PD is a metabolic disorder also known as Von Gierke's Disease. It is an enzyme deficiency that prevents glucose from being released from cells into your blood. Which results in enlarged liver and kidneys. \nIt is genetic, and recessive, so a person needs to get both copies of the gene to have it. It can be treated, but not reversed.
666248
Mostly this is something that is due to inheritance and then to our lifestyle and eating habits that are in our control.  So get into the habit of exercise every day, like walking or ride a bike.  And eat healthy too.  This will do a lot to help.  The other big factor is stress.  Take things in stride.  Live for the moment.  Have fun, fun, fun.  Stay nice and be happy.  It does work.  And don't ever worry, like worry about blood pressure.  Let the doctors do what they do, and you do what you do.
849726
You "heal" obesity by eating less and moving more.  That is all.  If you have a thyroid issue, then you see a medical doctor, and they can help you.
1062088
Lipo was designed to just get rid of stubborn fat, not as a cure all. If you don't keep up with diet and exercise, the weight will all come back. And the bad part? It'll come back in a bigger quantity.
773851
There is no known cause for PCOS. The only link between that and diabetes is insulin resistance.
10865
Doctors today say that the best diet - as in way to eat - is to eat six small meals a day. This helps keep your body going throughout the day & spaces out the intake of needed vitamins & nutrients. No matter what, you need to eat at least three solid meals a day or you set yourself up for metabolic problems. I know you didn't ask but I want to share my own story with you in order to highlight what can happen if you don't eat enough...it should help you to understand how important it is:\n\nI've been overweight most of my life but, until a few years ago, did not know that this was due to a metabolic condition rather than just eating too much. My problem was that I would literally forget to eat &, on a normal day, could only be counted on to eat one meal. Though I was extremely overweight, I was never hungry & always thought that if I ate less meals, I would shock my system into losing weight.\n\nWell, I shocked my system alright. Not only did I develop diabetes as an outgrowth of the other metabolic conditions but I messed up my system so bad that there is a chance I will never be able to have children. Why? Because I starved my body of the proper protiens & nutrients for so long that it might not ever recover. The effects of years of mistreating my body in this manner - without even realising I was doing it - even extend to the fact that I recently had to have all of my teeth pulled; having not gotten the proper balance of protiens, etc over all of those years, my teeth literally wasted away in my mouth! I can assure you that none of this has been fun & I am not making a single thing up just to get my point across.\n\nIt has taken my literally years now to even attempt to get myself back in shape. The diagnosis of diabetes scared me silly & I knew I needed to make some significant changes to take whatever procautions I could to live a long & happy life with my husband. I've since learned that you have to eat to live & be healthy. I eat anything I want, including sugar, but I do so in moderation & always with a mind to what I am eating & when I get full. It took me years to teach myself how to know when I was hungry &, honestly, I still have some times that I eat only because I know I need to do so. I have lost over 150 lbs. in the past few years doing just this & the effects have been amazing. I no longer show any signs of diabetes &, although I miscarried, actually got pregnant & kept it for a number of months. At this point we are hoping that my body heals itself in enough time to beat the ticking of my biological clock :)\n\nThe point here is that you can do some terrible things to your body without even realising it & the effects of these things can be much more far-reaching than you might ever think. If you want to live a nice long life, have children & be healthy, you need to start taking control of it now. Not doing this same thing when I was younger has caused me more heartache than I ever considered possible. Had I not ignored the signs of what was happening to me - through missed periods & feeling tired all of the time - I might not be in the recovery stage that I am today. Please don't let the same thing happen to you...
337159
All these quick-fixes for fat reminded us of DHEA, a wonder compound of the 1990s. DHEA (dehydroepiandrosterone) was supposed to be the Vise Grips of drugs. snake oil?Like the all-powerful super-pliers, it was expected to solve problems yet undiscovered. It was (to overstate it slightly) the cure for which there was no unsusceptible disease.\n\nIn studies over the past 10 years, this naturally occurring hormone prevented cancer and helped fight viruses, mostly in rodents. It restored vitality and libido to the aged and happiness to the depressed, and even slowed the tentacles of Alzheimer's disease. It made vaccines more effective in the elderly, and lowered bad (LDL) cholesterol, thus presumably protecting the heart and arteries.\n\nJust when we began hoping that DHEA would fight flab and change the oil in the old Chevy... along came evidence that DHEA could increase insulin sensitivity, a primary cause of adult-onset diabetes. This blood-sugar disorder affects about 14 million Americans, according to the American Diabetes Association.\n\nAnd it does seem that large doses of DHEA can help fight the bulge -- at least if you're a dog. In a study to be published in Obesity Research, University of Wisconsin-Madison scientists Ilene Kurzman and Gregory MacEwen fed a group of dogs a low-fat, high-fiber diet. Half of the dogs -- chosen at random -- also got a high dose of DHEA, but their identity was not known to the researchers during the study.\n\nDHEA worked: while the control dogs lost 5.5 percent of excess body weight per month, the group treated with DHEA lost 10 percent, says Kurzman.\n\nMeet the cure-all\nBut long before these results were due for publication, DHEA had gained a reputation as a cure-all. After Food and Drug Administration regulations were changed in 1994, it began appearing on drug store shelves as a "dietary supplement."\n\nYet despite years of study and a great deal of new enthusiasm, DHEA is not something that can be recommended for controlling obesity, or much else either. "I would not recommend it for weight loss," says MacEwen, a professor of veterinary medicine who's studied DHEA for almost a decade. "When you look at the rodent studies, you see that they were feeding it at about 0.1 percent of total diet."\n\nfat cell That's a lot -- about 100 times the dose per kilogram of body weight in the pills at your drugstore. (Dietary supplement pills are formulated to restore the DHEA level to what's in the blood of a 30-year-old.)\n\nFurthermore, as MacEwen points out, most of the obesity research has been done in mice and rats. "But a rodent is not a small human, and DHEA may have a totally different effect in people." Specifically, rodents normally have much less DHEA than humans do, so they're not necessarily a good species to test the effects of supplements.\n\nWhat do we know about DHEA?\nIt's a natural hormone produced by the adrenal gland (that's the little critter hitchhiking on the kidney). The concentration peaks in humans about age 25 -- (need we add at the time of peak health and vitality? OK, we added it).\n\nFrom that peak, blood levels decline at about 2 percent per year, raising the prospect that the hormone's disappearance could be linked to all sorts of problems of aging.\n\nLike "not getting it" about cyber-electro-funk music? No. Like decreased immune function, heart attacks, and gaining weight.\n\nIt's not known whether DHEA is actually a hormone, or just a precursor ("raw material") for the hormones testosterone and estrogen. If it's a precursor, then taking supplements could be dangerous -- since estrogen and testosterone can both speed the development of cancers. Indeed, MacEwen says, the only side effect seen in his dog study was an increase in aggressiveness, as the dogs "tended to take on male characteristics toward other dogs." And that's a sign that DHEA is being
938186
There are no easy solutions, the best thing is to look for small improvements you can make to your lifestyle and then stick to them.\n\nDiet:- Look at what and when you eat. Use diversionary tactics to avoid occasions where you find you are eating between meals.\n\nExercise:- This doesn't have to mean exercises, walking briskly exercises the heart and burns-off the calories! - Consider taking-up a sport as a social activity - something you can strive to get better at whilst getting exercise - perhaps something like Table-Tennis or Badminton.
190080
Treatment    Return to top \n\nTreatment for this disease concentrates on avoiding low blood sugar. Frequent feedings are needed during the day, especially foods that contain carbohydrates (starches). Neither fruit nor milk sugar can be adequately broken down in this disease, and are best avoided.\n\nOvernight fasting is avoided by continuous feedings (through a tube that goes through the nose into the stomach) of sugars or uncooked cornstarch. The tube can be put in at bedtime and taken out each morning. A medication called allopurinol, which can lower blood uric acid and decrease the risk for gout, is often used.\n\nSynonyms:\nvon Gierkes disease\nGlucose-6-Phosphatase Deficiency Glycogen Storage Disease\nType I Glycogenosis\nHepatorenal Glycogenosis\n\nIn Type I Glycogen Storage Disease, the enzyme glucose-6-phosphatase is defective in the liver, kidney, and small intestine, where it normally functions. However, the metabolic problem is centered in the liver.\n\nPatients with Type I GSD are unable to release glucose from glycogen. They cannot maintain their blood glucose levels and within a few hours after eating develop hypoglycemia (low blood sugar). The low levels of glucose in the blood of these patients often result in chronic hunger, fatigue, and irritability that is especially noticeable in infants. Symptoms of hypoglycemia only begin to appear when the interval between feedings increases and the infant sleeps through the night or when an illness prevents normal feeding routine. If the blood sugar falls to a low enough point, some patients may experience seizures. \n\nSince Type I patients are able to store glucose as glycogen but not release it normally, with time the stored glycogen builds up in the liver. Hormones, lactic acid, triglycerides, and other products are increased in the blood as the body attempts to raise blood sugar. Fats are stored in the liver along with the glycogen, which leads to enlargement of the liver. The liver does its many other functions normally, and there is not usually any evidence of liver failure. The kidneys are also enlarged due to glycogen accumulation. \n\nThe continued presence of low blood sugar eventually leads the body to experience severe biochemical abnormalities and delayed growth and development. Untreated patients may have muscle wasting and a buildup of body fat.\n\nWhen blood studies are done, it is discovered that there is usually a low blood sugar, elevated blood lactic acid, elevated cholesterol and other fats, and elevated uric acid concentration. High blood pressure has also been seen in a number of patients and when this occurs, appropriate treatment must be given.\n\nIn addition to the problems described above, patients with Glycogen Storage Disease Type Ib can develop frequent bacterial and fungal infections, due to abnormal functioning of the white blood cells. They may also develop chronic pancreatitis, chronic inflammatory bowel disease, and Crohn's disease. \n\nThe diagnosis of Type I GSD will always include blood studies, x-rays (including some of the kidneys), measurements, and ultrasound of the liver. Gene-based mutation analysis can be used to diagnose the majority of Type I patients, while liver biopsy analysis may be needed in some cases. \n\nThe treatments of Type I Glycogen Storage Disease are aimed at correcting the biochemical abnormalities and promoting growth and development. Current treatments consist of providing a continuous source of glucose by giving the patient glucose drinks frequently during the day and, in most cases, continuously overnight. Many medical centers have replaced these glucose feedings with uncooked cornstarch doses, mixed in water, Kool-Aid, or formula. \n\nLinks:\nvon Gierke Disease info from National Organization for Rare Disorders            [NORD]\nOMIM GSD Type I on Online Mendelian Inheritance in Man\n     searchable database\nGSD Type I from emedicine\nAmerican Liver Foundation information on Type I\nvon Gierke D
339451
First thing to do is see the doctor. Make sure you are ok inside first before changing your diet or activity level. If they sat your fine then go for the change in lifestyle. You did the first step by realizing this problem. Do not depend on diet pills for the cure, you'll need to eat smaller amounts and get more active, which can be the hardest part sometimes. Put away the TV controller and get outside. Walk, run, take horse riding lessons, Join a workout class 3x a week, Drink water or Gatoraide..no soda. Its so bad for you, even diet pop. Eat a reasonable amount of fruits and veggies and lay off on the excess amounts of meats and greasy fatty foods. Good buy McDonalds. It will be so hard to follow through but after a time you will feel so great about yourself and your body will follow. Be positive, your doing the right thing. And will live longer too..and your loved ones will like that a lot.
158892
There is no cure for Polycystic Ovarian Syndrome, medications used depends on the symptooms you are having.\n\nHere is a website that explains more:\n\nhttp://www.4woman.gov/faq/pcos.htm
204880
I think you are a bit confused. You think that a cure exists and it is being hidden?\n\nType II Diabetes, which used to be called adult-onset diabetes, almost always results from lack of exercise and obesity. It can be easily prevented by controlling one's weight and by exercising. Even people who have it can often "cure" themselves by exercising and eating sensibly. This is far easier than waiting for some magical medicine or treatment to appear. \n\nType I diabetes appears to be an autoimmune disease. We are slowly understanding the immune system, but it is very complicated and we are making only slow progress against many of them, like lupus, rheumatoid arthritis, multiple sclerosis, etc. With so many diseases to deal with, it is hard to know which diseases to put our scarce research resources towards. Everyone believes that the disease they are personally most interested in is the the most important.
22480
Don't know if it can be cured or not. There is medication that can work well.\n\nThere is also some evidence that gout is influenced by diet. Here are some suggestions that may improve gout: \n\nDecrease consumption of red meat. \nDecrease consumption of all seafood. \nAvoid coffee and all other sources of caffeine (which can raise uric acid levels). \nDrink lots of water to help flush uric acid out of your system. \nMinimize alcohol consumption. \nIf you're overweight, try to lose excess pounds.
442292
I also have PCOS and I highly recommend the SugarBusters diet.  It helped immensely.  I've lost about 25 lbs on it.
38412
It helps to get rid of fat.
473454
Eat healthy and exercise.  Cardio is the best exercise you can do to lose fat.  It increases your metabolism and insulin levels which will help break down fats and sugars in your system.\n\nThat is the smart, sensible way.  If you want a quick fix you aren't going to find one because even liposuction won't change your lifestyle.