Is the return of blood to the heart in an elite athlete different from that in an untrained person?
483322
Yes circulation is increased in trained athletes.  The blood also carries and delivers more oxygen to the cells
231067
it depends on your energy expenditure during the race, the 5% improvement of HCT improves oxygen delivery to tissues about 2.5%. so the capacity for exercise would improve by 2.5%.
91097
It is probably pride and that Americans don't understand a thing about cycling. Offcourse there is doping in cycling. But then is the question what is seen as doping. I mean it is not normal the tour the france to cycle everyday about 180 kilometers in the max. Your body must suffer it is not healthy anymore. So they will need something to keep their bodies healthy and keep on with everybody and perform perfect the next race. I know Lance does have an extreme VO max, and he has certain abilities rarely seen by people. But they will take something to keep their bodies healthy, and then is that doping too? Doping is everything what is on the doping list I know. But this line is thin. There is this Belgium doctor who has been wirting a book about it, what is doping. He was the doctor of the Festina team in 1997. So to say is Lance doped, just how you want to answer this question.
757695
I also have an enlarged heart and I am 67 and have led a very rewarding life so far.  I excelled in sports and I have kept myself physically fit for all these years without the enlarged heart causing me any problems.  One can lead a normal life if he/she doesn't dwell on the bad side of this condition.
867617
It's alot of both. Genetics are what gets you to that pro athlete status and hard work and preparation are what separates you from the group and takes you to the next level. Without genetics, a person can practice and prepare as much as they want, but it will honestly take you only so far. There have been many people with a ton of heart, but how many of them actually were true champions. Alot of them were scrappers and a few squeaked in as champion, but you have to have a certain genetic make-up to really excel at athletics.
399254
weightlifting using diet and cardiovascular activities. It is what competitive body builders do
480482
Has to do with oxygen demand.  During exercise your heart rate goes up because oxygen is being used much faster.  The faster pulse gets blood and oxygen to tissues faster.\n\nWhen you stop exercising, there is less demand for oxygen so the heart rate goes back down.  If it stayed high, it would cause high blood pressure and damage the vessels.
428884
So that his body gets adapted... as he does more exercise, his heart will pump more forcefully, so it will undergo a compensatory increase in size... and this will make him able to pump more blood to those muscles!
725715
Harder for a few days. But when you get back to Ca, it will be easier. That's why athletes train at altitude..............
869540
He didn't take drugs, he just trained harder than anyone else.\n\nAn independent physiologist familiar with the study commented: “This study shows that long term training has a lot bigger effects than we thought. They followed Armstrong – a well-known hard trainer – and the changes in his efficiency over seven years are really quite amazing. We wouldn’t be surprised if some major physiological changes happened, but here’s a real quantified example.”\nHe added: “Generally there are two ways to improve efficiency: Train your maximum capacity to be very high, or train your sub-maximal capacity to be very efficient. In Armstrong’s case, he did both. In the lab they measured his performance against standard oxygen consumption and by the end of the study he was much more efficient utilizing the same amount of oxygen. But on the road,” he pointed out, “it means he can go faster and get more guys off his wheel.”\nEffect of cancer, therapy nil\nThe period that started when Armstrong was 21 and just turning professional and ending at age 28 with his first TdF victory, also included his cancer diagnosis, surgery, chemotherapy and recovery. About eight months after chemotherapy ceased (August 1997), Armstrong was tested in Coyle’s laboratory in the same manner as in his other four visits. The results showed that he “displayed no ill-effects from his previous surgeries and chemotherapy” and were in line with measurements expected from highly trained athletes during periods of detraining, Coyle added later.
67467
No.  The heart is just a muscle.  If you exercise, it beats more, but gets stronger and lasts longer.
512403
There are three major systems in your body that require lots of blood.  Your brain, your muscles and your digestive track.  Typically, your body is going to keep your brain fully supplied.  If you fill your stomach with food and then start strenuous excersise, your body has to make some choices.  Usually, your stomach loses and all that food just sits there irritating you.  The reality is that your body can handle this without any real issues.  How the body handles blood also explains how a boxer can be knocked out.  A solid blow to the head causes the blood control to blank out, allowing a lot of blood to go to the digestive track.  The result is that suddenly the muscles and brain are under supplied and the boxer falls over.
1097365
Treadmill stress electrocardiography is employed widely to detect underlying cardiovascular disease in asymptomatic persons. To determine the frequency and significance of an abnormal S-T segment response to exercise in young athletes with increased left ventricular (LV) mass induced by physical conditioning, 75 male isometrically trained athletes without evidence of heart disease (mean age 21 years) and 48 nonathletic young normal subjects were studied by M-mode echocardiography and exercise electrocardiography. Ventricular septal and posterior free wall thicknesses and calculated LV mass were significantly greater in athletes than in control subjects. An abnormal S-T segment response to exercise was present in 7 (9%) of the 75 athletes, all of whom had a LV mass greater than 275 g and in 5 the LV mass was above the 95th percentile of that of the control subjects. An abnormal treadmill exercise test result also was present in 3 (6%) of 48 control subjects. Seven of the 10 subjects with abnormal exercise test results had radionuclide angiograms at rest and with exercise, each of which was normal. It is concluded, therefore, that (1) abnormal exercise test results occur commonly in both athletes and non-athletes; (2) almost 10% of isometrically trained athletes have "false-positive" exercise electrocardiograms, suggesting that this test has major limitations in screening for underlying cardiovascular disease in athletes; and (3) in athletes, a relation was present between "physiologically" increased LV mass and false-positive exercise test results.
278295
Does entering a competition where you know you are going to loose count? If so then yes, all the time.\n\n(When you run a marathon, with thousands of people, it's a good bet that you will get beat by the guy who's on the Olympic team.)
772616
The pages listed under this information section  contain summaries of relevant topics written by many different experts in the field for the Montgomery Heart Foundation for Cardiomyopathy. The information contained in these summaries was originally collected in 1997 to put together a printed brochure for patients, families and health care professionals.  Although these summaries are now 6 or 7 years old, they have been re-reviewed and we believe  contain basic and general information that is still helpful to patients and families today.\n\n \nWhile the entity of the "athlete's heart" has been recognized for over 100 years, only in the last two decades has the application of echocardiography and other noninvasive imagine techniques permitted definition with some precision of the alterations in cardiac dimensions associated with athletic conditioning.  Echocardiography has demonstrated that long-term athletic training leads to an increase in left ventricular (LV) mass due to increases in LV diastolic cavity dimension, wall thickness, or both.  These changes in cardiac morphology are relatively mild in absolute terms, and the differences between athlete and non-athlete populations are statistically significant but generally small.  Furthermore, cardiac alterations associated with training differ somewhat depending on the particular sporting discipline in which the individual athlete participates.  In particular, the changes in LV wall thickness, cavity dimensions, or both associated with long-term athletic training may be more striking in certain sports such as distance running, swimming, cycling and rowing/canoeing.  It is in athletes raining in such sports that the differential diagnosis is more likely to be raised.\n\nDifferential Diagnosis\n \nDilated Cardiomyopathy\nIn an important minority of athletes, the increase in LV end-diastolic cavity dimension that occurs with training overlaps that which is characteristic of certain pathologic entities.  While LV cavity dimension is athletes is usually in the range of 53 to 58 mm, in some individuals it may extend into what is regarded as the pathological range of >60 mm (up to 70 mm), and thereby resemble dilated cardiomyopathy.  However, the absence of LV systolic dysfunction is usually sufficient to distinguish such physiologic ventricular enlargement induced by training from dilated cardiomyopathy. \n\nArrhythmogenic Right Ventricular Dysplasia (ARVD)\nBecause highly trained athletes may demonstrate right ventricular enlargement and a variety of depolarization, repolarization and conduction abnormalities on the ECG, the differential diagnosis between athlete's heart and ARVD may arise.  Identification of ARVD by echocardiography may be exceedingly difficult because of technical limitations in imaging right heart morphology (and assessing right ventricular function), and also because the spectrum of disease is broad.  Demonstration of right ventricular segmental or global dysfunction or substantial cavity enlargement supports the diagnosis.  Magnetic resonance imaging, however, affords a more reliable noninvasive diagnosis of this condition.  In ARVD, ECG's frequently show T wave inversion in V1-V3.\n\nHypertrophic Cardiomyopathy\nThe dilemma of distinguishing clinically between athlete's heart and structural heart disease most frequently arises with respect to hypetrophic cardiomyopathy (HCM).  While at present there is no single approach that will definitively resolve this question in all such athletes, several strategies are described here that alone or in combination offer a large measure of clarification in most instances for this often compelling differential diagnosis.  The definition of HCM employed here is that of a patient (or athlete) with evidence of a hypertrophied and nondilated LV in the absence of another cardiac of systemic disease that could itself produce hypetrophy of the magnitude present in that individual.\n\nWall Thickness\nIn
63187
Are you talking about sports?...its "blood-doping"\nhttp://www.rice.edu/~jenky/sports/epo.html
488125
Exercise benefits you in many ways. Cardiovascular exercise increases your endurance and the strength of your heart.  That is why elite athletes have low pulses because their hearts are stronger. \n\nAlso, strength training helps increase your insulin sensitivity which will reduce your risk of developing type 2 diabetes.\n\nIt will decrease your risk of heart disease, diabetes, obesity, and several other diseases. There are too many benefits to enumerate.