what sort/kinds of food are probihited from a person with goiter?
479208
There are no dietary restrictions for a person with hypothyroidism (goiter).
310934
meat,all grains,fish,chicken,liver,eggs.....
348195
balanced iodine intake is better. low-glycemic, high-fiber, lower-calorie diet, optimal timing of meals for maximum hormonal impact, thyroid-damaging foods to avoid, helpful herbs and supplements, \n\n\nother than this all diets are good. but for hypothyroid , tablets like electrocyin is must to be taken daily.
225844
One of the best foods that you can eat in cases of hypo-thyroid. It is rich in many important minerals. Antibacterial and anti-viral activity in brown Laminaria type seaweed known as kelp. It supposedly kills herpes virus, for example. Kelp may also lower blood pressure and cholesterol. Wakame boosts immune functioning. Nori kills bacteria and seems to help heal ulcers. A chemical from wakame seaweed is a clot-buster, in one test twice as powerful as the common drug heparin. Most types of seaweed have anti-cancer activity. Might aggravate acne flare-ups.\n\nYou can find it in some health food stores.
572932
-fish\n-whole grains\n-lots of water and tea\n-vegetables\n-some low sugar fruits\n-healthy fats such as fish oils and olive oil
240643
Whole Wheat breads and grains, and pasta, lots of fruit and vegetables, tuna, chicken white breast only, lean ground beef, and rice.  Egg whites, and yogurt, mozzarella cheese only.
635054
a bit of every type of food :-)\nproteins, fats, carbohydrates, fresh fruit and vegies and meat
745755
spaghetti noodles and sauce.... \npeanut butter\nramen noodles\ndried beans\nbisquick baking mix\nground beef\nstuff that would last a while, is cheap, and would be easy enough to whip up something out of if I got hungry.  I've been in that situation before....  Ate a lot of macaroni
1121766
The Thyroid Foundation says not to, as does Mary Shoman (an expert in the field):\n\n"Is goitre due to lack of iodine in our diet? Should I eat kelp to get rid of this goitre?\n\nANSWER:\nIodine deficiency is the most common cause of goitre in the world but NOT in this country. We have had iodized salt for two generations, and there are many other sources of iodine now in our diets so that, if anything, we are receiving more iodine than in most places in the world. There is no point whatsoever in adding more iodine to the diet and that includes kelp, a form of seaweed high in iodine often sold in health food stores. The problem is that too much iodine is just as dangerous as too little iodine. Excess iodine can also cause goitres and can either cause a low thyroid state, (hypothyroidism) or excess output of thyroid (hyperthyroidism). It also aggravates Hashimoto's thyroiditis. In Canada it is a mistake to ingest kelp."\n\nMary Shoman said:\n\n"While some herbalists and vitamin proponents recommend iodine tablets or kelp supplements (which are high in iodine) for people with thyroid problems, you need to be extremely careful about any decision to take iodine or kelp supplements if you are on thyroid hormone replacement therapy."\n\nRead more: http://thyroid.about.com/cs/drugdatabase/f/kelp.htm\n\nHope that helps!
614829
Anything on the Mediteranean diet (what the Italians eat) Italians have less heart disease than those of us in US and UK, and this is attributed to their diet. Lots of lovely lean meat and fresh fish with vegetables and salads dressed in olive oil. Add to this as much garlic as you can stand....or garlic capsules if your partner complains. It has the side effect of keeping the vampires away!\n\nTry cookin fresh salmon by wrapping in foil with lemon and parsley and baking in the oven for 15 minutes, medium heat. Serve with potato wedges (preferably home made and without extra salt) Add garlic for flavour instead of salt. Choose your favourite salad or veg to go wit this dish. If you don't like these foods much, don't worry, you can get used to them, and you will soon wonder why you ever ate anything else, because they are Real food and very good for your health generally, as well as your heart.\n\nKeep your intake of salt as low as possible - lots of processed foods contain this.\n\nSorry, but you should avoid smoking and large quantites of alcohol!! But a glass of wine a day with your evening meal could be beneficial - check with your doc.\n\nTry to keep your stress levels low. Do some very gentle exercise and some relaxation to your favourite music.\n\nAsk for and give lots of HUGS!! Very therapeutic "food" for the soul.
284531
Got milk? Also, salmon, sardine or any other fish with the bone in, and green leafy vegetables. Try taking a vitamin, calcium with vitamin D.
331221
A goiter is an enlargement of the thyroid gland. Persons with enlarged thyroids may have normal function of the gland (euthyroidism), thyroid deficiency (hypothyroidism), or overproduction of the hormones (hyperthyroidism). Goiter may be congenital or acquired, endemic or sporadic.\nThe goiter often results from increased pituitary secretion of thyrotropic hormone in response to decreased circulating levels of thyroid hormones. Thyroid enlargement may also result from infiltrative processes that may be inflammatory or neoplastic. Goiter in patients with thyrotoxicosis is caused by thyrotropin receptor-stimulating antibodies.\n\n\n Figure 561-1 Congenital goiter in infancy. A, Large congenital goiter in an infant born to a mother with thyrotoxicosis who had been treated with iodides and methimazole during pregnancy. B, A 6-wk-old infant with increasing respiratory distress and cervical mass since birth. The operation revealed a large goiter that almost completely encircled the trachea. Notice the anterior deviation and posterior compression of the trachea. Partial thyroidectomy completely relieved the symptoms. It is apparent why a tracheostomy is not adequate treatment for these infants. The cause for the goiter was not found.\n561.1 Congenital Goiter\nCongenital goiter is usually sporadic and may result from a fetal thyroxine (T4 ) synthetic defect or the administration of antithyroid drugs or iodides during pregnancy for the treatment of thyrotoxicosis. Goitrogenic drugs and iodides cross the placenta and at high doses may interfere with synthesis of thyroid hormone, resulting in goiter and hypothyroidism in the fetus. The concomitant administration of thyroid hormone with the goitrogen does not prevent this effect, because insufficient amounts of T4 cross the placenta. Iodides are included in many proprietary preparations used to treat asthma; these preparations must be avoided during pregnancy because they have often been a cause of unexpected congenital goiter. Amiodarone, an antiarrhythmic drug with a 37% iodine content, has also caused congenital goiter with hypothyroidism. Even when the infant is clinically euthyroid, there may be retardation of osseous maturation, low levels of T4 , and elevated levels of thyroid-stimulating hormone (TSH). In women with Graves disease on antithyroid drugs, these effects can occur when the mother takes only 100–200?mg of propylthiouracil/24?hr; all such infants should undergo thyroid studies at birth. Administration of thyroid hormone to affected infants may be indicated to treat clinical hypothyroidism, to hasten the disappearance of the goiter, and to prevent brain damage. Because the condition is rarely permanent, thyroid hormone may be safely discontinued after the antithyroid drug has been excreted by the neonate, usually after a week.\nEnlargement of the thyroid at birth may occasionally be sufficient to cause respiratory distress that interferes with nursing and may even cause death. The head may be maintained in extreme hyperextension. When respiratory obstruction is severe, partial thyroidectomy rather than tracheostomy is indicated ( Fig. 561–1 ).\nGoiter is almost always present in the congenitally hyperthyroid infant. These goiters usually are not large; the infant manifests\n\n1882\nclinical symptoms of hyperthyroidism, and the mother often has a history of Graves disease (see Chapter 562.1 ). TSH receptor-activating mutations are also a recognized cause of congenital goiter. \nWhen no causative factor is identifiable, a defect in synthesis of thyroid hormone should be suspected. Neonatal screening programs find congenital hypothyroidism caused by such a defect in 1/30,000–50,000 live births. If the infant is hypothyroid, it is advisable to treat immediately with thyroid hormone and to postpone more detailed studies for later in life. Because these defects are transmitted by recessive genes, a precise diagnosis is helpful for genetic counseling. Monitoring subsequent
702765
Eat more whole foods.  Avoid all soy.  Soy slows down the thyroid function.  Use Celtic Sea salt.  Take 3 tablespoons of virgin coconut oil per day.  I take 1 tablespoon with each meal.  The coconut oil will rev up your metabolism.  Take a whole mulitvitamin.  I like http://www.drrons.com/ his does not have any of the additives.  And you will eventually not need thyroid medication.  You can also check out Dr. Bruce Rind's website he is an expert in throid and adrenal glands problems.  About 20 percent of his patients fly in from out of town.  His office is in Washington, DC.\n\nhttp://www.drrind.com/
637522
avoid cabbage and soy. natural whole foods are best, especially ones that contain iodine.
83532
Don't believe that crap about blueberries or bannanas.  They are lacking in protiens and fats that you need.  Cheezeburger is probably the most realistic answer here but there are many possibilities, e.g. quinoa, soybeans, fish, most meats.
206109
My dad had gout and he had to give up corned beef.  Every time he ate corned beef, he had a terrible bout with gout.\n\nI found the list below:\n\nOffal foods such as: Liver, Kidneys, Tripe, Heart, Tongue, etc. \nExcessive amounts of red meat and poultry. \nShellfish, mussels, anchovies, fish roe, herring, mackerel, sardines, shrimp, and scallops. \nPeas, legumes, spinach, lentils and beans. \nYeast. \nMushrooms \nasparagus \nAlcohol intake should at least be reduced (beer and wine especially
60725
Expanding goiter?  Do you mean avoidance of the thyroid gland enlarging due to goiter?  Goiter can develop because of excessive thyroid hormone production or the failure to produce enough hormone.\n\nA diet deficient in iodine can cause a goiter but this is rarely the cause because of the readily available iodine in our diets. A more common cause of goiter in America is an increase in thyroid stimulating hormone (TSH) in response to a defect in normal hormone synthesis within the thyroid gland. -- So, basically, iodine isn't always the answer.\n\nTreatment involves getting the thyroid hormone levels back to normal, usually with medication. When the medication takes effect, the thyroid may begin to return to its normal size. However, a large nodular goiter with a lot of internal scar tissue will not shrink with treatment. If the goiter is uncomfortable, causes overproduction of thyroid hormone unresponsive to medications, or becomes cancerous, the entire thyroid gland may have to be surgically removed.