what is the positive and negitive aspects of genetic mutations?
478328
Nothing is so black and white in science. Let's take the example of sickle cell anemia, this inherited disease is caused by a genetic mutation which results in abnormal sickle hemoglobin (Hb S) being made. Red blood cells become deformed (sickle/crescent) shaped upon deoxygenation and become stuck in blood vessels. This is a chronic and painful disease which damages organs and shortens the life span of the patient. \n\nA positive aspect of this mutant hemoglobin is that patients suffering from Sickle cell trait (i.e. a proportion of their blood cells are normal), have an advantage when it comes to Malaria, a disease which kills 1.3 million a year and is found mostly in West Africa where sickle cell anemia is also most common. The malaria parasite hides in red blood cells, but because of the damaged sickle RBC structure it is extremely difficult for the parasite to survive in the human body.
154585
Chances of genetic mutation are 100%.  They are happening all the time in your body, but your body fixes them most of the time.  However, sometimes it doesn't catch them and thats when you get cancer.
659200
I'm not sure what you are asking because of the typos. But here goes an answer for what I think you are trying to ask.\n\nA point mutation involving the addition of a BASE early in the coding, The protein resulting from The gene will be entirely different from the point of addition, since the reading frame will be moved forward. \n\nExample (HYPOTHETICAL)\n\nNormal DNA\nATCGGTACCATT\n\nNormal mRNA\nUAGCCAUGGUAA  The reading frame would be UAG-CCA-UGG-UAA...\n\nmutated mRNA (a C added by mistake)\nUAGCCCAUGGUAA The reading frame would be UAG-CCC-AUG-GUA..\n\nThis can cause the protein to be shorter, because it can create a stop codon somewhere with the addition, also it can make it longer, a stop codon no longer present where it should be.\n\nBasically the protein would be non-functioning.
225669
When thereis a mutation in the body cell. I COULD be past on. it's not a 100% chance. but it's possible. \ncan you give me the 10 points since i'm the only one who answered it .
591012
well...\nu could talk about how little we know about whether organisms like bacteria and viruses have consicousness and a probable effect that might happen if we change their genomes . we dont really know whether those kind of organisms have an "individuality" or anything, so thats where i would start off from.\nhope this helped
1079358
Sickle cell trait carriers.\nNot sickle cell anemia.\nThe carriers of this trait are resistant from Plasmodium falciprium (aka malaria).\nThis trait is prevalent amongst the African race and provides resistance and gives us another example of evolution.\nAnother example of a helpful mutation happens in Europeans and European Americans, this is the delta-32 mutation.  Having this mutation provides resistance to the bubonic plague and HIV.  This occurs in your immune cells called macrophages that close the membrane that doesn't allow this bacteria and virus to enter the macrophage.  If you have this mutation, you'll never get HIV/AIDS or Bubonic Plague.\n\nLike it or not:\nEvolution has happened, will happen and will continue to happen.
433542
Where is the genetic revolution headed? Without question, it contains great potential for good in the form of better drugs, better medical care, and improved understanding of how living things work. But there is another side to the revolution.\n\n“We want perfect babies,” says Jeremy Rifkin. “We want perfect plants and animals. We want a better economy. There’s no evil intent here. The road to the Brave New World is paved with good intentions.\n\n“Step-by-step, we are deciding to engineer parts of the genetic code of living things. Two important questions emerge: If we’re going to engineer the genetic code, what criteria does this society establish for determining good and bad, useful and dysfunctional genes? And I would like to know whether there is an institution anyone here would trust with the ultimate authority to decide the genetic blueprints for a living thing?”\n\nThese are questions that deserve answers. Who is better equipped than the Creator of DNA to determine what is a good or a bad gene? He is the one who knows the innermost workings of the genetic code, as is shown by David at Psalm 139:13-16: “You kept me screened off in the belly of my mother. I shall laud you because in a fear-inspiring way I am wonderfully made. Your works are wonderful, as my soul is very well aware. My bones were not hidden from you when I was made in secret, when I was woven in the lowest parts of the earth. Your eyes saw even the embryo of me, and in your book all its parts were down in writing, as regards the days when they were formed and there was not yet one among them.” Would you not prefer to trust Him with the ultimate authority to decide the genetic blueprints for all living things?\n\nFor more info visit http://watchtower.org
19783
TTACGGTCACCAAGCGTG. A mutation is a chemical or physical change that alters the sequence of bases in DNA molecule.Anything that causesmutation is called a MUTAGEN
708384
Go to answers.com and type molecular mutagensis at search you get links go through them.
307678
It affects us at the DNA level by increasing the level of mutations.  These mutations can cause abnormal growths, cancerous cells, & other unpleasant things.\n\nHowever it is possible that some of these mutations could be benificial.
995002
Mutations occur frequently, not like in the cartoons, but small changes happen in chromosomes that are not inherited from the mother or the father.  Some of them cause terrible diseases, others may not cause problems at all.
309255
NO it is not, it is a cellular mutation that can be reversed. To find our more check out the link.
686786
It's possible to have mutations in the DNA that won't change the amino acid sequence. Usually only 2 of the 3 bases of a given codon are necessary to specificy the particular amino acid.\n\nAs a result, a mutation could occur in the DNA which would not change the protein. However, it's still a heritable mutation, and could be used to establish relationships between individuals and species.
186135
Not necessarily. It could be a mutated gene, a side effect of another health issue, or it could be a recessive gene thus making it in fact genetic.
999403
The mechanisms of evolution include mutation.\n\nThe ultimate source of all genetic variation is mutations. They are permanent, transmissible changes to the genetic material (usually DNA or RNA) of a cell, and can be caused by "copying errors" in the genetic material during cell division and by exposure to radiation, chemicals, or viruses. In multicellular organisms, mutations can be subdivided into germline mutations that occur in the gametes and thus can be passed on to progeny, and somatic mutations that often lead to the malfunction or death of a cell and can cause cancer.\n\nNot all mutations are created equal; simple point mutations (substitutions), which comprise the vast majority of genetic variation, usually can only alter the function or level of expression of existing genes. Gene duplications, which may occur via a number of mechanisms, are believed to be the major mechanism for the introduction of new genes; most genes belong to larger "families" of genes derived from a common ancestral gene (two genes from a species that are in the same family are dubbed "paralogs"). Finally, large chromosomal rearrangements (like the fusion of two chromosomes in the chimp/human common ancestor that produced human chromosome 2) almost invariably result in a speciation event.\n\nTherefore it is not likely that all humans will “mutate” equally or at the same time.
880463
there are 2 types of mutation: the ones that happen in DNA and the ones that happen during protein synthesis in RNA. Only the mutations of DNA are heritable, which can pass to off springs during meiosis and gamete formation. RNA mutations are not heritable because RNA is not passed to the off springs. In RNA mutation, all types of mutation (deletion, point mutation, insertion and duplication) happens but because it is in RNA it only affects the making of 1 protein in that one individual.
353863
Like any other mutation.  DNA damage usually, or chromosomal problems such as nondisjunction.  Often p53 will be inactivated by multiple different mechanisms including deletion of the chromosomal region (chr17 in humans I believe), and point mutations of the coding region of the gene.  DNA damage is promoted by loss of repair pathways (due to mutation, etc), but is caused by UV radiation, or IR radiation, or other stress such as chemotherapy drugs, or carcinogens.  DNA damage can occur numerous ways, and there are many different repair pathways.  The damage has to first occur, then be 'missed' by repair pathways, and then the damage has to be at a critical region (ie p53), to promote tumorogenesis.