I have a lab bio question?
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============================================================\nAspergillus niger is a fungus and one of the most common species of the genus Aspergillus. It causes black mold on certain types of fruit and vegetables, and is a common contaminant of food. It is one of the common molds known to produce aflatoxin.\n============================================================\n\n\nClinical Features \n\nIn immunosuppressed hosts: invasive pulmonary infection, usually with fever, cough, and chest pain. May disseminate to other organs, including brain, skin and bone. In immunocompetent hosts: localized pulmonary infection in persons with underlying lung disease. Also causes allergic sinusitis and allergic bronchopulmonary disease. \n\nEtiologic Agent\n\n Aspergillus fumigatus, A. flavus. Less commonly A. terreus, A. nidulans, A. niger. \n\n\nReservoir \n\nUbiquitous in the environment. Found in soil, decomposing plant matter, household dust, building materials, ornamental plants, items of food, and water. \n\nIncidence \n\nNot reportable. Population-based data available for San Francisco suggest a rate of 1-2 per 100,000 per year. \n\n \nSequelae \n\nIf severe granulocytopenia persists, mortality rate can be very high (up to 100% in patients with cerebral abscesses). Patient outcome depends on resolution of granulocytopenia and early institution of effective antifungal drug therapy. \n\n \nTransmission \n\nInhalation of airborne conidia (spores). Nosocomial infection may be associated with dust exposure during building renovation or construction. Occasional outbreaks of cutaneous infection traced to contaminated biomedical devices. \n\nRisk Groups \n\nPersons with severe, prolonged granulocytopenia (e.g., hematologic malignancy, hematopoietic stem cell and solid organ transplant recipients, and patients on high-dose corticosteroids). Rarely, persons with HIV infection. \nSurveillance No national surveillance exists. Active surveillance is being conducted among hematopoietic stem cell and solid organ transplant recipients in selected U.S. hospitals. \n\n \nChallenges \n\nIdentifying modifiable risk factors for disease in immunocompromised persons. Improving understanding of sources and routes of transmission from the environment. Developing sensitive and specific methods for earlier diagnosis.\n \nOpportunities \n\nDevelopment of rapid antigenemia and antigenuria tests and molecular probes may facilitate earlier clinical diagnosis. Availability of improved molecular typing methods may assist in epidemiologic studies.
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some cells to take in more. Cell permiablility.\n\nhttp://www.answers.com/topic/cell-permeability
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Sorry for my previous answer, I was mistaken. \nI believe answer is B. A is correct, but I don't think it is why it affect target tissue for loger time. I don't think C is correct, but it could be. D is certainly wrong and E too. I haven't seen this in a while. Sorry I couldn't be more help.
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Hi!\nI change my field of study from physics to biophysics but still its a great pleasure to solve a problem in physics if you dont mind.\nWish to see your project.\nBye
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Absolutely..Cell Bio is a great area.  I've been dealing with cell biology for a whole lot of years,  and I still find it fascinating.
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hi, i work 4 a book company, and 1 of our regular books is on cell and molecular biology, if you email me, i can probably give you an answer on exactly what ur question is.
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A very good place for answers to questions like this one is \nhttp://users.rcn.com/jkimball.ma.ultranet/BiologyPages/\n\nVery useful for biology students.
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http://www.newton.dep.anl.gov/newton/askasci/1993/biology/bio021.htm
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Intracellular K+ should be about 140 mM (140 mEq/L)\nExtracellular K+ should be about 4 mM (4 mEq/L)\n\nTherefore, the ratio of intracellular to extracellular is about 35:1
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Ah, AP biology class. Let me guess, pureed beef liver and hydrogen peroxide? I hated that lab, but I remember writing the lab report.\n\nYour results do not mean enzymes work harder if there is more substrate around. First, visualize the solution on a molecular level and picture a lone enzyme molecule floating in water with substrate. The probability of an enzyme molecule bumping into a substrate molecule and doing its thing in any given microsecond increases if there are more substrate molecules to bump into!\n\nDid that help?
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Im a microbiologist working in a pathology lab.....its a bit slow at the moment - people are too healthy!
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you use a 3d molecular bio particle scanner.  most of your high end medical research facilities will have one. just call and ask for scanning
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emm...is this your homework??well..it seems that the question is not from you (individually). Hmm..for more understanding, check out this website:\n\nhttp://faculty.clintoncc.suny.edu/faculty/Michael.Gregory/files/Bio%20101/Bio%20101%20Lectures/Biochemistry/biochemi.htm\n\nI hope it solves your dillema, good luck :)
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You can measure it in a number of ways depending on the protein.\n\n-- pH may vary directly with concentration\n-- optical rotation measurements may work for some\n-- adding a dye to the protein and reading on a colorimeter\n-- Another way would be to degrade or react the protein in solution and then to measure the byproducts of the degradation/reaction by something as simple as a titration
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if ur talking about cells... i think it is organelles...not sure
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This is not a pure biology question. Should be bio-chemistry.
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http://www.people.virginia.edu/~rjh9u/IntroBio/bio201/lectures/Lecture06.html\nit's all there for you, outlined and organized. hope this helps!