what is the most effective drugs for staph aureus?
478614
Today, S. aureus has become resistant to many commonly used antibiotics. In the UK, only 2% of all S. aureus isolates are sensitive to penicillin with a similar picture in the rest of the world. The β-lactamase resistant penicillins (methicillin, oxacillin, cloxacillin and flucloxacillin) were developed to treat penicillin-resistant S. aureus and are still used as first-line treatment. First line treatment for MRSA is currently glycopeptide antibiotics (vancomycin and teicoplanin).
991271
MRSA = methicillin-resistant Staphylococcus aureus\n\nWhat is MRSA?\n\nMRSA stands for methicillin-resistant Staphylococcus aureus. It is a type of bacterium commonly found on the skin and/or in the noses of healthy people. Although it is usually harmless at these sites, it may occasionally get into the body (eg through breaks in the skin such as abrasions, cuts, wounds, surgical incisions or indwelling catheters) and cause infections. These infections may be mild (eg pimples or boils) or serious (eg infection of the bloodstream, bones or joints).\n\nThe treatment of infections due to Staphylococcus aureus was revolutionised in the 1940s by the introduction of the antibiotic penicillin.\n\nUnfortunately, most strains of Staphylococcus aureus are now resistant to penicillin. This is because Staphylococcus aureus has 'learnt' to make a substance called ß-lactamase (pronounced beta-lactamase), that degrades penicillin, destroying its antibacterial activity.\n\nSome related antibiotics, such as methicillin and flucloxacillin, are not affected by ß-lactamase and can still be used to treat many infections due to ß-lactamase-producing strains of Staphylococcus aureus. Unfortunately, however, certain strains of Staphylococcus aureus, known as MRSA, have now also become resistant to treatment with methicillin and flucloxacillin.\n\nAlthough other types of antibiotics can still be used to treat infections caused by MRSA, these alternative drugs are usually not available in tablet form and must be administered through a drip inserted into a vein.\n\nWho gets infection with MRSA?\n\nMRSA infections most often occur in patients in hospitals and are rarely seen among the general public. As with ordinary strains of Staphylococcus aureus, some patients harbour MRSA on their skin or nose without harm (such patients are said to be 'colonised'), whereas other patients may develop infections. \n\nSome patients are at increased risk of developing infection. They include those with breaks in their skin due to wounds (including those caused by surgery), indwelling catheters or burns, and those with certain types of deficiency in their immune system, such as low numbers of white cells in their blood.\n\nWhen MRSA spread from an initial site of colonisation to a site where they cause infection in the same patient (eg spread from the colonised nose to a wound), the resulting infection is described as 'endogenous'.\n\nIn addition to causing endogenous infections, MRSA can spread between patients, usually by direct or indirect physical contact. For example, hospital staff attending to a colonised or infected patient may become contaminated or colonised with MRSA themselves (perhaps only briefly). They may then spread the bacteria to other patients with whom they subsequently have contact. These patients may in turn become colonised and/or infected. The spread of MRSA (or for that matter other bacteria) between patients is called cross-infection.\n\nSome strains of MRSA that are particularly successful at spreading between patients may also spread between hospitals, presumably when colonised patients or staff move from one hospital to another. These strains are known as epidemic MRSA (or EMRSA for short). \n\nCan the spread of MRSA be controlled?\n\nThere are several steps that may be undertaken to minimise the spread of MRSA between patients.\n\n\nHospital staff should wash their hands scrupulously, preferably using antibacterial soap and disposable towels.\n\n\nPatients colonised or infected with MRSA may be kept away from other patients by being placed in separate rooms, either alone or with other patients who also have MRSA. Access to such rooms should be restricted to essential personnel.\n\n\nHospital staff should wear gloves and disposable gowns prior to having physical contact with MRSA patients. Before leaving the room, they should discard these safely, and wash their hands.\n\n\nVisitors and carers likely to have a lot
697524
Garlic is the best, most effective broad-spectrum antibiotic, antiviral, antifungal herb known. Echinacea and Garlic are the Herbal Dynamic Duo.  You can mash it up and apply it externally, wrapping the area in saran wrap overnight and/or take it internally.   Here is a link to others:  http://curezone.com/forums/fm.asp?i=356231
102237
tinactin or miconazole.  available at walmart.
786533
Methicillin-resistant Staphylococcus aureus (MRSA) is a specific strain of the Staphylococcus aureus bacterium that has developed antibiotic resistance to all penicillins, including methicillin and other narrow-spectrum β-lactamase-resistant penicillin antibiotics.[1] MRSA was first discovered in the UK in 1961 and is now widespread, particularly in the hospital setting where it is commonly termed a superbug.\n\nMRSA may also be known as oxacillin-resistant Staphylococcus aureus (ORSA) and multiple-resistant Staphylococcus aureus, while non-methicillin resistant strains of S. aureus are sometimes called methicillin-susceptible Staphylococcus aureus (MSSA) if an explicit distinction must be made.\n\nAlthough MRSA has traditionally been seen as a hospital-associated infection, there is currently an epidemic of community-acquired MRSA in the USA. The abbreviations CA-MRSA (community-associated MRSA) and HA-MRSA (hospital-associated MRSA) are now commonly seen in the literature.\n\nStaphylococcus bacteria are extremely common in the environment (and a third of humans world-wide are estimated to carry it on their bodies and are not harmed by it), and is usually not a problem to healthy individuals. Historically, staph infections begin only after the individual has suffered a skin break or open wound. Recently, due to institutional overuse of antibiotics, strains of staphylococcus aureus have developed drug resistance. If an individual has a weak immune response to the bacteria, a MRSA infection can result even with no apparent open wound. The symptoms can range from skin boils to necrotizing fasciitis, popularly known as flesh-eating disease. MRSA infections are typically combatted with vancomycin, however, vancomycin-resistant staphylococcus has recently appeared.\n\nAlthough Noskin and others report that a patient infected with MRSA is five times more likely to die than other patients; it is not clear that patients who are infected with MRSA have an increased death rate. Wyllie et al. report a death rate of 34 percent within 30 days among patients infected with MRSA, while among MSSA patients the death rate was similar at 27%\n\nVancomycin and teicoplanin are glycopeptide antibiotics used to treat MRSA infections. Teicoplanin is a structural congener of vancomycin that has a similar activity spectrum but a longer half-life (t½). Both drugs have low oral absorption and must therefore be administered intravenously for systemic infections. One of the problems with vancomycin is not just that its route of administration is inconvenient, but also that it is inferior in terms of its efficacy compared to antistaphylococcal penicillins.\n\nSeveral new strains of MRSA have been found showing antibiotic resistance even to vancomycin and teicoplanin; those new evolutions of the MRSA bacteria are dubbed vancomycin intermediate-resistant Staphylococcus aureus (VISA). Linezolid, quinupristin/dalfopristin, daptomycin, tigecycline are more recent additions to the therapeutic arsenal, generally reserved for severe infections which do not respond to glycopeptides. Less severe infections may be treated by oral agents including: linezolid, rifampicin+fusidic acid, pristinamycin, co-trimoxazole (trimethoprim+sulfamethoxazole), doxycycline, and clindamycin.
232386
Pfizer\n\nhttp://www.pfizerch.com/default.aspx
579135
Sudafed will not help.  It is a decongestant.  What you need is an antihistamine like Benadryl (diphenhydramine) or Claritin (loratadine).\n\nI'm not sure why, but my pharmacist strongly encouraged me to NOT use Visine or similar products.  If you want an effective, quick-acting eye drop, you could try Patanol, although it is only available by prescription.
1072037
1) Aspirin\n\n2)Penicillin\n\n3)Small Pox Vaccine\n\n4)Viagra - just kidding\n\n5)Hand Washing\n\nThe greatest I suspect is small pox vaccine.  The reason that I say that is prevention is better than a cure.  Remember the saying:  An ounce of prevention is worth a pound of cure.
565505
I use the orange flavored Listerine.  Works pretty good and it gets the bacteria out of the sore.
324100
Go to herbalhealer.com and check out colloidal silver,olive leaf extract,grapefruit seed extract,& oregano oil. These are very potent anti-viral/bacterials and I have used them for years. Read the testimonials of people who have had cures with these and other natural products.
312492
well,here is a useful brief answer:\nA-Tetracycline:\n# mode of action:\n1.Bacteriostatic.\n2.Prevents bonding  of tRNA to mRNA on ribosome.\n# uses:\n1.Treats intestinal amebiasis.      \n2.Effective against Rickettsia and Chlamydia.     \n3.Against syphilis and gonorrhea.  \n\nB-Streptomycin:\n# mode of action:\n1.Bacteriostatic \n2.Inhibits peptide bond formation.  \n# uses:\n1.Tuberculosis.\n2.Used for tularemia,plague.  \n2.Urinary tract infections.  \n3.Enteric infections.  \n4.Nosocomial infections.  \n5.Against penicillin-resistant gonorrhea       \n\nC-Polymyxin B:\n# mode of action:\n1.Bacteriocidal  \n2.Causes disorientation of lipoprotein constituents resulting in a distortion.     \n# uses:\n1.Against Pseudomonal infection.      \n2.Against Gram -ve in wounds,abrasions,and burns.\n3.Against E.Coli infection.\n\nD-Pencillin:\n# mode of action:work by inhibiting the formation of peptidoglycan cross links in the bacterial cell wall.\n# uses:\n1.STD:like syphilis & gonorrhea.\n2.Cardiac infection:Bacterial Endocarditis.  \n3.Brain infectins:Meningitis,Encephalitis.\n4.Respiratory infections:Pneumonia,Lung abscess.\n5.Miscellanous:Tonsilitis,Pharyngitis.\n6.Skin infections.\n7.Prophylasis:Rheumatic Fever .\n8.Septicemia in children.\n\nE-Cephalosporins:\n# mode of action:work by inhibiting the formation of peptidoglycan cross links in the bacterial cell wall.\n# uses:\n1.Against Staphylococcal & Streptococcal infections.\n2.Against Escherichia coli,Klebsiella pneumoniae and Proteus mirabilis.\n3.Hospital-acquired infections.
860958
He probably has MRSA. There are several antibiotics that can be used. Don't worry. Take him to a Children's Hospital if your Dr. claims that he cannot help.
724315
MRSA stands for methicillin-resistant Staphylococcus aureus. It is a type of bacterium commonly found on the skin and/or in the noses of healthy people. Although it is usually harmless at these sites, it may occasionally get into the body (eg through breaks in the skin such as abrasions, cuts, wounds, surgical incisions or indwelling catheters) and cause infections. These infections may be mild (eg pimples or boils) or serious (eg infection of the bloodstream, bones or joints).
1020575
it's MRSA -- methcillin resistant staph aureus -- it's a bacterial infection that is resistant to one of the most potent anti-biotic, methcillin. you have to be hospitalized for this, but there is a cure. doctors now are using Vancomycin - IV for treatment of MRSA.
539033
For me its XENICAL. It has no side effect and really effective.
1155500
multi drug resistant bacteria.ex---psuedomonas staphylococcus
1013196
This might sound silly but the best thing is Sudocrem which is a zinc cream that is used for babies nappy rash.\nFrom experience it is brill for sunburn and sore skin\nIt protects and heals\nOr any own brand zinc cream (eg Boots own)