What are gorters in thyroids?
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A goitre (or goiter) (Latin struma) is a swelling in the neck (just below adam's apple or larynx) due to an enlarged thyroid gland. They are classified in different ways:\n\nA "diffuse goitre" is a goitre that has spread through all of the thyroid (and is contrasted with a "simple goitre", "single thyroid nodule" and "multinodular goitre". \n"Toxic goitre" refers to goitre deriving from inflammation, neoplasm, or malfunction of the thyroid, while "nontoxic goitre" refers to all other types (such as that caused by lithium or an autoimmune reaction.) \n\nCauses\nThe most common cause for goitre in the world is iodine deficiency (E01). Other causes are:\n\nHashimoto's thyroiditis (E06.3) \nGraves-Basedow disease (E05.0) \nJuvenile goitre due to congenital hypothyroidism (E03.0) \nNeoplasm of the thyroid \nThyroiditis (acute, chronic) (E06) \nSide-effects of pharmacological therapy (E03.2)
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Having a normally functioning thyroid gland.\neu=normal\nthyroid=take a wild guess\nThe previous guy is talking about Sick Euthyroid Syndrome. You are asking about Clinically Euthyroid.  There's a huge difference.(My roommate is a nurse)
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This going to be a long post but will answer the bulk of your questions. Hope it helps. This is taken from a powerpoint lecture on Thyroiditis so the formatting may be off.\n\nThyroiditis\n\nInflammation of the Thyroid Gland\n  \n\nThyroiditis is an inflammation (not an infection) of the thyroid gland. Several types of thyroiditis exist and the treatment is different for each.\n\n\nHashimoto's Thyroiditis. Hashimoto's Thyroiditis (also called autoimmune or chronic lymphocytic thyroiditis) is the most common type of thyroiditis. It is named after the Japanese physician, Hakaru Hashimoto, that first described it in 1912. The thyroid gland is always enlarged, although only one side may be enlarged enough to feel. During the course of this disease, the cells of the thyroid becomes inefficient in converting iodine into thyroid hormone and "compensates" by enlarging (for a review of this process see our function page). The radioactive iodine uptake may be paradoxically high while the patient is hypothyroid because the gland retains the ability to take-up or "trap" iodine even after it has lost its ability to produce thyroid hormone. As the disease progresses, the TSH increases since the pituitary is trying to induce the thyroid to make more hormone, the T4 falls since the thyroid can't make it, and the patient becomes hypothyroid. The sequence of events can occur over a relatively short span of a few weeks or may take several years.\n\nTreatment is to start thyroid hormone replacement. This prevents or corrects the hypothyroidism and it also generally keeps the gland from getting larger.\n\nIn most cases the thyroid gland will decrease in size once thyroid hormone replacement is started.\n\nThyroid antibodies are present in 95% of patients with Hashimoto's Thyroiditis and serve as a useful "marker" in identifying the disease without thyroid biopsy or surgery.\n\nThyroid antibodies may remain for years after the disease has been adequately treated and the patient is on thyroid hormone replacement.\n\n\nDe Quervain's Thyroiditis. De Quervain's Thyroiditis (also called subacute or granulomatous thyroiditis) was first described in 1904 and is much less common than Hashimoto's Thyroiditis. The thyroid gland generally swells rapidly and is very painful and tender. The gland discharges thyroid hormone into the blood and the patients become hyperthyroid; however the gland quits taking up iodine (radioactive iodine uptake is very low) and the hyperthyroidism generally resolves over the next several weeks.\n\nPatients frequently become ill with fever and prefer to be in bed.\n\nThyroid antibodies are not present in the blood, but the sedimentation rate, which measures inflammation, is very high.\n\nAlthough this type of thyroiditis resembles an infection within the thyroid gland, no infectious agent has ever been identified and antibiotics are of no use.\n\nTreatment is usually bed rest and aspirin to reduce inflammation.\n\nOccasionally cortisone (steroids) (to reduce inflammation) and thyroid hormone (to "rest" the thyroid gland) may be used in prolonged cases.\n\nNearly all patients recover and the thyroid gland returns to normal after several weeks or months.\n\nA few patients will become hypothyroid once the inflammation settles down and therefore will need to stay on thyroid hormone replacement indefinitely.\n\nRecurrences are uncommon.\n\n\nSilent Thyroiditis. Silent Thyroiditis is the third and least common type of thyroiditis. It was not recognized until the 1970's although it probably existed and was treated as Graves' Disease before that. This type of thyroiditis resembles in part Hashimoto's Thyroiditis and in part De Quervain's Thyroiditis. The blood thyroid test are high and the radioactive iodine uptake is low (like De Quervain's Thyroiditis), but there is no pain and needle biopsy resembles Hashimoto's Thyroiditis. The majority of patie
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have your thyroid checked out
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I think the first diagnosis is not related to thyroid problem.\nTapeworm can result in changes in neck lymph node (cervical node).\nThyroid nodule can be benign hyperplasia, adenoma or carcinoma (cancer).  \nBlood tests can be helpful in the work up along with history and physical exam.\nThyroid scan (like X-ray study) and/or ultrasound may be necessary.  A quick and relatively non-invasive fine needle aspiration of the thyroid nodule can also be helpful to determine whether the nodule is cancer or not.\nEndocrinologist normally is experienced in dealing with thyroid nodule.\nGood luck.
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If you suspect that you have hyperthyroidism, you should really see a doctor.  You shouldn't depend on the looney-tunes here on Yahoo! Answers.  But if you just want background information:\n\nHyperthyroidism:\n\nCauses:  Hyperthyroidism or thyrotoxicosis occurs when the thyroid releases too many of its hormones over a short (acute) or long (chronic) period of time. Many diseases and conditions can cause this problem, including:\n\n    * Graves' disease\n    * Non-cancerous growths of the thyroid gland or pituitary gland\n    * Tumors of the testes or ovaries\n    * Inflammation (irritation and swelling) of the thyroid due to viral infections or other causes\n    * Ingestion of excessive amounts of thyroid hormone\n    * Ingestion of excessive iodine\n\nGraves' disease accounts for 85% of all cases of hyperthyroidism.\n\nSymptoms:\n    * Weight loss\n    * Increased appetite\n    * Nervousness\n    * Restlessness\n    * Heat intolerance\n    * Increased sweating\n    * Fatigue\n    * Frequent bowel movements\n    * Menstrual irregularities in women\n    * Goiter (visibly enlarged thyroid) may be present\n\nTreatment:  Treatment varies depending on the cause of the condition and the severity of symptoms. Hyperthyroidism is usually treated with antithyroid medications, radioactive iodine (which destroys the thyroid and thus stops the excess production of hormones), or surgery to remove the thyroid.\n\nIf the thyroid must be removed with radiation or surgery, replacement thyroid hormones must be taken for the rest of the person's life.\n\nBeta-blockers like propranolol are used to treat some of the symptoms including rapid heart rate, sweating, and anxiety until the hyperthyroidism can be controlled.\n\nP.S.  I'm not a doctor.  All of this information is from publicly available sources on the internet.
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Thyroid adenomas are usually benign. If the growth is not removed and is large enough, it will cause hyperthyroidism.  The symptoms can often be treated successfully with anti-thyroid drugs.  However, if the drugs do not work, surgery is recommended.\n\nSymptoms of hyperthyroidism are:\nnervousness\nmoodiness\nweakness\ntiredness \nfast or irregular heart beat\nhand tremors\nrespiratory difficulties\nprofuse perspiration\nred, itchy skin\nfrequent diarrhea \nhair loss\nweight loss
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Try http://thyroid.about.com/blthyroid.htm \nThis site has a ton of good information about thyroid disease.
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What is hyperthyroidism? \n\nHyperthyroidism develops when the thyroid gland makes too much thyroid hormone. Thyroid hormone controls your metabolism—how your body turns food into energy—and influences your heart rate, digestion, muscle and bone strength, and cholesterol levels. When you have too much thyroid hormone, all of your body's functions speed up. \n\nWhat causes hyperthyroidism? \n\nGraves' disease, also known in Europe as von Basedow's disease, is the most common cause of hyperthyroidism. Graves' disease causes the thyroid gland to produce too much thyroid hormone. Graves' disease, like many thyroid problems, often runs in families. \n\nHyperthyroidism can also be caused by thyroid nodules—growths in the thyroid gland—and by thyroiditis, which is inflammation of the thyroid gland. \n\nThis topic mostly focuses on hyperthyroidism caused by Graves' disease. However, many of the symptoms of hyperthyroidism are similar even when the causes are different.\n\nWhat are the symptoms of hyperthyroidism? \n\nYou may have hyperthyroidism if you:\n\nFeel nervous, moody, weak, or tired. \nHave hand tremors; have a fast or irregular heartbeat; or have trouble breathing, even when you are resting. \nAre sweating a lot, and have warm, red skin that may be itchy. \nHave frequent bowel movements. \nHave fine, soft hair that is falling out. \nLose weight even though you are eating normally or more than usual. \n\nHyperthyroidism symptoms are different depending on how old you are. For example, older adults may lose weight, not feel like eating, have an irregular heartbeat, or have chest pain. Symptoms can be easy to spot in some people and harder to notice in others.\n\nHow is hyperthyroidism diagnosed? \n\nDoctors diagnose hyperthyroidism by asking questions about your medical history, doing a physical exam, and ordering blood tests that tell how much thyroid hormone your body is making. \n\nHow is hyperthyroidism treated? \n\nThere are three treatments for hyperthyroidism. Antithyroid medication and radioactive iodine are the ones doctors use most often. Rarely, a person may have surgery. One or more of these treatments will lower your thyroid hormone levels and make you feel better. Once you have been treated for hyperthyroidism, you will need to have your thyroid hormone levels checked regularly, because there is always a chance that hyperthyroidism will come back. \n\nIf you do not get treatment, hyperthyroidism can lead to heart problems, bone problems, and thyroid storm, a life-threatening condition that occurs when the thyroid gland releases a large amount of thyroid hormone in a short period of time.
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Have your thyroid evaluated! The thyroid can screw with all the hormones in your body, and make strange things happen. Go to the doctor and get a complete metabolic and hormone profile. That will zero in on the problem, adn it can be corrected.
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d\nHypothyroidism \n\n\n \n Day One\n\n What Is It?\n\n How Do I Know I Have It?\n\n What Is the Treatment?\n\n 10 Questions to Ask Your Doctor\n \n \n Get the WebMD Daily Newsletter\n \n\nWhat Is It?\n\nHypothyroidism is an underactive thyroid gland, meaning the gland doesn't produce enough thyroid hormones. The thyroid gland is a butterfly-shaped organ located in your neck. It produces, stores, and releases hormones that influence a variety of body functions, including your body's metabolism, oxygen use, growth, and development.\n\nIf you have hypothyroidism, your body's processes slow down.\n\nFor example, a fetus or baby with severe hypothyroidism can develop a condition called cretinism. The lack of growth-inducing hormones results in mental retardation, very short stature, and muscular abnormalities. When children produce too little thyroid hormone, their muscles, bones, brain, and reproductive organs do not develop properly. Nowadays, testing can identify and prevent these disorders.\n\nSevere hypothyroidism causes a problem called myxedema, especially in the elderly, characterized by dry, thick skin and other symptoms of hypothyroidism, but also slowing of mental functioning, reduced functioning of the heart and lungs, and an increased risk of death from infections.\n\nMilder forms of the condition are most common.\n\nHypothyroidism has a number of potential causes, including overtreatment of hyperthyroidism and autoimmune disorders, in which the body's immune system turns on itself and destroys thyroid gland tissue. It can also occur as a side effect of drugs being taken for other, unrelated conditions. A common genetic cause of hypothyroidism is called Hashimoto's thyroiditis.\n\nThe condition generally affects women over age 60, although 1 of every 4,000 infants is also born with hypothyroidism.\n\nAlthough some cases of hypothyroidism are not curable, most cases can be controlled by medication.
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Common questions in healthcare. The answer is that shellfish collect iodine and hold onto it like a sponge holds water. Until iodized salt came around, those folks in the midwest, far from any salt-water shellfish, had problems with not having enough iodine in their diet, so they would end up with goiters. If you eat shellfish, you'll get the same reaction to medical iodine.
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Hypothyroidism is a condition in which the body lacks sufficient thyroid hormone. Since the main purpose of thyroid hormone is to "run the body's metabolism", it is understandable that people with this condition will have symptoms associated with a slow metabolism. Over five million Americans have this common medical condition. In fact, as many as ten percent of women may have some degree of thyroid hormone deficiency.   Hypothyroidism is more common than you would believe...and, millions of people are currently hypothyroid and don't know it! \nThere are two fairly common causes of hypothyroidism. The first is a result of previous (or currently ongoing) inflammation of the thyroid gland which leaves a large percentage of the cells of the thyroid damaged (or dead) and incapable of producing sufficient hormone. The most common cause of thyroid gland failure is called autoimmune thyroiditis (also called Hashimoto's thyroiditis), a form of thyroid inflammation caused by the patient's own immune system. The second major cause is the broad category of "medical treatments".  As noted on a number of our other pages, the treatment of many thyroid conditions warrants surgical removal of a portion or all of the thyroid gland. If the total mass of thyroid producing cells left within the body are not enough to meet the needs of the body, the patient will develop hypothyroidism. Remember, this is often the goal of the surgery as seen in surgery for thyroid cancer. But at other times, the surgery will be to remove a worrisome nodule, leaving half of the thyroid in the neck undisturbed. Sometimes (often), this remaining thyroid lobe and isthmus will produce enough hormone to meet the demands of the body. For other patients, however, it may become apparent years later that the remaining thyroid just can't quite keep up with demand. Similarly, goiters and some other thyroid conditions can be treated with radioactive iodine therapy. The aim of the radioactive iodine therapy (for benign conditions) is to kill a portion of the thyroid to [1] prevent goiters from growing larger, or [2] producing too much hormone (hyperthyroidism). Occasionally, (often?) the result of radioactive iodine treatment will be that too many cells are damaged so the patient often becomes hypothyroid a year or two later. This is O.K. and usually greatly preferred over the original problem. There are several other rare causes of hypothyroidism, one of them being a completely "normal" thyroid gland which is not making enough hormone because of a problem in the pituitary gland. If the pituitary does not produce enough Thyroid Stimulating Hormone (TSH) then the thyroid simply does not have the "signal" to make hormone, so it doesn't.\nSymptoms of Hypothyroidism\n\nFatigue\nWeakness\nWeight gain or increased difficulty losing weight\nCoarse, dry hair \nDry, rough pale skin\nHair loss \nCold intolerance (can't tolerate the cold like those around you)\nMuscle cramps and frequent muscle aches\nConstipation\nDepression\nIrritability\nMemory loss\nAbnormal menstrual cycles\nDecreased libido\n\nEach individual patient will have any number of these symptoms which will vary with the severity of the thyroid hormone deficiency and the length of time the body has been deprived of the proper amount of hormone. Some patients will have one of these symptoms as their main complaint, while another will not have that problem at all and will be suffering from a different symptom. Most will have a combination of a number of these symptoms. Occasionally, some patients with hypothyroidism have no symptoms at all, or they are just so subtle that they go unnoticed.  Note:   Although this may sound obvious, if you have these symptoms, you need to discuss them with your doctor and probably seek the skills of an endocrinologist.  If you have already been diagnosed and treated for hypothyroidism and you continue to have any or all of thes
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Understanding Thyroid Problems -- Treatment\n\nWebMD Medical Reference \n\nThe Basics | Symptoms | Detection & Treatment\n\nHow Do I Know If I Have Them?\n\nA doctor can diagnose hyperthyroidism and hypothyroidism by testing the levels of thyroid hormones in your blood.  Doctors measure hormones secreted by the thyroid itself, as well as thyroid-stimulating hormone (TSH), a chemical released by the pituitary gland that triggers hormone production in the thyroid.\n\nWhen you are hypothyroid, higher quantities of TSH are circulating in your blood as your body attempts to foster increased production of thyroid hormones.  The reverse is true with hyperthyroidism, in which TSH levels are below normal and circulating thyroid-hormone levels are high.\n\nTo determine the cause of hyperthyroidism, doctors often use radioactive iodide uptake tests, which track the amount of iodide absorbed by the thyroid gland. Iodide, obtained from the foods we eat, is a key ingredient in the manufacture of thyroid hormone, so the amount of iodide the thyroid absorbs is a reliable indicator of how much hormone the gland is producing.  For this test, you must swallow a small amount of radioactive iodide in liquid or capsule form.  After a predetermined wait, the doctor places an instrument over your neck to measure how much of the radioactive iodide has gathered in your thyroid.\n\nIf the results of this test suggest that the gland is collecting excessive amounts of iodide, the doctor may then conduct a radioactive iodide uptake scan.  In this test, the doctor uses a special film to create a picture that shows the exact location of the radioactive iodide in your thyroid gland.  The scan will reveal, for example, if the iodide is collecting in adenomas, indicating that the nodules are responsible for the excess hormone.  If the scan shows that the iodide is spread equally throughout the tissue, the whole thyroid is involved in the excess production.\n\nSome doctors believe that blood tests may not be sensitive enough to detect milder forms of hypothyroidism.  Instead, they advocate monitoring your body's basal (resting) temperature.  To track your basal temperature accurately, you must closely follow certain guidelines:  Shake the thermometer below 95° F at night and place it where you can reach it without getting out of bed.  The following morning, before you get out of bed, take your temperature via your armpit for 10 minutes while staying as still as possible.  Keep records of your temperature for at least three days.  (Women should do this during the first two weeks of the menstrual cycle, as their basal temperature may rise during the latter half.)  Normal basal body temperatures fall between 97.4° F and 97.8° F.  If your basal temperature is consistently low, you could be mildly hypothyroid.\n\nIf you have one or more adenomas, your doctor will want to keep careful records of when they were first found and how they develop, since not all adenomas produce excess thyroid hormone. In fact, most of these nodules are not malignant, especially if they remain the same size over long intervals.  (Cancerous tissue, by contrast, will undergo noticeable growth.)  Nodules that appear suddenly are typically fluid-filled cysts and are often benign.  They can be evaluated with a noninvasive ultrasound exam.  If blood tests indicate that the nodules are producing excess thyroid hormone, and if you have other symptoms, your doctor will treat you for hyperthyroidism.\n\nIn any case, you should receive periodic checkups if you have a nodule on your thyroid gland, since you may become hyperthyroid in the future.  If your blood tests show elevated hormone levels, your doctor may recommend other tests, including radioactive iodide uptake tests and scans that indicate whether the nodules are "hot" or "cold."  Hot nodules, or those that are actively trapping iodide and producing too much thyroid hormone, are rarely cancerous.  But cold nodules -
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A goiter is an enlargement of the thyroid gland. Persons with enlarged thyroids may have normal function of the gland (euthyroidism), thyroid deficiency (hypothyroidism), or overproduction of the hormones (hyperthyroidism). Goiter may be congenital or acquired, endemic or sporadic.\nThe goiter often results from increased pituitary secretion of thyrotropic hormone in response to decreased circulating levels of thyroid hormones. Thyroid enlargement may also result from infiltrative processes that may be inflammatory or neoplastic. Goiter in patients with thyrotoxicosis is caused by thyrotropin receptor-stimulating antibodies.\n\n\n Figure 561-1 Congenital goiter in infancy. A, Large congenital goiter in an infant born to a mother with thyrotoxicosis who had been treated with iodides and methimazole during pregnancy. B, A 6-wk-old infant with increasing respiratory distress and cervical mass since birth. The operation revealed a large goiter that almost completely encircled the trachea. Notice the anterior deviation and posterior compression of the trachea. Partial thyroidectomy completely relieved the symptoms. It is apparent why a tracheostomy is not adequate treatment for these infants. The cause for the goiter was not found.\n561.1 Congenital Goiter\nCongenital goiter is usually sporadic and may result from a fetal thyroxine (T4 ) synthetic defect or the administration of antithyroid drugs or iodides during pregnancy for the treatment of thyrotoxicosis. Goitrogenic drugs and iodides cross the placenta and at high doses may interfere with synthesis of thyroid hormone, resulting in goiter and hypothyroidism in the fetus. The concomitant administration of thyroid hormone with the goitrogen does not prevent this effect, because insufficient amounts of T4 cross the placenta. Iodides are included in many proprietary preparations used to treat asthma; these preparations must be avoided during pregnancy because they have often been a cause of unexpected congenital goiter. Amiodarone, an antiarrhythmic drug with a 37% iodine content, has also caused congenital goiter with hypothyroidism. Even when the infant is clinically euthyroid, there may be retardation of osseous maturation, low levels of T4 , and elevated levels of thyroid-stimulating hormone (TSH). In women with Graves disease on antithyroid drugs, these effects can occur when the mother takes only 100–200?mg of propylthiouracil/24?hr; all such infants should undergo thyroid studies at birth. Administration of thyroid hormone to affected infants may be indicated to treat clinical hypothyroidism, to hasten the disappearance of the goiter, and to prevent brain damage. Because the condition is rarely permanent, thyroid hormone may be safely discontinued after the antithyroid drug has been excreted by the neonate, usually after a week.\nEnlargement of the thyroid at birth may occasionally be sufficient to cause respiratory distress that interferes with nursing and may even cause death. The head may be maintained in extreme hyperextension. When respiratory obstruction is severe, partial thyroidectomy rather than tracheostomy is indicated ( Fig. 561–1 ).\nGoiter is almost always present in the congenitally hyperthyroid infant. These goiters usually are not large; the infant manifests\n\n1882\nclinical symptoms of hyperthyroidism, and the mother often has a history of Graves disease (see Chapter 562.1 ). TSH receptor-activating mutations are also a recognized cause of congenital goiter. \nWhen no causative factor is identifiable, a defect in synthesis of thyroid hormone should be suspected. Neonatal screening programs find congenital hypothyroidism caused by such a defect in 1/30,000–50,000 live births. If the infant is hypothyroid, it is advisable to treat immediately with thyroid hormone and to postpone more detailed studies for later in life. Because these defects are transmitted by recessive genes, a precise diagnosis is helpful for genetic counseling. Monitoring subsequent
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I think you should ask your doctor about poly cystic ovarian disease and if he will check your thyroid.  Good Luck.
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Hypothyroidism Symptoms \n\n   1.  Insufficient production of thyroid hormones.\n   2. A pathological condition resulting from severe thyroid insufficiency, which may lead to cretinism or myxedema.\n\n\nHypothyroidism\n\nA disorder in which the thyroid gland produces too little thyroid hormone causing a decrease in the rate of metabolism with associated effects on the reproductive system. Symptoms include fatigue, difficulty swallowing, mood swings, hoarse voice, sensitivity to cold, forgetfulness, and dry/coarse skin and hair. \n\nHypothyroidism FAQS :\n\nWhat are thyroid hormones?\nThyroid hormones are produced by the thyroid gland. This gland is located in the lower part of the neck, below the Adam's apple. The gland wraps around the windpipe (trachea) and has a shape that is similar to a butterfly - formed by two wings (lobes) and attached by a middle part (isthmus).\n\nThe thyroid gland uses iodine (mostly available from the diet in foods such as seafood, bread, and salt) to produce thyroid hormones. The two most important thyroid hormones are thyroxine (T4) and triiodothyronine (T3), which account for 99.9% and 0.1% of thyroid hormones present in the blood respectively. However, the hormone with the most biological activity is T3. Once released from the thyroid gland into the blood, a large amount of T4 is converted into T3 - the active hormone that affects the metabolism of cells.\n\nThyroid hormone regulation- the chain of command\nThe thyroid itself is regulated by another gland that is located in the brain, called the pituitary. In turn, the pituitary is regulated in part by the thyroid (via a "feedback" effect of thyroid hormone on the pituitary gland) and by another gland called the hypothalamus.\n\nThe hypothalamus releases a hormone called thyrotropin releasing hormone (TRH), which sends a signal to the pituitary to release thyroid stimulating hormone (TSH). In turn, TSH sends a signal to the thyroid to release thyroid hormones. If a disruption occurs at any of these levels, a defect in thyroid hormone production may result in a deficiency of thyroid hormone (hypothyroidism).