what is the chemical component of eggplant that stimulates the blood glucose level?
473061
It is suspected to be a phospholipid, but hasnt been determined yet.  Nor has it been determined if this effect is real, as far as I know
263027
Glucose is oxidized with bromine.
1064113
Sugar is used by the body during many of life's processes. It is just another chemical that exists in food and is used by the body. A diabetic is not making enough of the chemical that the body uses to control sugar use, called insulin. The level of sugar goes out of control and can cause diseases shock, coma and death. The diabetic has to control the level of sugar be regular eating habits and blood monitoring. When needed they can take insulin to help control blood sugar.
1103095
AVANDIA, ACTOS,GLUCOPHAGE, GLUCOVANCE,  \nHUMALIN, HUMALOG
439402
Steroids are certainly known to raise blood sugar level in diabetics. However, the absorption of the steroids into the blood stream with a nasal spray is lower than that with oral steroids. So whether it will raise your Blood sugar level(BSL) will depend upon the dose and frequency of usage of the nasacort spray. It would be best to monitor your BSL with a home glucometer, if u have one, and see if it is indeed causing that- in which case the dose of your medicines/insulin may need to be adjusted. Else it would be best to ask your doc regarding the same.
981401
Oatmeal and fresh fruit.  Can't beat it.  Complex carbs for long term energy (oatmeal), and simple carbs (fruit) for short term energy spikes.  Plus it can help reduce your cholesterol
151155
All Sugars whether aldose or ketose, comes under the category of carbohydrates. Giving Positive Molisch test is a characteristic property of all carbohydrate (e.g sugars, potatotes). That is why sugars give a positive molisch test.
370822
If you are not diabetic, and you don't faint in the streets from too low blood glucose, then your problem -- whatever it is -- would not seem to be immediately life threatening. And the erratic changes are not likely due to insulin. Here's why. \n\n=======\nbackground\n\nMetabolic control in the human body is fairly simple in principle. Just involved and interconnected, and only partially understood even now. \n\nYou absorb glucose from food in the intestines (after breaking down complex carbohydrates so it's possible to absorb them) and it goes into the blood. From there it can be absorbed by body cells and insulin is required for about 2/3 of them to do the absorption. Only two types of cells storage significant amounts of glucose internally (liver cells and muscle cells). As the glucose is used as fuel within cells, they either convert internal stores back to glucose, or absorb more from the blood. Glucose absorption is controlled in the 2/3 of the total by insulin, which is required for those cells to absorb glucose. When the beta cells (which are the only source of insulin) see high blood glucose, they release insulin. In non-diabetics, this causes those 2/3 of cells to absorb glucose, the blood level to drop, and the release of insulin to cease. \n\nWhen glucose levels are low, and so insulin levels are as well, liver cells (but not muscle cells) to release stored insulin into the blood. The result is that blood glucose is kept at a more or less constant level, save just after meals when it rises somewhat. \n\nNote that insulin, and exercise, are the only control for reducing glucose levels. There are several situations in which glucose levels are raised by opposing hormones. All of the stress hormones (adrenalin, for instance) cause the liver to output stored glucose and so to raise blood glucose levels. Another hormone, glucagon, also causes the liver to output stored glucose to the blood, and it's sometimes carried by those whose glucose levels are characteristically too low. Infections also usually cause glucose levels to rise. \n\nSo, \n\nfood and any of several signals to the liver cause blood glucose to rise. \n\ninsulin and exercise cause blood glucose to go down; glucose is strongly conserved and is never lost in normal circumstances. \n\nBlood glucose levels vary somewhat in the normal usual course of things. The fluctuations you note in your case, if abnormal, are most likely due to one of these controls.
534998
sugar.. and sweet stuff.. mainly saccharides...
378337
Glucose is a hyperglycemic it increases the BGL. It is used for diabetic hypo. In normal people this increase in glucose level stimulates insulin so the level falls. Its a balance between insulin, lowering blood glucose and other hormones like glucagon, adrenalin,steroids which increase the glucose level. Insulin is absent or low or ineffective in Diabetics so there is hyperglycemia. in diabetes--hypo occurs when you take the pill or shot and dont have food. the blood sugar falls dangerously low and the patient faints. The brain needs glucose 24*7. if there isnt glucose for 8 min or so there will be brain damage. In that case u give glucose IV and/or glucagon.
888992
Fruit and vegs r verry good and healty but u need fibbers to so milcschakes good to.\nJust not to mutch sugars and all the E123,E....,E.....\nthats in pakket food.
621410
c - glucagon actually triggers a higher blood sugar level
456017
Both Tollen's and Benedict's reagents are used for the detection of reducing sugars/aldehydes (such as glucose).  Sucrose is a disaccharide consisting of an aldehyde (glucose) and a ketone (fructose).  \n\nIn Benedict's reagent, sucrose is not broken down into its monosaccharide components and thus elicits no reaction/color change.\n\nIn Tollen's reagent, sucrose is broken down into glucose and fructose monomers, but this process occurs very slowly.  Thus you will eventually see the reduction of silver I to silver metal (as observed visually by "mirror" formation on the reaction container) as sucrose is broken down and glucose is liberated allowing it to react.
193015
Disaccharidases are produced in the brush border of the intestinal mucosa, so, the best answer appears to be 'B' - intestinal juice.\n\nGood luck!\n\nP.S. you have several of these questions on here.  The questions are either poorly conceived and/or the answers are poorly conceived - that is, things presented are not truly consistent with the information in medical textbooks (e.g. Guyton's "Medical Physiology" or Gropper's "Advanced Nutrition and Human Metabolism"), therefore you need to refer to your textbook or your class notes to see how this info. is being 'simplified'.  From these sources, hopefully, you will find the answers that best suit the teacher's thoughts.
836692
glucose, catalyzed in humans by ptyalin.
1094036
J and gand..are both absolutely correct. If you are diabetic and checking your sugar and you are having difficulty controling it you need to see your diabetes management nurse or physician. Now there are a few other things that can also cause a rise in am sugars that would be the use of corticosteroids for the control of inflamation or endocrin disorders. Corticosteroids can actually cause a non diabetic to require the use of diabetic medications during the course of there treatment (that cortisol thing going on there)
350702
(m)\n\nHerbs for Diabetes\nSince antiquity, diabetes has been treated with plant medicines. Recent scientific investigation has confirmed the efficacy of many of these preparations, some of which are remarkably effective. Only those herbs that appear most effective, are relatively non-toxic and have substantial documentation of efficacy are covered here.\n\nPterocarpus marsupium (Indian Kino, Malabar Kino, Pitasara, Venga)\nThe tree is the source of the Kino of the European pharmacopeas. The gum-resin looks like dried blood (Dragon's blood), much used in Indian medicine. This herb has a long history of use in India as a treatment for diabetes. The flavonoid, (-)-epicatechin, extracted from the bark of this plant has been shown to prevent alloxan-induced beta cell damage in rats.\n\nBoth epicatechin and a crude alcohol extract of Pterocarpus marsupium have actually been shown to regenerate functional pancreatic beta cells. No other drug or natural agent has been shown to generate this activity.\n\nBitter Melon (Momordica charantia)\nBitter melon, also known as balsam pear, is a tropical vegetable widely cultivated in Asia, Africa and South America, and has been used extensively in folk medicine as a remedy for diabetes. The blood sugar lowering action of the fresh juice or extract of the unripe fruit has been clearly established in both experimental and clinical studies.\n\nBitter melon is composed of several compounds with confirmed anti-diabetic properties. Charantin, extracted by alcohol, is a hypoglycaemic agent composed of mixed steroids that is more potent than the drug tolbutamide which is often used in the treatment of diabetes. Momordica also contains an insulin-like polypeptide, polypeptide-P, which lowers blood sugar levels when injected subcutaneously into type 1 diabetic patients. The oral administration of 50-60 ml of the juice has shown good results in clinical trials.\n\nExcessively high doses of bitter melon juice can cause abdominal pain and diarrhea. Small children or anyone with hypoglycemia should not take bitter melon, since this herb could theoretically trigger or worsen low blood sugar, or hypoglycemia. Furthermore, diabetics taking hypoglycemic drugs (such as chlorpropamide, glyburide, or phenformin) or insulin should use bitter melon with caution, as it may potentiate the effectiveness of the drugs, leading to severe hypoglycemia.\n\nGymnema Sylvestre (Gurmar, Meshasringi, Cherukurinja)\nGymnema assists the pancreas in the production of insulin in Type 2 diabetes. Gymnema also improves the ability of insulin to lower blood sugar in both Type 1 and Type 2 diabetes. It decreases cravings for sweet. This herb can be an excellent substitute for oral blood sugar-lowering drugs in Type 2 diabetes. Some people take 500 mg per day of gymnema extract.\n\nOnion and Garlic ( Allium cepa and Allium sativum)\nOnion and garlic have significant blood sugar lowering action. The principal active ingredients are believed to be allyl propyl disulphide (APDS) and diallyl disulphide oxide (allicin), although other constitutents such as flavonoids may play a role as well. \n\nExperimental and clinical evidence suggests that APDS lowers glucose levels by competing with insulin for insulin-inactivating sites in the liver. This results in an increase of free insulin. APDS administered in doses of 125 mg/ kg to fasting humans was found to cause a marked fall in blood glucose levels and an increase in serum insulin. Allicin doses of 100 mg/kg produced a similar effect. \n\nOnion extract was found to reduce blood sugar levels during oral and intravenous glucose tolerance. The effect improved as the dosage was increased; however, beneficial effects were observed even for low levels that used in the diet (eg., 25 to 200 grams). The effects were similar in both raw and boiled onion extracts. Onions affect the hepatic metabolism of glucose and/or increases the release of insulin, and/or prevent insulin's destruction. \n\nThe