What is S. saprophyticus that is found on the skin?
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Staphylococcus saprophyticus as a common cause of urinary tract infections.\n\nHovelius B, Mardh PA.\n\nUntil the last decade, coagulase-negative staphylococci occurring in urine specimens were usually regarded as a contaminant. In the early 1970s, i.e., more than ten years after the original demonstration of Staphylococcus saprophyticus in urine specimens, this species became recognized as a frequent cause of urinary tract infections (UTI). In young women, S. saprophyticus is, after Escherichia coli, the second-most-frequent causative agent of acute UTI. Patients with UTI caused by S. saprophyticus usually present with symptomatic cystitis. Signs and symptoms of renal involvement are also often registered. The urine sediment of a patient with UTI caused by S. saprophyticus has a characteristic appearance microscopically. Chemical screening methods for bacteriuria do not always succeed in diagnosing UTI caused by S. saprophyticus. Even when such an infection occurs above the neck of the bladder, low numbers of colony-forming units (less than 10(5) cfu/ml) of S. saprophyticus are comparatively often found in the bladder and voided urine. S. saprophyticus is usually susceptible to antibiotics commonly prescribed for patients with UTI, with the exception of nalidixic acid. The bacterium has a capacity for selective adherence to human urothelium. It causes direct hemagglutination. The adhesin for S. saprophyticus is a lactosamine structure. This staphylococcal species produces an extracellular enzyme complex that can inhibit growth of both gram-positive and gram-negative bacteria.\n\nref: http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=6377440&dopt=Abstract\n__________________________________________________________
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It is seen in systemic lupus erythematosus, esp. after the patient's face has been exposed to sunlight, and in seborrheic dermatitis, tuberous sclerosis, and dermatomyositis.
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Here you go!\n\nhttp://dermatology.cdlib.org/114/NYU/NYUtexts/1116045.html\n\nhttp://www.thedrsdr.com/diseases/necrobiotic_xanthogranuloma.htm\n\nhttp://archderm.ama-assn.org/cgi/content/abstract/128/1/94\n\nhttp://www.histopathology-india.net/NX.htm\n\nhttp://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12064218&dopt=Abstract\n\nBest answer?
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It is a protrusion from a fungus, if I remember correctly. It is necessary for feeding saprophytically, that is secreting digestive enzymes into food then absorbing the digested food.
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by what i have heard its found in dirt. it could be something due to hygiene. They found in the study that the people using the bauch and lombe (moisture lock)were acquiring the fungus so these were being removed from stores.
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Seriously, talk to your dermatologist.  Get a diagnosis and then look that up on medical websites.  Yahoo Answers is not the place to be asking about infections - Yahoo Answers is filled with people who give the wrong answers just to be cruel.  \n\nCall your dermatologist right away if your antibiotics aren't working or if your health issue has worsened.  Only s/he can diagnose you properly; by looking at your skin and all the other things.
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It is Paget's disease.\nPaget's disease shows varying radiological features depending on the stage of activity and the site. The most common type is the honey comb or spongy appearance which is widespread.\nThanks for your hint !!
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it's a type of ingrown particle..\n\ni've had those...  i don't get them too often.\n\nif this keeps up..  call the dermatologist.. have them look it over and see if they can't stop that mess.
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Sounds like psoriasis. Google it.
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Saprophytic Bacteria and Saprophytic Fungi.We can't do without them.
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it could be a lymph gland.  Do you have any pimples or sores, or a sore throat or cold?  Lymph glands are there to fight infecton.\nBy the way - what is "crapping"?
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Poss fungal infection.  Ever heard of Black Hairy Tounge?  It is REAL!!  I've only seen it once.\n\n\nSEE A DOCTOR!!!!!!!
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Skin infections are common and may be caused by bacteria, fungi or viruses. Breaks in the skin integrity, particularly those that inoculate pathogens into the dermis, frequently cause or exacerbate skin infections. Bacterial skin infections caused by corynebacteria include erythrasma, trichomycosis axillaris and pitted keratolysis. Staphylococci may cause impetigo, ecthyma and folliculitis. Streptococcal skin infections include impetigo and erysipelas. Human papillomavirus skin infections present as several different types of warts, depending on the surface infected and its relative moisture, and the patterns of pressure. The many dermatomycoses (skin infections caused by fungi or yeasts) include tinea capitis, tinea barbae, tinea cruris, tinea manus, tinea pedis and tinea unguium (onychomycosis). Candidal infections occur in moist areas, such as the vulva, mouth, penis, skinfolds and diaper area. Wounds caused by wood splinters or thorns may result in sporotrichosis. Animal bites may result in complex, serious infections, requiring tetanus and, possibly, rabies prophylaxis in addition to appropriate antibiotic therapy.
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My guess it is a fungus. Try asking your dermatologist. They should know. Or, if you get your nails done, your nail stylist may also know. Good luck.
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Overview of Leprosy\n\n\nLeprosy is also known as Hansen's disease. It is a chronic infectious disease, that primarily affects the nerves and the skin. In particular, the hands the face are affected, where numbness and disfigurement may occur. Rarely, does the disease affect individuals in developed parts of the world. It is most commonly found in South America, Africa and parts of Asia. The disease is not easily transmitted from one person to another. \n\n\nCauses of Leprosy\n\n\nLeprosy is caused by infection with the bacterium Mycobacterium leprae. It is thought to transmitted through airborne droplets or by direct contact with infected areas in other people. \n\n\nSigns and Symptoms of Leprosy\n\n\nLeprosy develops very slowly. Usually, no symptoms are present for several years. At that point, numbness of the hands, feet and face may occur. Gradually, the skin over the affected areas becomes thicker and deformed. Coloration may change.
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Hidradenitis suppurativa is a chronic, scarring disease that affects the apocrine, or sweat glands. It is a common skin condition but is frequently misdiagnosed. Hidradenitis suppurativa does not occur before puberty. Most people are between 20 to 40 years old when they develop hidradenitis suppurativa. It can be a disabling and distressing skin condition. \n\nCause of Hidradenitis Suppurativa\nThe exact cause of hidradenitis suppurativa is disputed. Because it looks similar to acne, some investigators believe that it is caused by the formation of keratin plugs in the follicles. Because hidradenitis suppurativa occurs in areas that contain apocrine, or sweat, glands, other investigators believe that it is caused by a blockage of the apocrine gland. In either case, the plugged gland or follicle becomes larger, ruptures, and becomes infected.  Obesity and cigarette smoking may be triggering factors.\n\n Appearance of Hidradenitis Suppurativa\nHidradenitis suppurativa occurs in areas of the body that contain sweat glands including the armpits, groin, buttocks, scalp, and under the female breast. When the condition is mild it may only present as a cluster of 2 or 3 blackheads that communicate under the skin. Once the disease begins, it gets progressively worse. The extensive, deep inflammation leads to painful abscesses. These heal incompletely and cause cord-like scars on the skin. The infection can progress under the skin in tracts linking the abscesses. These boils can be very painful and restrict movement of the affected area. \n\nDiagnosing Hidradenitis Suppurativa\nHidradenitis suppurativa is diagnosed clinically based on its appearance. There are no lab tests or biopsies that establish the diagnosis. In the early stages, it can be misdiagnosed as an isolated boils. Many people are embarrassed by the multiple infections thinking they are ingrown hairs in sensitive areas. They do not go to the health care provider until the boils have become numerous or intolerable. \n\nTreatment of Hidradenitis Suppurativa\nTreating hidradenitis suppurativa is difficult. It is best treated in the early stages before extensive scarring develops. However, it must be diagnosed correctly. The following are some of the treatment options: \n\nAntibiotics - Oral antibiotics may help suppress the inflammation especially in mild cases, but they do not cure the disease. The same antibiotics that are used for acne are used for hidradenitis suppurativa, only in higher doses. These antibiotics include erythromycin, tetracycline, minocycline, and doxycycline. Topical antibiotics do not help. \n\nIsotretinoin (Accutane) - Isotretinoin may be helpful in mild cases, but the response is unpredictable. Sometimes isotretinoin is used to reduce the size of the affected areas prior to surgery. Isotretinoin does have significant side effects and must not be used by pregnant women. \n\nSteroid injection - Injecting a steroid into an inflamed lesion is sometimes helpful. The injection helps reduce inflammation and may be used if an area is especially painful. Steroid injections are normally combined with other treatments. \n\nSurgery - In moderate to severe cases, surgery is the most definitive option. Taking out only the immediately affected area only leads to recurrence of the disease. The treatment of choice is called wide local excision. In this procedure, the affected area and surrounding normal tissue are surgically excised. The excision is not stitched back together but left open to heal from the inside out. Skin grafts may be placed in the excised area to speed healing.
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A powdery, white substance had long been observed on toes and fingers but was always assumed to be a simple, waxy secretion. Currie and a fellow graduate student decided to analyze the substance and found that it was a bacterium from the genus Streptomyces, the group of bacteria well known for producing antibiotics for humans. Further investigation revealed that, unlike general antibiotics for humans, the bacterium produces antibiotics that specifically attack the fungal pathogen Escovopsis.