2D echo cardiogram result show left ventricular end diastolic dimension decreased from 76 to 65mm.Is ds gud?
460755
any increase in ef is usually good.It means your heart is pumping more efficiently. The main thing you should worry about is why(leaky heart valve, virus, alcohol consumption) your heart was dilated in the first place and try to fix it. The aortic size is harder to evaluate the place I work at now that would be large but other places use different measurements and that could be normal. Go to www.seemyheart.org its a web site by the American society of Echocardiography for patients to understand there results
417140
That's surprising. Coarctation of the aorta should likely have been detected already unless you have been asymptomatic your whole life. Depending on the location of the coarctation that may explain the relative lack of symptoms. The bicuspid aortic valve often is not diagnosed and is present in a percentage of the normal population and usually not a major problem.\n\nIf you have questions on your condition, you should really talk with the cardiologist who is managing your case as he/she will know the specifics of your case and be able to address the issues.
1152108
This isn't really a question, but a test result. What is shows is a heart that has decresed blood supply that causes changes in the EKG, which is the electrical output of your heart with exertion. \n\nThe marks on an EKG are labelled P-QRS-T. The P is the contraction of the upper chambers of your heart, the QRS is the contraction of the larger lower chambers of the heart, and the T is the relaxation phase of those lower chambers, called the ventricles. The lead numbers and letters represent directions through the heart, and which ones have changes show the cardiologist what areas have decreased blood supply. "ST" changes are the place in the PQRST the hungry muscle shows up. "Ischemia" means dimininshed blood supply.\n\nThis usually means the coronary arteries which feed the heart blood have a blockage or other restriction of flow, from clots, build up of plaque or other damage. \n\nMore tests are needed to determine what kind of problem exists. A nuclear scan would show whether muscle has already been permanently damaged  or if it is possibly reversible. The "gold standard" would be a heart catherteriztion to map the exact problem, and even fix it with a balloon or by placing a stent.\n\nA test result like this one indicates a need for more follow up and whoever has this needs to promptly follow up with a cardiologist, preferably an interventional one. It's not a time to panic but to take necessary action.\n\nHope that answers some of your question, but this should be discussed with the doctor until you are satisfied with what it means.
294509
The entire LV wall function is not contracting / relaxing fully to its limit.\n\nWALL MOTION ABNORMALITIES\nNormally the contraction of the heart has a wringing motion, due to the arrangement of cardiac muscle fibers. For global evaluation, 2-D each is a useful tool. The SAX planes show all of the walls, except the apex. The best blood supply is at the base of the heart. \n\n\nHYPERDYNAMIC \ncompensatory decrease in systolic wall thickening \n\n\nHYPOKINETIC \nslow moving wall shows decrease in fractional shortening \n\n\nAKINETIC \nwall may thin in systole \n\n\nPARADOXIC \nwall motion is compensatory and moves opposite of normal \n\n\nDYSKINETIC \nwall motion compensates for abnormal adjacent walls
643959
In diastolic dysfunction (impaired relaxation) it slows the heart rate, increasing filling time ultimately resulting in increased cardiac output.\n\nIn systolic dysfunction (impaired contraction) it is thought to work on decreasing the sustained sympathetic stimulation seen with left ventricular dysfunction which can cause the b receptors to down regulate.  It also decreases the risk of fatal arrythmia associated with sympathetic surges.  It decreases myocardial oxygen demand.  There is some decrease in contractility initially, however doses are usually titrated gradually and tolerated.  After months of treatment contractility can actually improve due to the reasons described above.\n\nAdditionally, people are usually not started when in florid heart failure.
219977
Nuclear tests are great at telling you where there is deficit of blood flow.  That implies narrowing of a particular coronary artery.  "Decreased tracer uptake" in the septal wall means the artery that supplies the septum (the musculafr divider between left and right side of the gheart) may be blocked to some degree (but not fully).  It means you are at the risk of a heart attack to this area and may need an angiogram to take a closer look (and to open the blockage if ned be). Work with your cardiologist about it and good luck!
918373
My honest feeling is the BP readings taken are not correct. Such high and low changes are next to impossible. You need to tak him to a cardiologist for evaluating the position and treating him. Blood Pressure - high or low - is termed as silent killer. Please see the doctor without delay. \n\nNow a short note from Wikipedia about BP for your inforamtion -\n\nBlood pressure is the pressure exerted by the blood at right angles to the walls of the blood vessels. Unless indicated otherwise, blood pressure refers to systemic arterial blood pressure, i.e., the pressure in the large arteries delivering blood to body parts other than the lungs, such as the brachial artery (in the arm). The pressure of the blood in other vessels is lower than the arterial pressure. Blood pressure values are universally stated in millimetres of mercury (mmHg). The systolic pressure is defined as the peak pressure in the arteries during the cardiac cycle; the diastolic pressure is the lowest pressure (at the resting phase of the cardiac cycle). The mean arterial pressure and pulse pressure are other important quantities.\n\nTypical values for a resting, healthy adult human are approximately 120 mmHg systolic and 80 mmHg diastolic (written as 120/80 mmHg), with large individual variations. These measures of blood pressure are not static, but undergo natural variations from one heartbeat to another or throughout the day (in a circadian rhythm); they also change in response to stress, nutritional factors, drugs, or disease-
208776
Yes.\n  A cardiac cath not only shows in more detail the exent of the disease, but also if there are compenstory mechanisms set up by other arteries to help these blockages.(i.e. backfilling from other arteries.).\n  Also a Left ventriculargram will be performed to assess the function of the heart muscle, and see how effective it is and if there are any areas of muscle no longer working.\n  If there are problems there when doing the cath, then they can be fixed, there and then by placing balloons or stents into the affected artery. (This is called a PCI or percutaneous coronary Intervention.)
224694
It's not really bad, actually the normal range is:\n100/60 - 140/90, so it's just your bottom number (dyastolic) that's high on this reading and I imagine if you were to check it again it would probablly go down. You would tend to be more concerned if during a 2 week period it stayed at this level most of the time.
486381
No, an echo is only an ultrasound scan and is non-invasive.  What you are probably experiencing is stress caused by your worrying about the echo.  You probably have furred up cardiac arteries and the stress is causing them to narrow further and giving you the pain (angina!).\n\nYou seriously need to see your doctor and ask about having an angiogram, it could be that you either need antiangiotensive medication, an angioplasty or even a coronary artery bypass graft.
770469
No connection, as far as my personnal experience is concerned. My cholestrol was at 350, but my BP was 120/80 in 1996.
368832
Nikki - I'm happy to give you some information as a cardiologist, but the best thing for you to do is to go back and see your cardiologist in order to have these questions answered because there are several things important to making a diagnosis that you don't mention.  One of the big factors is how old you are and what your risk factors for coronary disease are (high blood pressure, lipid levels, family history, whether you smoke).  Also, it helps to know what brings on your shortness of breath and/or chest discomfort.  Is it with activity only?  Does it happen at random?  How long does it last?  What makes it go away?\n\nLet's talk about your bicuspid aortic valve first.  A bicuspid aortic valve is an aortic valve that has developed abnormally such that two of the three cusps have fused into one.  There are two main issues with this.  The first is that bicuspid aortic valves have a much higher than normal tendency to become stenotic, meaning it can become calcified, hardened, and interfere with the normal ejection of blood from your heart.  The first sign of this would be a heart murmur.  Over time, if the valve becomes severely stenosed, it can cause chest pain, shortness of breath with activity, or fainting spells.  Patients with bicuspid aortic valves should undergo routine echocardiograms every one to two years to assess the valve function.  Once the valve becomes severely stenosed, surgery to replace the valve should be discussed.  Not all patients with bicuspid valves will go on to have aortic stenosis, but those that do typically require surgery in their 40's or 50's.\n\nThe second issue with patients with bicuspid aortic valves is that they commonly have aneurysms (or dilatation) of the aortic root and/or ascending aorta.  This is important because patients with a bicuspid aortic valve are also at a higher risk for dissection (a tear in the lining of the aorta) than other patients with the same degree of aortic dilatation.  Therefore, this is also something that should be followed by serial echocardiograms.  The recommendation for surgery in a patient with bicuspid aortic valve is an ascending aortic aneurysm of approximately 5.0 to 5.5cm, but this varies somewhat from doctor to doctor.  (This is a bit earlier than would be recommended for a patient without a bicuspid aortic valve.)\n\nBeta-blockers, such as atenolol, have been shown in one study to reduce the rate of growth of the ascending aorta in a group of young males with Marfan syndrome.  Since patients with bicuspid aortic valves have histologically similar changes in their aortas, this data has been extrapolated and applied to patients with bicuspid aortic valves as well.  While there are no hard and fast rules, I would personally keep all patients with a bicuspid aortic valve and an enlarged aortic root on beta-blockers for life.  This reduces the blood pressure and amount of stress placed on the wall of the aorta, and this is how it is thought to reduce growth of the aneurysm.  For this reason, patients with a dilated aortic root and/or ascending aorta are advised to restrict their activity to moderate aerobic exercise (no training for marathons or that sort of thing) and no isometric exercises.  That's primarily heavy weight lifting, but really includes any activities where you Valsalva (straining, such as when you're constipated), so stomach crunches are sort of on the borderline of what would be recommended.\n\nTwo last thing about bicuspid aortic valves... there is a strong familial tendency, so most cardiologists would screen siblings of patients with a bicuspid aortic valve as well.  Also, as one of the previous doctors recommended, you should have a prescription to take antibiotic prophylaxis before dental procedures to prevent infection of the valve.  By the same token, you should make sure you get regular dental care because poor dental health increases your risk as well.\n\nNow regarding the shortness of breath and che
866194
An enlarged heart is a vague term but yes it is always a concern.  Since this is your daughter's boyfriend, I must assume that he is relatively young.  If this is correct he most likely has a congenital cardiomyopathy.  And as this may be transmitted to his children, there is a need for proper evaluation - for your daughter's boyfriend, and his future (possibly your grandchildren) children.
490520
Left axis deviation just means that the overall forces in your heart tend to be more leftward than average.  It could be nothing, but probably needs to be investigated further.\n\nPossible causes (not a complete list):\n\nNormal variant \nAscites \nHigh diaphragm \nLeft Atrial hypertrophy \nLeft Bundle Branch Block \nWolf-Parkinson-White Syndrome (WPW Syndrome) \nChronic Obstructive Pulmonary Disease (COPD) \nHyperkalemia \nRight ventricular ectopic rhythms\n\nSome of these are curable.  Or it may not be anything at all(normal variant).\n\nFollow up with your doctor!
465578
Cardiac Diastole is the period of time when the heart relaxes after contraction in preparation for refilling with circulating blood. Ventricular diastole is when the ventricles are relaxing, while atrial diastole is when the atria are relaxing. Together they are known as complete cardiac diastole.\n\nDuring ventricular diastole, the pressure in the (left and right) ventricles drops from the peak that it reaches in systole. When the pressure in the left ventricle drops to below the pressure in the left atrium, the mitral valve opens, and the left ventricle fills with blood that was accumulating in the left atrium. Likewise, when the pressure in the right ventricle drops below that in the right atrium, the tricuspid valve opens, and the right ventricle fills with blood that was accumulating in the right atrium.\n\nnever heard of the depolarisation but though, sorry!
434299
Diastolic pressure is the pressure in the circulatory system in between heartbeats. The blood isn't at any pressure inbetween beats, whether you've just exercised or are sleeping.  If anything, I would guess it might be a little lower if blood vessels dilate (expand) to let off some heat after exercise, as the volume of space for the same amount of blood might increase a little?
1110693
well yes as all that weight puts a strain on the heart not to mention all the other organs but shortness of breath can also be due to other things so see your dr