what factors contribute to rsv?
466858
RSV is actually a much more common disease than most people realize. Almost all children catch it before two years of age but in most cases it is simply a minor cold with low grade fever and no one knows it was RSV. It can become serious for several reasons:\n\nThe child was born premature and has an underdeveloped immune system.\n\nThe child is less than six months of age meaning they still rely of the antibodies from the mother for immunity.\n\nThe child has another infection at the same time therefore reducing the effectiveness of the immune system.\n\nThe child has an immune disorder or anything else compromising the ability of the immune system to ward off infection.
894354
family history\nobesity\npoor diet\nlack of exercise
1179209
Repondez.\n\nIt is important to say "RSVP Please" otherwise people might think you know what the svp stands for.
71016
I work in a small hospital in New Mexico and this year, especially in November and December, we have had our ER full of flu patients, some of the elderly and very young among the sickest.  I would like to add that RSV alone has caused a few of our littlest ones to need ventilators.  A friend lost his grandchild (7 months old) due to RSV & pneumonia.  Two days ago the one month old nephew of a co-worker stopped breathing and had to be resussitated.  This baby also had RSV.  The flu has hit hard and now is on the wane....RSV is still around and adults as well as older children can be infected and the carriers to infants and toddlers.  I know you asked about the flu, but sometimes those who believe they have the flu have instead another virus or infection like RSV and even Whooping cough.  This year RSV in our town seems to be "deadly".
477508
Background: Upper respiratory infections are common infections and include pharyngitis, sinusitis, epiglottitis, laryngotracheitis, and the common cold. Viruses play a significant role in the pathogenesis of many of these infections. Bacteria and other organisms also are responsible.\n\nPharyngitis is an inflammatory process of the pharynx, hypopharynx, uvula, and tonsils that can be caused by viral or bacterial infection and, occasionally, both. Distinguishing between these infections is important because rheumatic fever and acute glomerulonephritis may complicate untreated group A beta-hemolytic streptococcal infections (GAS), but they usually can be prevented by appropriate antibiotic treatment.\n\nSinusitis is an inflammatory process involving the paranasal sinuses (maxillary, frontal, ethmoid, and sphenoid). It usually is a bacterial complication of a viral upper respiratory infection.\n\nEpiglottitis is a life-threatening disease observed most frequently in children aged 1-6 years, often during the fall and winter. Although less common, it also can affect adults.\n\nLaryngotracheitis usually is the result of viral infection. The subglottic area and trachea are involved, whereas the area above the true vocal cords is spared. When children younger than 5 years have the infection, it is called croup.\n\nThe common cold is a mild, self-limited, catarrhal syndrome caused, for the most part, by members of 5 families of viruses. A small proportion of colds are complicated by bacterial infections of the paranasal sinuses and the middle ear, which require antimicrobial therapy.\n\nPathophysiology: In infectious pharyngitis, bacteria or viruses may invade the pharyngeal mucosa directly, causing a local inflammatory response. Other viruses, such as Rhinovirus, cause irritation of pharyngeal mucosa secondary to nasal secretions.\n\nStreptococcal infections are characterized by local invasion and release of extracellular toxins and proteases. In addition, M protein fragments of certain serotypes of GAS are similar to myocardial sarcolemma antigens and are linked to rheumatic fever and subsequent heart valve damage. Acute glomerulonephritis may result from antibody-antigen complex deposition in glomeruli.\n\nThe most common local predisposing cause of suppurative sinusitis is a viral upper respiratory tract infection. Inflammation and edema in the ostial meatal complex can obstruct the sinus ostium, leading to hypo-oxygenation of the sinus, disturbed ciliary and mucous blanket function, and diminished local resistance.\n\nIn epiglottitis, supraglottic structures become inflamed and edematous, leading to narrowed airway and respiratory compromise. Epiglottitis in children almost always is caused by Haemophilus influenzae type b (HIB). In adults, it frequently is nonbacterial.\n\nLaryngitis mainly affects children and begins with a prodrome of a few days of a mild viral upper respiratory infection. As the infection extends to the proximal trachea, diffuse inflammation with exudate and edema of the subglottic area causes narrowing of the airway. The cricoid ring of the trachea, in the immediate subglottic area, is the narrowest portion of the airway in a child. A small amount of edema can cause significant airway obstruction. Inspiratory stridor occurs when air flows through this narrowed subglottic area.\n\nInfection with common cold viruses characteristically is short in duration and self-limited.\n\nFrequency:\n\n    * In the US: Children in day care in the United States are estimated to have an upper respiratory infection approximately every 3 weeks from the age of 6 months to 2 years. The incidence then decreases, and, by the time of school entry, a child has about 3-6 episodes of upper respiratory infection per year. Most of these infections include pharyngitis and/or laryngitis. Viral upper respiratory infections frequently occur in mini-epidemics. They are more common in the winter, except for those caused by enteroviruses, which
597808
Many different bacteria and viruses can cause pneumonia. For typical pneumonia, the commonest one is Streptococcus pneumoniae. Others include Haemophilus influenzae, Klebsiella, Pseudomonas aeruginosa, antibiotic-resistant Staphylococcus aureus, etc.\nFor atypical pneumonia, it can be caused by coronavirus, adenovirus, influenza virus, parainfluenza virus, respiratory syncytial virus, Legionella pneumophila, etc.
860305
I believe it has something to do with the fact that, viruses such as pneumonia affect  the older (and very young) moreso than an adult in his/her prime because the immune system is weakened.  Either by age or because it is premature (as in with children)  As you grow older, your body weakens and is once again more susceptible to disease/sickness.
885934
Viral\n  Menningitis\n  Mono\n  HIV\n  Influenza\n  Herpes\n\nBacterial\n  Common Cold\n  Tonsilitis\n   Laryngitis\n   TB\n   Staph
950764
Start taking Zinc.  Clinically proven to help prevent and shorten colds.
3181
Usually common in a person with a reduced immnunity, to infection, in most cases it is caused by either a virus or bacteria. causes of viral pneumonia include adenovirus, respitory syncytial virus, the most common bacterial pneumonia is caused by streptococcus pneumoniae.  commonly occurs in alcoholics,the elderly, infants or as a complication to a serious illness
604807
In regards to what? Blood? Sex typing? Genes? Cars? You need to be more specific!!
372093
Genetic makeup is the most important factor. This will affect the hormones responsible for the growth of everything, including sexual organs.
560850
1By close contact with sputum smear positive individuals. 2. environmental factors that lowers the resistance , like malnutrition, poor and over crowded housing, alcoholism and/or drug addiction ,heavy smoking, corticosteroid therapy. Sometimes related with oh er diseases, like influenza, whooping cough. More common with diabetes mellitus, cirrhosis of liver, following partial gasterectomy. .  Due to immuno suppression.  HIV infection/AIDS.IV drug abusers, Renal failure and Family history.
129552
CPI fails to degrade STAT1 It has been reported that mutant SV5 containing mutations in the shared N termini of V and P protein (known as CPI– virus) accelerates viral gene expression and causes increased cell death, with characteristics of apoptosis (60). Even though the mutant SV5 (CPI– virus) is different from rSV5VC, they may induce cell death through similar pathways. CPI– virus encodes two mutant proteins, whereas rSV5VC encodes a truncated V protein with a very short half-life. Thus, the phenotype of rSV5VC is likely a result of loss of V function whereas phenotypes of CPI– virus may be due to gains as well as losses of functions of both V and P mutant proteins. It is not unexpected that mutations in the shared N termini of P and V proteins of CPI– virus affect viral gene expression, as SV5 P is essential for viral RNA replication and transcription. It is possible that overexpression and early expression of viral proteins (including glycoproteins F and HN) in CPI– virus-infected cells can cause ER stress and trigger cell death in a fashion similar to that seen with the induction of apoptosis by rSV5VC infection, in which viral gene expressions are not affected in rSV5VC-infected cells (since it still encodes a wt P protein). Alternatively, increased UPR response in rSV5VC-infected cells may be a result of increased ER stress induced by overexpression of viral glycoproteins even though the increase was not obvious in rSV5VC-infected cells. \n\nMitochondria play an essential role in intrinsic apoptotic pathways through caspase-dependent and caspase-independent pathways. In rSV5VC-infected cells, apoptosis was caspase dependent but caspase 9 activity was not detected and an inhibitor against caspase 9 did not affect rSV5VC-induced apoptosis, suggesting that a previously unidentified caspase was activated through mitochondria or that mitochondria did not play a determining role in rSV5VC-induced apoptosis. Caspase 12 localizes to ER, and there is no evidence of its association with mitochondria. At present, it is not clear how ER stress induced by rSV5VC infection affects mitochondrial function. Studies of the relationship between ER stress and mitochondrion damage in virus-infected cells are in progress.
852878
To get a cold, you need to get a version of a cold virus into the nose or eye ducts. Touching your nose is enough if you have touched a virus from something like a door handle.\nWorrying, and stress weakens your immune system, making you more susceptible to colds.\nAn healthy diet, plenty of fruit and veg, and water aids the immune system.\n\nIf somebody sneezes in your presents the best avoidance advise, is to temporarily breath through your mouth.\n\nChristmas is a common time for lots of people to get colds, partly because they are trying to do too much, it becomes stressful, partly because lots of people meet around that time, and cold viruses abound. People are most infectious before they have obvious symptoms.
928827
How old is your son? (Could be "mono" if he's a teenager). Did they do any throat/NP cultures? Any respiratory symptoms? What was the differential cell count? This time of year (depending on where you live) respiratory viruses get started. If it is a virus, antibiotics won't help. If it's something like strep throat or "walking pneumonia" then antibiotics will kick it pretty quickly. Could also be a secondary bacterial infection following a viral infection. Just make sure he takes the whole course of the antibiotics - failure to do so is one of the leading causes of antibiotic resistant bacteria. Good luck!!
73051
All sorts of factors affect the suitability of the human body fluids to sustain life; these include properties like temperature, salinity, and acidity, and the concentrations of nutrients such as glucose, various ions, oxygen, and wastes, such as carbon dioxide and urea. Since these properties affect the chemical reactions that keep bodies alive, there are built-in physiological mechanisms to maintain them at desirable levels.\n\nHowever, it should be noted that homeostasis is not the reason for these ongoing unconscious adjustments. Homeostasis should be thought of as a general characterization of many normal processes in concert, not their proximal cause per se. Moreover, there are numerous biological phenomena which do not conform to this model, such as anabolism.