treatment of osteoporosis?
401380
It is well established that estrogen replacement during menopause protects bone mass and helps protect against the risk of osteoporotic fractures. The accelerated bone loss during menopause has little relationship to the amount of calcium intake. After age 60, however, the proper attention to calcium intake is very important and has been shown to increase bone density. An important study published in 1992 showed that elderly women who took supplemental dietary calcium (with vitamin D) had a 30% less fractures (including hip fractures) than similar women who did not take supplemental calcium. When these investigators looked at these women again 3 years later, they still had a significantly less rate of fractures, and, they had a 15 % decreased chance of death. Most endocrinologists feel that the use of calcium supplements are safe (and inexpensive) and recommend it to most (all?) elderly men and women unless specific contraindications exist. it helps to take calcium with vitman d in them ..
385516
No, it's not.  You need more than just calcium if you are experiencing a weakness of bone.  You need vitamin D as well.  Try taking Viactic as the other answer says.  And regular checkups with your doctor are important to make sure you do not have osteoporosis.
142400
Moderate, regular exercise improves bone density.  A sedentary lifestyle increases the rate of bone loss that typically occurs starting by the 4th decade of life.  Adequate dietary intake of calcium, vitamin D, numerous other vitamins and minerals, protein and more can have positive effects on bone density.
66193
my mom used estroven to help her menopause. it really helped when she took it regularly.
517956
You must have osteopenia and not osteoporosis. Osteoporosis is defined as a T-Score more than -2.5. You have to start taking calcium so as not to make it worse. Also, see your MD to ascertain if you have anyother condition or if you are taking any medications that may make this worse. Good luck.
1133391
To compare the relative sensitivity and specificity of bone turnover indexes for bone loss or gain in early postmenopausal women, we performed a multicenter trial in 236 menopausal women (mean age, 51 yr), who were randomized to hormone replacement therapy (HRT) or calcium supplementation (CS; 500 mg/day) for 1 yr. Two markers of bone formation, osteocalcin (OC) and bone alkaline phosphatase (BSAP), and two markers of bone resorption, urinary N-telopeptide (NTx) and urinary free deoxypyridinoline (fDpd), as well as spine and femoral neck bone mineral density (BMD) were measured at baseline and 3, 6, and 12 months after treatment. Women receiving HRT (n = 105) showed a significant increase in spine BMD (+2.5%; P < 0.0001) and hip BMD (+1.0%; P = 0.02) compared to women receiving CS, who showed a decline at both sites (-1.1%; P < 0.01). All four markers showed time-dependent decreases in women receiving HRT (P < 0.001) and no change in women receiving CS alone. When baseline indexes of turnover were stratified by quartile, there was a significantly greater increase in BMD among those with the highest NTx, OC, and BSAP levels compared to that in those with the lowest NTx, OC, and BSAP levels (P < 0.05). The highest quartile for percent change from baseline to 6 months in fDpd, BSAP, and NTx was also associated with the greatest change in spine BMD at 1 yr. Receiver operator characteristic curves for percent change from baseline to 6 months in an individual marker to 1 yr change in BMD during HRT revealed that the percent change in NTx provided the greatest discrimination between gain and loss of BMD. When subjects receiving HRT were compared by their positive or negative skeletal response at 1 yr and their baseline turnover marker, initial NTx values were significantly higher in those that gained bone than in those that lost bone (P = 0.0002). CS women in the highest quartile for NTx at baseline had significantly greater decreases in spine BMD than subjects with the lowest NTx values (P < 0.005), although this was not true for fDpd (P < 0.20). In conclusion, for early postmenopausal women there are differential responses of biochemical markers to HRT and CS. Baseline urinary NTx and serum OC were the most sensitive predictors of change in spine BMD after 1 yr of either HRT or CS. Similarly, the percent change in NTx and OC from baseline to 6 months best predicted bone gain or loss. We conclude that markers of bone formation and resorption can be used clinically to predict future BMD in early postmenopausal women.
1182079
Yes, there are certain medications that can actually increase your bone thickness.  But I think prevention is always better!
685944
Best treatment is going to a GYN/OBS doctor\nBut losing over weight,and using some hormonal drugs are always usefull
505709
Find a doc who does natural hormone replacement therapy.\nTry ACAM (American Academy for the Advancement of Medicine)
625402
by eating calcium to stimulate bones, eat carbohydrates, proteins etc\n\nstimulatio from growth hormone, somatotropin hormone, tyroxin, thymusin, etc
240317
See:\nhttp://www.nlm.nih.gov/medlineplus/ency/article/000621.htm#Treatment
420217
Osteopenia refers to bone mineral density (BMD) that is lower than normal peak BMD but not low enough to be classified as osteoporosis. Bone mineral density is a measurement of the level of minerals in the bones, which indicates how dense and strong they are. If your BMD is low compared to normal peak BMD, you are said to have osteopenia. Having osteopenia means there is a greater risk that, as time passes, you may develop BMD that is very low compared to normal, known as osteoporosis.\n\nSome people who have osteopenia may not have bone loss; they may just naturally have a lower bone density. Osteopenia may also be the result of a wide variety of other conditions, disease processes, or treatments. Women are far more likely to develop osteopenia and osteoporosis than men. This is because women have a lower peak BMD and because the loss of bone mass speeds up as hormonal changes take place at the time of menopause. In both men and women, the following factors can all contribute to osteopenia: \n\nEating disorders or metabolism problems that do not allow the body to take in and use enough vitamins and minerals \nChemotherapy, or medications such as steroids used to treat a number of conditions, including asthma \nExposure to radiation \nHaving a family history of osteoporosis, being thin, being white or Asian, getting limited physical activity, smoking, regularly drinking cola drinks, and drinking excessive amounts of alcohol also increase the risk of osteopenia and, eventually, osteoporosis.\nWhether you will tend to develop osteopenia is, in part, already determined. Things like whether you have any family members who have had osteoporosis or osteopenia, whether you have chronic asthma that requires you to take steroids, and how much calcium and vitamin D you got while you were growing up are beyond your control now. However, if you are a young adult or if you are raising children, there are things you can do to help develop strong bones and help slow down osteopenia and prevent osteoporosis.\n\nYour bones don't reach their greatest density until you are about 30 years old, so for children and people younger than 30, anything that helps increase bone density will have long-term benefits. To maximize bone density, make sure you get plenty of calcium and vitamin D through your diet and by spending a little time in the sun, get weight-bearing exercise on a regular basis, don't smoke, and avoid excessive alcohol or cola drinks. If you have children, teach them to make a nutritious diet and exercise part of their normal routine, make sure they play a little in the sunshine to help their bodies make more vitamin D, and help them avoid smoking and alcohol as they get older.\n\nIf you're older than 30, it's still not too late to make these lifestyle changes. A balanced diet and regular exercise will help slow the loss of bone density, delay osteopenia, and delay or prevent osteoporosis.
880425
I am puzzled as to why you are on Flexeril for osteoporosis & chodromalacia, unless you are using it as a sleeping pill.  You are awfully young for osteoporosis - are you on medications to help with that?  Have you considered surgical intervention for the knees?  It is better to fix the underlying problem than to try and treat the symptom (pain).\n\nLong term use of narcotics can cause you to not only become tolerant, but to develop extreme sensitivity to pain - things that don't normally cause pain might feel painful to you (opiate-induced hypersensitivity)\n\nI would consult a pain management specialist with the goal of getting off narcotics completely.  Contact your local hospital's anesthesia department for a referral.\n\nGood luck.
342669
No, there is no cure, but there are some treatments that are effective for some people.  Check out www.helpforkp.com for treatment options.
410431
Overview\nIn recent years, advances have been made in the treatment of ankylosing spondylitis and related diseases. There is a lot that can be done to relieve the pain and stiffness of the AS and recent studies show that the new biologic medications (TNF-a Inhibitors) can potentially slow or halt the disease progression in some people. \n\nA common treatment regimen involves medication, exercise and possibly physical therapy, good posture practices, and other treatment options such as applying heat/cold to help relax muscles and reduce joint pain. In severe cases of AS, surgery may also be an option.
108136
I've been told that if you drink lots of Milk that it can be helpful in strengthening your bones.  Give that a shot!!
692589
No there is no cure, but medications can lessen the effects.  However the damage cannot be reversed short of joint replacement.