how does decreased lung compliance influence ventilation?
397422
Lung compliance is a measure of how elastic (how much something wants to return to its original shape) the lung tissue is. It influences both breathing in and out. When breathing in the lung needs to be able to stretch to accomodate more air. When breathing out, the compliance/elasticity  of the stretched lungs will help to force the air out. If a person has decreased lung compliance they will not be able to expel as much air when they breathe out, so they can only inhale a smaller volume of fresh air. Thus a person with decreased lung compliance takes more frequent, shallower breaths.
1013255
There aren't really may complications from it.  PEEP (positive end expiratory pressure) is used when patients are being ventilated because their alveoli (the little things inside the lung where exchange of oxygen actually takes place) tend to collapse on themselves.  PEEP is used because it never allows the air to completely leave the lung and therefore the alveoli stay filled with air a little bit.  If the PEEP setting is too high, it may cause the alveoli to burst, but this will be seen on an X-ray and the setting will be changed.  PEEP is a good thing, though and doesn't usually cause problems.  Even if it does cause a few alveoli to rupture, this doesn't cause any long term damage.  Worst case scenario is that they may partially collapse a lung, but this is very, very rare and really easily treated with a chest tube for a few days.  When patients are mechanically ventilated they are monitored very closely to prevent these kinds of problems.  Hope this helps
60510
Yes, the amount of air affects how firm the outer part is. The more air, the more resistance to flex the skin has due to the pressure, the "bouncier" it will be.
340646
hmm... well as you inhale, the pressure in your lungs decreases.  And according to the bernoulli principle, it decreases proportionally to the rate of inhalation.
232358
inhalation is breathing in, exhalation is breathing out
417764
It's a question of the relative amounts of muscle and fat, together with the density of your bones. Basically, if your overall body density (mass/volume) is >1 you will sink, if it's <1 you will float.\n\nThe air in your lungs *does* make a difference, since taking a deep breath both expands your volume, and increases the amount of low-density stuff in that volume. If I have a lungful of air, I can just about float; breathe out, and I sink.
476345
no, but it can aggravate them. For example, i have had pneumonia twice because my lungs are weak and prone to that, also I frequently get bronchitis.
326337
your question basically mean how does obstrustive or restrictive lung disease affect ventilation.  \n\nIn obstruction lung disease, the airway itself is full of obstruction and not allowing sufficient air travel to the aveolar for gaseous exchange.\n\nIn restrictive lung disease, the lungs tissue is restricted and not air can travel through due to the non-plastic pattern of the tissue
807461
?? \n\nWhy would there be any disadvantages associated with adequate ventilation since breathing is occuring at a normal rate and the oxygen is being delivered to the cells?
780534
In normal breathing, air is drawn in through the bronchial passages and down into the increasingly fine network of tubing in the lungs called the alveoli, which are many thousands of tiny sacs surrounded by capillaries. These absorb the oxygen and transfer it into the blood. When toxins such as smoke are breathed into the lungs, the particles are trapped by the hairs and cannot be exhaled, leading to a localised inflammatory response. Chemicals released during the inflammatory response (trypsin, elastase, etc.) begin to break down the walls of alveoli (alveolar septum). This leads to fewer but larger alveoli, with a decreased surface area and a decreased ability to take up oxygen and lose carbon dioxide. The activity of another molecule called alpha 1-antitrypsin normally neutralizes the destructive action of one of these damaging molecules.\n\nAfter a prolonged period, hyperventilation becomes inadequate to maintain high enough oxygen levels in the blood, and the body compensates by vasoconstricting appropriate vessels. This leads to pulmonary hypertension. This leads to enlargement and increased strain on the right side of the heart, which in turn leads to peripheral edema (swelling of the peripherals) as blood gets backed up in the systemic circulation, causing fluid to leave the circulatory system and accumulate in the tissues.\n\nEmphysema occurs in a higher proportion in patient with decreased alpha 1-antitrypsin (A1AT) levels (alpha 1-antitrypsin deficiency, A1AD). In A1AD, inflammatory enzymes (such as elastase) are able to destroy the alveolar tissue (the elastin fibre, for example). Most A1AD patients do not develop clinically significant emphysema, but smoking and severely decreased A1AT levels (10-15%) can cause emphysema at a young age. In all, A1AD causes about 2% of all emphysema. However, smokers with A1AD are in the highest risk category for emphysema.
614278
Survey says:  *ding*\n\nOUT!\n\nBecause:  The diaphragm acts to inhale.  So when functions cease, the diaphragm relaxes and air is exhaled.\n\nNot *all* the way out, though, since the rib cage will still support the lungs, some air remaining.
430076
b)less,because while you exercising your lungs needs more extra oxygen,makes your body need to breath more air
98888
Assuming you're talking about in the body: increased ventilation increases the amount of oxygen in your alveoli (lungs) and decreases the amount of CO2, the pH is buffered under normal circumstances by bicarbonate but more CO2 would lower the pH. Decreased ventilation gives the opposite effect of the ones mentioned.\n\nHope this helps
331726
During an Asthma attack, a chemical is released in the body which causes the linining of the airway and in some cases the bronchus and lungs to swell. This means that there is a reduced area for air to travel though. This can often be demonstrated by breathing through the middle of a toilet roll (the cardboard bit) - this is your open airway; and then breathing through a reglar McDonalds straw - your reduced airway.\n\nVentilation is then reduced. The side effects of this are increased acidity in the bloodsteam (Carbon Dioxide Levels Rise) and in severe cases the person will faint and possibly go into shock.\n\nAsthma is easy to treat with an inhaler such as Ventolin. These contain drugs which have anti-inflamatory properties which counter the effect of the chemicals causing the swelling.\n\nTom Walvin\nBiologist
668599
The ph has a huge effect on the Respiratory system.\nYour body will try to compensate to keep your ph between 7.35-7.45 at all times.\nCo2 is acid\nHCO3 (bicarb) is a buffer\nand they will be adjusted to regulate the ph\nThe way the respiratory system is involved is with the co2\n\nwhen a patient ie... diabetic ketoacidosis has a bicarb of 5, ur body is extremly aciddic... so the respiratory rate will increase and u will have kussmal respirations, fast and hard.... ur body is trying to blow off co2, to make the body more alkolitic.. and raise the ph from the lowered hco3...\n\nnext when a patient is retaining co2, and the ph goes way down, the hco3 goes up to try to bugger the acidic effect of the co2, ur body will breathe slower... and this is a respiratory failure...
635596
The one major symptom is shortness of breath, early on in the disease it come with mild exertion, as the disease worsens the shortness of breath becomes more constant.  Coughing becomes a problem, takes alot of work to bring up the junk forming in the lungs.  Panic breathing becomes a problem, this is when you just can't seem to get enough air into your lungs it is truly frightening.  COPD is treated with bronchodilators, anti-spasmodic medications and steroids usually in the form of inhalers.  This delivers the medication directly to the lungs where it will do the most good.  Oxygen is eventually required, since the COPD patient has a hard time getting enough air into his lungs, the higher oxygen concentration helps.  Further treatment is surgical.  Lung reduction surgery can help but in the end is only temporary.  The only way to be rid of COPD alltogethor is by lung transplant.  This is how I got rid of mine nearly 9 years ago.
108341
what do u mean??? isn't more lung capacity supposed 2 be greatt?????.. what the dangerous part? i mean.. u can breath more.. u can hold ur breath more...