why does the hypocalcemia causes CNS stimulation plz can u answer?
395676
low ca levels lower the threshold for neural firing.\nIn the peripheral nervous system this manifests as tetany and in the Cental nervous sytem the manifestation is convulsions( quite rare)
423043
You would need to get on some medication like synthroid. Talk to your doctor.
782836
Magnesium is chemically similar to calcium, but cannot be used by the muscle for contracting (unlike calcium). Since the total salt concentration in our body is regulated by osmosis, raising the Mg concentration will lower the Ca concentration.\n\nCramps are caused by too extensive muscle contraction (i.e. lots of Ca connecting various strands in the contracted state). By offering less Ca in the cell fluid, the contractions can be reduced somewhat.
415922
No, its a bit simpler than that.  It has to do with loss of ATP production through aerobic resperation.  The brain, though only 3-4 pds, uses 20% of the total calories in the body, because of the massive requirement of neurons to maintain the ionic gradient, primarily through Na+/K+ ATPase, an energy-using enzyme.  This ion pump maintains the hyperpolarization required to keep the neuron from depolarizing until a specific signal, such as binding of neurotransmitter, opens the Na+ channels.  There is very little storage of glycogen or creatine in the brain, and thus there has to be a constant supply of blood glucose to maintain this.  A drop in glucose below a spcific concentration causes a dramatic decrease in production of ATP, thus the Na+/K+ ATPase cannot keep up with the ionic leakage of Na+ out of the cell and K+ into the cell.  The relative intraneuronal negativity is lost, and there is uncontrollable depolarization of neurons, as voltage-gated channels open.  Two things then happen- 1) This uncontrolled firing of the neuron is transmitted, via axons, to other neurons (themselves already partially depolarized because of lack of energy for Na+/K+ ATPase) and 2) the neurons that depolarize cannot hyperpolarize again because there isn't sufficient glucose to produce the ATP needed for the ion pumps.\nThe previous was only for hypoglycemia.  If your blood glucose in normal, i.e. you are not diabetic, then your blood glucose levels should *never* drop below a certain level-resting, or fasting, blood glucose is carefully maintained in a non-diabetic person.  A drop in blood glucose sufficent to effect the K+ channels would be significantly below normal, and indicate diabetes.  We do work on both diabetic neuropathy and stroke, and the interactions between them.  There are people here that do research on epileptic seizures, and I can ask about the theories on increased seizures in the morning, if you are interested.
280564
calcium is maintaind in the blood by the action of parathyroid hormone and calcitrol\nReceptors monitor the level and if it is low, parathyroid acts on bone to release calcium and increase intestinal absorption, if it is high the hormones act on bone to bind more calcium and the gut to stop absorbing it
158907
Calcium and Phosphorus levels are regulated by calcitonin from the thyroid gland and parathyroid hormone from the parathyroid glands.  In a healthy individual, calcium and phosphorus have an inverse relationship, that means that when one goes up, the other goes down.  Serum calcium tends to decrease i people who have osteoporosis or inadequate intake of calcium.  Calcium needs Vitamin D to aid in absorption.  Eat foods high in calcium, or take supplements with both Vit D and Calcium. Calcium supplements should be taken 1-2 hrs after a meal to increase intestinal absorption.\nIs your low calcium-high phosphorus levels caused by an underlying condition, such as renal failure, Crohn's disease, or chronic inflammatory bowel disease?  Is it caused by improper functioning of the thyroid and parathyroid glands? If so, the underlying condition must be treated in order for the mineral balance to be restored, thus allowing more calcium to be absorbed.\nSee your Doctor, to treat the underlying problem, then add calcium and Vit D to your diet.
457667
it is called cramp,i bet she has fluid in her long and she has the imbalance of certain minerals.please give a full blood test to check for +K,+CA,+NA,then i can tell you
683371
Kerrie-\n\nWow, that's a big question.  First of all I'm a parent, not a neurosurgeon so don't take this as gospel.  My daughter is almost five and has spina bifida, hydrocephalus and Chiari-2 malformation.  She also has a g-tube (more on that later) but has not suffered from seizures.  She has had the Chiari surgery (it's called decompression) at a very young age-about 13 mos-because her Chiari is pretty pronounced and prior to the surgery she would quickly become sympomatic if her shunt failed (I think we're on about the 20th shunt!).  She was also de-tethered at the beginning of last year because of severe lower back pain.\n\nBasically Chiari means that the brain is shifted down and back in the skull.  This forces the brain stem down into a sort of funnel where it connects to the spinal cord.  When pressure builds in the brain the stem is compressed and nerves are temporarily or permanently damaged.  Typically the nerves that are affected control upper neck and head stuff- breathing, swallowing, vocal cord mobility, etc.  My daughter's cords were paralyzed at 6 mos which caused stridor and aspiration of small amounts of food- hence the g-tube.  During the Chiari decompression surgery bone is removed from around the brain stem making more room for the stem- when the pressure increases there is less effect on the nerves in the stem because there is more room.\n\nTethered cord means that the cord is attached to either the spinal column or scar tissue (in the case of spina bifida).  As the person grows the cord is stretched instead of floating in the CSF in the column.  Stretching the cord usually causes a loss of mobility but can cause back pain.  Actually you are the first parent I've heard from whose child is experiencing pain like my daughter did- usually mobility is the issue.\n\nIs your son suffering from lower back pain?  I haven't heard of Chiari causing back pain but I'm a salesman, not a neurosurgeon.  What I have learned is that the brain and spinal cord are all floating in one interconnected and balanced system of fluid.  Pressure building in one part of the system can cause issues with other parts of the system.  Unfortunately I've also learned that neurosurgery is basically a science of highly educated opinions.  Only so much can show up on an MRI and there are no hard-and-fast rules.  Some kids can tolerate a lot of ventrical pressure and some can't.  Some kids have severe Chiari but are never decompressed because they never show any symptoms- basically their nervous systems can just deal with it.  Some kids lose mobility when their cords are tehtered and others are in pain... you get the point.  At the end of the day it comes down to how much you trust and respect the surgeon and what they are basing their actions on.\n\nI still haven't answered your question, have I?  If your son is experiencing back I would think it's probably caused by the tethered cord so it makes sense that the surgeon wants to do that first.  It really doesn't matter, anyway.  If the cord is tethered that has to be fixed whether it's the cause of the pain or not.  For my daughter the de-tethering was much more difficult to recover from than the decompression surgery but the surgeons were clear that because of the amount of scar tissue from her spina bifida repair that it was probably the most complicated one they had seen.  The decompression was much simlpler- surgery took a few hours and she was home in a week.  It took her a while to get full mobility back in her neck (the surgery is done at the base of the neck) but that was just from the surgical pain.  The Chiari symptoms eased almost instantly and the whole thing was behind us in a month or so.  De-tethering was a much longer procedure with 6+ weeks in the hospital but again her procedure was VERY complicated because of her unique circumstances.  The pain went away after the surgery healed and two months after surgery she wa
293390
Lack of vitamin D causes rickets (insufficient deposition of salts resulting in soft bones). That's the most important you propably wanted to hear. I don't remember anything special about the other vitamins with respect to bone development.
848780
hmmm....there could be many reasons why this is happening. It could just be the way your system works, or it could be a medical problem like overactive thyroid or even Crohn's disease.  Please make an appointment with your doctor and discuss this with him/her.  It's probably nothing, but they're the health care professionals and this is their field of expertise.  I would strongly recommend finding out why this happens to you.
194758
It increases the neuronal firings in the areas the stimulant affects. It makes your central nervous system work more.
714952
THE ROLE OF NUTRIENTS IN HUMAN HEALTH - MINERALS \n\n(Foresight Article)\n\n\nCALCIUM\n\nCalcium is needed for the formation of strong bones and teeth, and for controlling blood clotting mechanisms and to regulate the excitability of nerves and muscles. Sufficient calcium is thought to protect against allergies, viruses and tooth decay.\n\nLack of calcium can cause rickets, back pain, osteoporosis, osteomalacia, insomnia, irritability, nervousness, tension, uneven heartbeat, indigestion, stomach cramps and spasms, constipation, pre-menstrual tension and cramping of the uterus.\n\nCalcium is lost from the bones during bedrest and with people on high-protein diets. Invalids and people on high-protein intakes should supplement with dolomite tablets or bone-meal, and increase dietary sources of calcium.\n\nCalcium can be used as a natural painkiller and some authorities advise taking calcium and Vitamin D in the week prior to menstruation, and at the onset of labour.\n\nCalcium is found in: \nMilk, Cheese, Bone broth, Green veg, Egg, Dolomite, Bone meal.\n\n\nMAGNESIUM\n\nMagnesium is needed for the production and transfer of energy, for muscle contraction, nerve excitability and protein synthesis. It also assists in the function of many enzymes. \n\nLack of magnesium brings involuntary muscle movements - gross tremor and twitching - convulsions and delirium. Insomnia, irregular heartbeat, leg and foot cramps and bedwetting can occur. Calcium deposits are found in the arteries and stones may form in the kidneys.\n\nMental symptoms include depression, poor memory, confusion, disorientation, hyperactivity, irritability, anxiety and an increased sensitivity to noise.\n\nProblems with the foetus include congenital abnormalities, calcium deposits and other abnormalities of the heart cells. The newborn may suffer anorexia, convulsions and perinatal death. \n\nOnly 20% of the magnesium is left in refined (white) flour, and only 17% is left in polished rice.\n\nMagnesium is found in: \nMilk, Eggs, Green veg, Seafood, Whole grains, Dolomite.\n\n\nPOTASSIUM\n\nPotassium is needed to regulate blood pH, keep the water balance, ensure the acidification of the urine, and help nerve conduction and muscle contractions.\n\nA shortage brings nervous irritability, mental disorientation, fatigue, listlessness, insomnia, muscle weakness, irregular pulse, constipation, cramping of muscles, low blood sugar and coma.\n\nA shortage can be induced by diuretics.\n\nPotassium is found in:\nWhole grains, Wheat germ, Soya flour, Nuts, All fresh fruit, All vegetables.\n\n\nCOPPER\n\nCopper aids the development of brain, bones, nerve and connective tissue.\n\nLack of copper can cause porous bones, loss of hair, demyelination, heart damage and anaemia.\n\nCopper deficiency in the foetus can result in depressed growth rate, depigmentation, anaemia, fine fragile bones, ataxia, small brain and perinatal mortality.\n\nCopper deficiency is rare however, and copper in excess can be toxic. Copper water pipes, especially new ones, or even old ones in areas where the water is soft and acid, may increase the copper load. Women who are on the contraceptive pill, some fertility drugs or are using the copper coil tend to accumulate copper. Copper jewellery and cooking utensils or kettles can also increase the copper load. The Foresight Clinicians advise hair analysis to check the copper level.\n\nOnly 37% of the copper is left in white flour, and only 75% in polished rice.\n\nCopper is found in:\nWhole grains, Liver, Kidney, Brain, Green veg, Mushrooms, Gelatin, Dried Yeast, Oysters, Nuts & Seeds.\n\n\nIRON\n\nIron is needed to make haemoglobin, the substance in the red blood cells which carries oxygen in the blood. It aids resistance to infection.\n\nShortage of iron can lead to weakness, shortage of breath, excessive fatigue, depression, mental confusion and poor memory.\n\nIron deficiency in the foetus can cause eye defects, bone defects, brain defects and neonatal mortali
84862
that would be because of the excess water-soluble vitamins that ur body is trying to eliminate so as to maintain homeostasis and prevent the complications of having too much of these vitamins
1181302
It is when your body isn't pulling out the vitamins from what you are eating. It isn't a good sign. If it is still happening after a few days, go see your doctor.
261271
reasons can be\n1)lack of vitamin K\n2)lack of calcium ions
826883
In hyperthyroidism,the body's metabolism is very much more quicker than normal person,so much heat is produced and there's  a lot of sweating.\nSweating leads to decease in body water (water loss is more than loss of salts) and increase in tonicity of blood,which all leads to secretion of angiotensin 2 which stimulate the thirst center in the brain to cause thirst sensation.\nAldosterone don't cause thirst.Angiotensin 2 also stimulate secretion of aldosterone too.Aldosterone is responsible for reserving body fluids and salts by decresing urine volume.
373625
stimulation from other nerver endings that are not "feeling pain" cause Ca++ to be released, allowing for analesic neurotransmitters to be released to interneurons, competng with pain causing effects from other places.