What environmental factor have  the development of juvenile diabetes or diabetes type 1?
403541
There is a theory that type 1 diabetes gets triggered in people who already have a tendency to develop the disease. They say people with certain tissue types are more likely to become diabetic, and you inherit these tissue types from your ancestors. A viral infection of some kind has been suggested as the trigger (maybe even the flu? or a cold?). The reason this is unusual is that one would expect such a deadly disease to get less common over time, rather than more common as it is. It is proposed that the diabetic tissue type offered some advantage to people living a marginal existence (hunting for food), and that this explains why more people are diabetic in modern times. Maybe modern living is too easy? (I know, it doesn't seem too easy to me either.)
161933
Diabetics has type I(A) and Type II (B). Mostly type II are related to environment. The S/S like thirsty, fading,tired, sleepy all the day, someone told you that your oral odour smell like sugar... \n\nWhen more severe, skin itchy, eye problem may happen.\n\nyou can consult with your physician or perform some blood glucose examination to see whether your blood glucose level is high or low
1096743
=========\ninterference with insulin production \n\nA sufficiently serious infection of the pancreas could cause enough destruction to induce Type 1 diabetes directly (destroyed beta cells, no insulin production). And, since Type 1 is most commonly an auto-immune disease in which the immune system has attacked and destroyed the insulin producing beta cells, anything which can start this misdirected response will do. Generally, it's thought  that it is viral infections which are mostly at fault: German measles, several of the Coxsackie family viruses, etc. It is probably not impossible that some bacterial infection could do more or less the same thing, but it's not generally seen clinically. \n\n=====================\nassociated issues\n\nThe increased glucose levels in the blood of diabetics are a help to many bacteria, so it's likely they will have an easier time being active in diabetics. And, diabetics very often have impaired  circulation which helps some bacterial infections since the immune system has reduced access to them. If this is bad enough, it can lead to the too common infections which are the cause of gangrene and of amputation. And, finally, there are some reports that diabetics' immune systems are somewhat less effective than non-diabetics'. But none of these are, quite, 'increased development of diabetes' due to bacterial infections.
888441
Here is a helpful website, hope it is helpful:\n\nhttp://ndep.nih.gov/diabetes/prev/prevention.htm
11679
Type 2 diabetes almost always has a slow onset (often years), but in type 1, particularly in children, onset may be quite fast (weeks or months). Early symptoms of type 1 diabetes are often polyuria (frequent urination) and polydipsia (increased thirst, and consequent increased fluid intake). There may also be weight loss (despite normal or increased eating), increased appetite, and irreducible fatigue. These symptoms may also manifest in Type 2 diabetes in patients who present with frank poorly controlled diabetes.\n\nThirst develops because of osmotic effects — sufficiently high glucose (above the 'renal threshold') in the blood is excreted by the kidneys but this requires water to carry it and causes increased fluid loss, which must be replaced. The lost blood volume will be replaced from water held inside body cells, causing dehydration.\n\nAnother common presenting symptom is altered vision. Prolonged high blood glucose causes changes in the shape of the lens in the eye, leading to blurred vision and, perhaps, a visit to an optometrist. All unexplained quick changes in eyesight should force a fasting blood glucose test. These are now quick (less than 5 minutes total), inexpensive (materials less than USD $1), and can be safely performed by almost anyone with trivial training.\n\nEspecially dangerous symptoms in diabetics include the smell of acetone on the patient's breath (a sign of ketoacidosis), Kussmaul breathing (a rapid, deep breathing), and any altered state of consciousness or arousal (hostility and mania are both possible, as is confusion and lethargy). The most dangerous form of altered consciousness is the so-called "diabetic coma" which produces unconsciousness. Early symptoms of impending diabetic coma include polyuria, nausea, vomiting and abdominal pain, with lethargy and somnolence a later development, progressing to unconsciousness and death if untreated.
613378
When you eat, your stomach produces glucose that acts like an acid to break down the food, pass it into your blood and absorb it into the body tissues. Insulin packs the food into your body tissues and, at the same time, flushes the excess glucose out of your body. Insulin is made by the pancreas and diabetes occurs when the pancreas fails to produce enough insulin to do these two jobs. Therefore an excess of glucose also builds up in the blood and bercause it is acidic, it eats away at your body's blood vessels. It attacks the liver, kidneys, eye retinas and the toes causing kidney disease, blindness and sometimes requiring amputation of the feet. So, the second part of your question is correct: the cells cannot deliver energy without insulin but there is also a build up of glucose in the blood which attacks the blood vessels.
109589
Type 2 diabetes is a lifelong disease that develops when the pancreas cannot produce enough insulin or when the body's tissues become resistant to insulin. \n\nThis usually develops in adults although it can develop at any age. \n\nType 2 diabetes is caused by insulin resistance, which occurs when the body's cells and tissues do not respond properly to insulin. Your weight, level of physical activity, and family history affect how your body responds to insulin. People who are overweight, get little or no exercise, or have diabetes in their family have an increased risk of developing type 2 diabetes.
173479
Nothing in particular, except smoking. Smoking causes characteristic damage to several body systems, and it reinforces the damage diabetes does. No one needs both at work. Give it up! If you have lost feeling in the feet, it's best by far to check them often for signs of blisters or cuts or skin breaks of any kind. The reason is that, if you can't feel it, you won't take care of it. Diabetes is the largest cause of amputation in adults in theh North America save trauma (like automobile accidents). The reason is largely reduced circulation, and reduced wound healing, and increased infection severity in many diabetics. Watch those feet.   \n\n\nNo food, or food type, is inherently bad for diabetics, and in the case of Type 1 diabetics, there is less concern for various kinds of food than for Type 2 diabetics, for whom excess weight seems to both bring on Type 2, but also to make more severe cases. \n\nThe problem for Type 1s is that, producing no insulin of their own, it's necessary to provide insulin from the outside, and to come as close as possible to the non-diabetic levels of insulin so that blood glucose levels are as near normal as possible. Insulin controls how, and how much, about 2.3 of body cells taek up glucose from the blood. Among much else, of course, for it is a hormone which has wide and varied effects throughout the body. \n\nThere are several things which affect how fast and high blood glucose goes: \n\n--First is how much food is eaten. In this case, sugar (sucrose), milk sugar (lactose), starch (many glucoses stuck together in chains), fat (a small part of fats -- the glycerol backbone -- can be converted to glucose in the body), and so on. All provide glucose, eventually, to the blood. One of the purpsoes of the 'exchange' system is to provide a handy way to estimate the food quantities eaten. \n\n--Second is exercise. Even without insulin, exercise (even mild exercise such as a brisk walk) causes glucose to be uptaken by muscle cells. Given any partiuclar amount of food, exercising before the meal will result in less inculin needed than otherwise, since the mechanisms are independent. teh effect lasts for some hours, so a brisk walk for 30 minutes just after luch will still increase glucose absorprion rates of food eaten at dinner.  \n\n--Third, is the amount of insulin. All other things being equal, more insulin means that more glucose will be absorbed from the blood by body cells and less insulin will mean less. \n\n--Fourth, type of insulin. There are several types of insulin. They vary largely by how long their effect (forcing absroption of glucose from the blood) lasts, and whether there is a marked peak in action. So, a given amount of regular insulin acts faster and more intensely than lente ('slow') insulin which takes longer to exhaust itself, or ultra lente ('very slow') insulin. NPH insulin falls between regular and lente in speed of action adn suration of action. The analog insulins (one from Lilly and one from Novo Nordisk) are much more quickly acting, beginning totake effect with 15 or 20 minutes and being done in an hour or perhaps an hour and a half. Two special insulins, Lantus and Detemir, are unusual in that they exhibit very little peak of action. Their insulin effect  takes place steadily over about 24 hours. \n\nLocation of administration also has a secondary effect on how long the insulin effect lasts. \n\n--Fifth is the composition of the food eaten.  For example, skim milk contains lactose (a mildly sweetish dual sugar) which is disassembled into its component simple sugars and absorbed by the intestines. Drinking the same amount of whole milk delivers the same amount of lactose, but much more slowly since the butterfat in the whole milk slows down digestive action on the lactose. A baked potato eaten 'dry' will be converted to glucose and absorbed, while the smae amount of potato eaten with butter or sour cream is much more slowly a
761824
Type II diabetes: possibly if you eat so much that is causes you to become over weight.\nType I diabetes: ABSOLUTELY NOT!!!  Type I diabetes is caused by the Beta cells in the Islets of Langerhans dying or not being able to produce insulin as a result of cellular damage or from them being attacked by the body's own immune system, it has absolutely nothing to do with diet and almost all to do with heredity and genetics.
406260
Help find a cure for Juvenile Diabetes.
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Both my brother and his wife have Type II diabetes.  They had a baby 05/20/04.  He was 8 weeks early because she developed pre-eclampsia (high blood pressure) and her kidneys began failing.  They had to induce labor to save her and my nephew.\n\nThey are both fine now.  He is into his terrible twos and not diabetic.
1060938
Yes, a friend of mine didn't know she had Type I diabetes until she got pregnant at 26! But, she's gotten used to it and just had her 3rd baby!!!
170655
The Cause of Type 1 Diabetes\n\nAutoimmune reactions, in which the body's white blood cells harm its own tissues, cause many diseases including type 1 diabetes, multiple sclerosis, Hashimoto's thyroiditis and arthritis, among others. We don’t known why immune cells target certain organs, and why in type 1 diabetes, only insulin-producing cells are killed.\n\nIn type 1 diabetes, the body's immune T-cells react against beta cells -- the insulin-producing cells in the pancreas -- effectively shutting them down and triggering disease onset.\n\nSo, it is known that type 1 diabetes is caused by the body's immune system turning against cells in the pancreas that produce insulin. What's remained unclear is the target for this immune response.\n\nSome studies report that albumin, the protein in milk, is a causal agent for that immune reaction. Also some theories point toward viruses. The glutamic acid decarboxylase is another molecule that has been believed to be the cause for that reaction.\n\nNow, after eight long years of careful research, scientists believe they've finally pegged insulin as the prime antigen -- immune system target -- responsible for this shutdown.\n\nDr. David A. Hafler, Breakstone professor of neurology at Harvard Medical School guided the research, which appears in the May issue of Nature. Unfortunately, the only way to adequately answer that question in humans is to examine tough-to-obtain pancreatic lymph tissues.\n\n"It took us years to get these tissues, to clone the cells and then to really characterize them and examine their activity," he said. “The results of that effort show that indeed the target -- the cause -- of type 1 diabetes may be T-cell reactivity to insulin," Hafler said.\n\nHis team in Miami has been testing insulin as a potential basis for a vaccine against type 1 diabetes. However, another expert in type 1 diabetes research, Edwin Gale --who is professor of diabetic medicine and head of the department of clinical science at the University of Bristol in England-- cautioned that a potent vaccine against the illness remains a distant goal.\n\nThese findings are very interesting for people with type 1 diabetes, but not for those with type 2 diabetes, since although in both cases the result is hyperglycemia, the causes are different. Reports Health Day News\n\n\nWhat Causes Type 2 Diabetes\n\nType 2 diabetes affects about 200 million people worldwide and has a predicted increase to 300 million by 2025. Also, nowadays more and more children and adolescents are developing Type 2 diabetes, which used affect mostly adults in the past.\n\nRecent studies reveal that genetics, obesity, fetal history, lack of activity, a diet high in fat and stress, all may play a role in the development diabetes.\n\nGenetic studies have shown that the association of some genes is the root of the cause of Type 2 diabetes. A healthy and active lifestyle for the whole family of a person with diabetes is then greatly recommended to protect their children and young people.\n\nLow birth weight has also been associated with Type 2 diabetes. It seems that poor nourishment of the fetus increases the risk of insulin resistance and Type 2 diabetes.\nIf that baby is overfed and becomes overweight during childhood, then the risk is a lot higher.\n\nOn the other hand, stress has been associated with insulin resistance, which leads to Type 2 diabetes. This happens due to the cascade of metabolic reactions that our emergency hormones trigger when we are stressed.\n\nObesity has long been associated with diabetes, as well as the lack of physical activity and a diet high in fat. This is thought to cause a condition called insulin resistance.\n\nWhat is insulin resistance?\n\nInsulin resistance means that your muscles, fat, and liver cells cannot use insulin properly. There can be a lot of insulin circulating, but the cells are incapable of using it to allow glucose entering the cells and be conv
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First of all contact your insurance company. They can be a valuable resource. Second, contact your local American Diabetes Association chapter, they can provide you with endocrinologists along with a vast wealth of information. Finally, visit www.childrenwithdiabetes.com there is some GREAT help available there.\n\nPlease feel free to email me with any other questions...I've been in your shoes. Hang in there, ok?
479324
let it be very clear that there are two types of diabetes \n1. type I IDDM\n2. type II NIDDM\n\ntype I or insulin dependant diabetes mellitus is caused by relative or absolute deficiency of insulin\n\ntype II or non IDDM is caused by decrease in insulin receptors in the body or its resistance. it is linked to obesity . it is usually not treated with insulin. there are certain drugs which reduce the absorption of carbs , these are used.\n \ndon't think to reduce insulin dependance, just follow what your doctor says, because the function of insulin is  a very complex mechanism and it not only affects your carbohydrate metabolism but also fat, protein and many other things.
224076
Type I diabetes is caused by genetic susceptibility alone. No one who has the type got it through being lazy or having a bad diet. There was no way to prevent it.\n\nType II is a lifestyle disease caused by susceptible people not taking care of themselves, eating a high carb/sugar diet and not exercising properly. The susceptibility is inheritable; however, the lifestyle choices belong exclusively to the individual.\n\nThe short answer is that, yes, diabetics can be a genetic disorder.
45764
No type one diabetes cannot be diagnosed until age 2 or 3