Can a person with HBSAG inactive carrier marry and lead a healthy life?
375178
Hepatitis B surface antigen (HBsAg) is a protein antigen produced by HBV. This antigen is the earliest indicator of acute hepatitis B and frequently identifies infected people before symptoms appear. HBsAg disappears from the blood during the recovery period. In some people (particularly those infected as children or those with a weak immune system, such as those with AIDS), chronic infection with HBV may occur and HBsAg remains positive. \nSometimes, HBV goes into “hiding” in the liver and other cells and does not produce new viruses that can infect others, or produces them in such low amounts that they cannot be found in the blood. People who have this form are said to be carriers. In other cases, the body continues to make viruses that can further infect the liver and can be spread to other people. In both these cases, HBsAg will be positive. The next test is helpful for distinguishing these two states. \n\nHepatitis B e-antigen (HBeAg) is a viral protein associated with HBV infections. Unlike the surface antigen, the e-antigen is found in the blood only when there are viruses also present. When the virus goes into “hiding,” the e-antigen will no longer be present in the blood. HBeAg is often used as a marker of ability to spread the virus to other people (infectivity). Measurement of e-antigen may also be used to monitor the effectiveness of HBV treatment; successful treatment will usually eliminate HBeAg from the blood and lead to development of antibodies against e-antigen (anti-HBe). There are some types (strains) of HBV that do not make e-antigen; these are especially common in the Middle East and Asia. In areas where these strains of HBV are common, testing for HBeAg is not very useful. \n\nAnti-HBe is an antibody produced in response to the Hepatitis B e antigen. In those who have recovered from acute hepatitis B infection, anti-HBe will be present along with anti-HBc and anti-HBs. In those with chronic hepatitis B, usually anti-HBe becomes positive when the virus goes into hiding or is eliminated from the body. In strains that do not make HBe antigen, anti-HBe is also positive.\nwww.labtestsonline.org/understanding/analytes/hepatitis_b/test.html \nSo from this and my slight background knowledge, it depends on your Hepatitis B e-antigen (HBeAg) reveals.\n\nDiscuss your concerns with your MD.
736067
As you can see by all the A$$ holes with their opinions, instead of answers, you have not chosen an easy road.   My husband has been in federal prison for 26 years, however, so I wish you the best.    You will have a very strong marriage if you can survive his incarceration.    It will test you and your family beyond anything most marriages have to deal with.\n\nAnna M. gave you the best advice.  However, some institutions are run more professionally than others.   Some will simply not respond to your request.   So  I would also advise to check with the state laws and county requirements, too, like blood tests, waiting periods, and so forth.  You can find all that information with a little searching on the internet for county offices.   I also found that calling the county offices yielded correct information for your circumstances, as they are usually familiar with those procedures in small prison towns.     \n\n At least he's in State.   Federal is awful.   I know of several couples who have waited over a year, with one woman traveling 600 miles each time to get renewed blood tests and licenses because of bureaucratic bull.   But she stuck with it.   So good luck to both of you.
729275
I dunno but I think I'd consider dropping him.  You don't want Hep. B, I assure you.  I'd rather have HIV.  Hep. B is a terminal disease and you need to avoid it at all costs.  So while he's still your "boyfriend", drop him.
761535
general anaesthesia  presents risk and certain danger to anyone, in particular to those with chronic health problems.  a carrier of the sickle cell trait isn't really a chronic health problem and i would have to agree with your surgeon......he knows best.  but if you are that concerned, get another opionion, say with a hemotologist, specializing in blood disorders.
985978
Here is more than you ever wanted to know about Hepatitis B. Hope this helps you. I urge you to visit the Hepatitis B Foundation Website. Click on the module for an interactive course on Hep B.\n\nHepatitis B is a serious liver infection caused by the hepatitis B virus (HBV). Although most people exposed to the virus recover completely, chronic infections can lead to severe liver damage, cirrhosis (scarring of the liver), liver cancer and death.\n\n\nHow is hepatitis B transmitted?\nHepatitis B virus is spread by contact with body fluids of an infected person. These body fluids include blood, semen, vaginal secretions, and saliva. In about 30-40% of cases, the means of transmission is not known.\n\n\nWhat is the risk of hepatitis B?\nThere are approximately 80,000 new cases of hepatitis B infection each year in the United States. About 90-95% of adults recover from hepatitis B in a few months, clearing the virus from their systems and developing immunity. They will never get hepatitis B again: however, blood tests will always show that they were once infected, and blood centers will not accept donated blood from these individuals. Up to 10% of young adults who get the disease become carriers – as many as 1.25 million Americans are hepatitis B carriers. Carriers are people who have the virus in their blood and can infect others, even though they do not feel sick and have no obvious signs or symptoms of hepatitis B.\n\nPeople infected with hepatitis B who are unable to clear the infection from their bodies in six months are chronically infected with hepatitis B. The younger the person when infected, the greater the risk that the disease will become chronic. Chronic hepatitis B means that the virus is in the blood, infecting liver cells and possibly damaging them. Chronic hepatitis B can lead to cirrhosis, primary liver cancer, liver failure and/or death. An estimated 5,000 people die each year of chronic liver disease associated with HBV infection.\n\nPeople who are exposed to blood or body fluids of an infected person are at risk. You may also be at risk if you:\n\n\n\n\nAre exposed to bodily secretions, including on the job – as first aid or emergency workers, firefighters, funeral directors, police personnel, dentists, and dental assistants, and medical personnel\n\nLive in the same household with an infected person\n\n\nHave sex with a carrier of chronically infected person or have more than one sexual partner\n\n\nInject illicit drugs\n\n\nReceived a blood transfusion prior to 1975 (when a test to screen blood was developed) or if you have received multiple transfusions of blood or blood products\n\n\nHave hemophilia\n\n\nWork or are a patient in a health care or long-term care facility\n\n\nWork or are incarcerated in a prison\n\n\nAre bitten so that the skin is broken by someone whose saliva contains the virus\n\n\nTravel to countries with a high incidence of hepatitis B\n\n\nAre a hemodialysis patient\n\n\nHow is hepatitis B diagnosed?\nThe most common symptoms of hepatitis B occur within 25-180 days following exposure and include loss of appetite, nausea and vomiting, fever, weakness or tiredness lasting weeks or even months, abdominal pain in the area of the liver (upper right quadrant), dark urine and light-colored stool, joint pain, and yellowing skin and eyes (jaundice).\n\nAn estimated 40% of people infected with hepatitis B have no recognizable signs or symptoms and do not know how or when they became infected. The only way the disease can be positively identified is through a blood test. Many people are surprised to learn when they donate blood that they have tested positive for hepatitis B. Hepatitis blood tests are not usually included in the routine blood tests of a physical examination.\n\nThe blood tests for hepatitis B may include anti-HBs (surface antibody test), HBsAg (surface antigen test), and anti-HBc (core antibody test). Although anti-HBs and anti-HBc may be found in the blood of individuals who
245441
You would be surprised at some of the reasons some people marry. But being able to tolerate them is a priority. \nStrange. LOL
1020119
I personally don't know how it feels to have diabetes but I do know how it feels to be married to someone who has Type I diabetes and to be related to others with Type II.  I am really not sure what you want to know or exactly what you are asking.
236409
hepatitis B is a disease that can be acquired thru unprotected sex, blood transfusion, needle sharing (drug users...exposure to blood)or organ transplant from infected individual.\nYour test will be non-reactive at a certain point. your body is producing antibodies to each hepatitis marker and they will be the determinant factor if you are still infectious or not. sadly, there is no known cure for hepatitis B. although your test may show negative to the antigen, you can still be a carrier.\nthe best way is to live a healthy life. as long as your body is healthy, you have little to worry. \nand the most important thing is...consult your doctor and have him do a work-up.
1067528
Sickle cell anemia is inherited as an autosomal recessive trait.  This means that in order for a child to have sickle cell in its full blown form, (s)he must inherit the trait from BOTH parents. If two carriers of the trait marry, these are the odds:\n\nBoth parents are Ss, so Ss crossed with Ss yields a 50% chance that a child will be a carrier (have sickle cell trait), a 25% chance that a child will be normal (not have any sickle cell at all in their genes), and a 25% chance that a child will have sickle cell disease. \n\nIf a person with sickle cell marries a normal person, then all of their children will be carriers (have the trait).\n\nIf a person who is a carrier marries a normal person, then 50% of the children will have the trait and 50% will be normal.\n\nIf a person who is a carrier marries a person with sickle cell,  50% of the children will be carriers, and 50% will have the disease.\n\nIn the unlikely case that two people with sickle cell disease marry, then ALL of their children will have the disease.\n\n\nNote that these numbers apply to EACH pregnancy: no matter what the outcome of each child, the chances are still the same for any siblings. So the only way a child can inherit the actual sickle cell disease is if the trait is carried by both parents or at least one of them actually has the disease.
239822
Yes it is... My husband and I are both A+ and we have two healthy boys.  There might be a problem if one of the parents have got a negative RH factor, but if the problem is known then the mother is given a shot at the appropriate time and then it is not a risk anymore.
1115203
Can a man have, let's say, B+? Yes.\nCan a woman have, let's say, B+? Yes.\nCan they get married? Yes.\n\nExact same blood type will not be a problem. Only way for problem is if baby is + and mom is -. Only way for that to happen is if Dad is + and mom is -.
783451
The 2 alleles responsible for whether normal or sickle red blood cells are HbA and HbS. The HbS allele (sickle cell gene) is recessive but more exactly, that allele is incompletely recessive (i.e. in the heterozygote HbAHbS, a few sickle red blood cells are still present besides the normal one, shows that HbS gene are still expressed at small rate). The HbS induces the polymerization of hemoglobin in red blood cells, leading to abnormal sickle shape of the cells, making it very difficult for these red blood cells to pass through the blood vessels and stuck there.\n\nOne person is called suffering from sickle cell anemia if they have the HbSHbS form, i.e. the homozygous recessive. In the HbAHbS the few sickle red blood cells are not enough to cause symptoms. \n\nOne interesting thing about this disease is that, while the HbSHbS form would be dangerous because the red blood cells cannot carry oxygen to cells and stuck in the blood vessels, the carriers (HbAHbS) would have advantage in malaria region. Since the malaria parasite spends parts of its life in red blood cells, these parasite cause the abnormal sickle cells to rupture, moreover, the polymerization of Hb disables the parasites to digest Hb. The white blood cells also kill the parasite better when they come to kill the stuck sickle cells. That why people with HbAHbS form have a higher fitness to the malaria environment than people with normal red blood cells.
1031609
I am an alternative medicinal therapist and an ex nurse. There should not be a health problem with a man this age. You would have a problem if you were 43. If your husband suspects that it is not his child he can ask for a DNA test to be performed. Many people have the same blood group but this type of testing has never been full proof, nowdays they use genetic printing -DNA. Just be carefull how you handle this. \n\nGloria(BSYA)\ngloriashealth@btinternet.com
817043
The answer is no, your chances are not 100%.\n\nIf your parents are Type I then it is likely you will never develop diabetes because Type I has not been genetically tied.  But of course if you are overweight and inactive then you do have a chance of developing Type II diabetes.\n\nIf your parents are Type II diabetics then you do have an increased risk of developing Type II diabetes is you are overweight and inactive.  Also excessive alcohol consumption and smoking can increase your risk if you have a family history of Type II diabetes.\n\nBy no means does your parents’ having diabetes mean you are predestined to have diabetes yourself.  If you manage your weight and keep active it is likely you will live a diabetes free life.\n\n\nJust a personal side note ... my grandfather had Type II diabetes which he developed in his sixties.  Two of my uncles are now diabetes which they developed in their late 40's - early 50's.  Both were heavy drinkers and extremely overweight.  Although both were active at their day to day jobs they had no other physical activity in their lives.  They have 4 other siblings none of which have diabetes.  So for my uncles they did have an increased risk but had my grandfather not had diabetes they may have still developed diabetes because of their lifestyles.  My father who 60, exercises, eats well and is healthy as a horse is extremely unlikely to ever develop Type II diabetes because he takes care of himself.  So I guess what I really want to say is take care of yourself as all of us should and you should live a long healthy disease free life.
139440
An aabb individual can only produce one type of gamete, ab.
150921
there is no known cure for hbv or hcv using this protocol. don't be duped in to buying expensive juices, fasting, flushing ect. (some of these protocols can be dangerous for those with these liver ailments as there are many bi-directional diseases associated with it)even tho there is a slight benefit using this stuff, just following a good diet (no red meat, iron, sodium) is the key. stay away from any liver damaging substances like alcohol, prescription pain meds.\nincrease your fruit intake as well as veggies containing no iron. and see your doctor. interferon is used in both hepatitis viruses. don't be afraid of those who claim the treatment is horrible....i as well as many other sure, had our bad days on treatment, but all in all, we felt much better and most of us dont have hcv anymore!!
506239
Hepatitis B surface antigen (HBsAg) is one of the most frequently performed tests for HBV. This HBV antigen is the earliest indicator of an active hepatitis B infection. This antigen may be present before symptoms of an HBV infection are present. If this antigen level remains high for more than 6 months, then you will probably become a carrier of HBV, meaning you can transmit it to others throughout your life. \nHepatitis B surface antibody (HBsAb) is also one of the most common tests for HBV. Usually this antibody appears about 4 weeks after HBsAg disappears and means that the infection is at the end of its active stage and you cannot pass the virus to others (you are no longer contagious). This antibody also protects you from getting HBV again in the future. The test is done to determine the need for vaccination; the antibody will be present after receiving the HBV vaccine series, showing that you have protection (immunity) from the virus. Occasionally your test may show that you have both the HBsAb and HBsAg antibodies; in this case, you are still contagious. \nHepatitis B core antigen (HBcAg). Currently, there is no test to find this antigen. \nHepatitis B core antibody (HBcAb) is an antibody to the hepatitis B core antigen. This antibody appears about 1 month after an active HBV infection. It can be found in people who had an infection in the past and in those with long-term (chronic) HBV. It usually is present for life. \nHepatitis B core antibody IgM (HBcAbIgM) is also an antibody to the hepatitis B core antigen. It shows a recent infection in the last 6 months. \nHepatitis B e-antigen (HBeAg) is an HBV protein that is only present during an active HBV infection. This test determines how contagious you are. Testing for this antigen can also be used to monitor the effectiveness of treatment for HBV. \nHepatitis B e-antibody (HBeAb) shows that the active stage of the HBV infection is almost over and your risk of being contagious is greatly reduced. HBeAb is usually present during chronic HBV infections. \nHBV DNA testing finds genetic material (DNA) from the hepatitis B virus. Currently, quantitative HBV DNA tests are done. A high HBV DNA level means that the virus is multiplying in your body and you are very contagious. If you have a chronic HBV infection, an elevated viral DNA level means you are at an increased risk for chronic hepatitis and may want to consider treatment with medications. Testing for HBV DNA also is important to monitor the effectiveness of treatment for chronic HBV infection. HBV DNA testing is a more sensitive test than HBeAg (above) for detecting HBV in the blood