Is there any treatment to sickle cell anemia?
374512
There is no specific treatment for sickle cell anemia. People with this disease should avoid high altitudes, maintain adequate fluid intake, and treat infections promptly. \nA bone marrow transplant is the only available treatment that can cure the disease, however, its use remains uncommon. \nDuring a sickle crisis, certain therapies may be necessary. Painful episodes are treated with analgesics and adequate liquid intake. Treatment of pain is critical. Non-narcotic medications may be effective, but some patients will require narcotics. \nHydroxyurea (Hydrea) was found to help some patients by reducing the frequency of painful crises and episodes of acute chest syndrome and decreasing the need for blood transfusions. There has been some concern about the possibility of this drug causing leukemia, but as yet there are no definitive data that Hydrea causes leukemia in sickle cell patients. \nThe main thing is to try to avoid sickle cell crisis, which include steps to reduce the chance of hypoxia, such as climbing high altitudes, and seeking medical attention quickly to counteract problems such as upper respiratory tract infections.
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there are many ways to treat it but not cure it.\nhttp://www.arthritis.org/
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Hi,\n\nI don't think there is a cure for this disease but there are several drugs available and coming through the system that may help ease the problems or slow them down.  \n\nTry these links:\n\nhttp://www.clevelandclinic.org/health/health-info/docs/2100/2112.asp?index=8877\nhttp://armor.typepad.com/bastardsword/2004/08/potential_cure_.html\nhttp://www.medicalnewstoday.com/medicalnews.php?newsid=35418
724539
Sickle-cell disease is a general term for a group of genetic disorders caused by sickle hemoglobin (HbS). In many forms of the disease, the red blood cells change shape upon deoxygenation because of polymerization of the abnormal sickle hemoglobin.\n\nHbN and HbS are alleles which are alternative forms of gene. All humans have 2 alleles for every gene (one from each parent).\nNormal healthy people have HbN HbN (HbN=normal hemoglobin).\nCarriers have HbN HbS (HbS=sickle hemogloblin).\nPeople suffering from sickle cell anaemia have HbS HbS. \n\nIn people heterozygous for HbS (carriers), the polymerization problems are minor, hence they don't suffer the effects of sickle cell anaemia. In people homozygous for HbS, the presence of long chain polymers of HbS distort the shape of the red blood cell, from a smooth donut-like shape to ragged and full of spikes, making it fragile and susceptible to breaking within capillaries. Carriers only have symptoms if they are deprived of oxygen (for example, while climbing a mountain) or while severely dehydrated.\n\nThe sufferers of the illness have a reduced life span. It is believed that carriers (sickle cell trait) are relatively resistant to malaria. Since the gene is incompletely recessive, carriers have a few sickle red blood cells at all times, not enough to cause symptoms, but enough to give resistance to malaria. Because of this, heterozygotes (HbS HbN) have a higher fitness than either of the homozygotes (HbN HbN & HbS HbS). This is known as heterozygote advantage. Those with HbN HbN are susceptible to malaria while those with HbS HbS will suffer from sickle cell anaemia.  \n\nThe malaria parasite has a complex life cycle and spends part of it in red blood cells. In a carrier, the presence of the malaria parasite causes the red blood cell to rupture, making the plasmodium unable to reproduce. Further, the polymerization of Hb affects the ability of the parasite to digest Hb in the first place. Therefore, in areas where malaria is a problem, people's chances of survival actually increase if they carry sickle cell anaemia.\n\nDue to the above phenomenon, the illness is still prevalent, especially among people with recent ancestry in malaria-stricken areas, such as Africa, the Mediterranean, India and the Middle East. In fact, sickle-cell anemia is the most common genetic disorder among African Americans; about 1 in every 12 is a carrier.
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Laser Photocoagulation and Photodynamic therapy.\n\nthough there is no cure for it.
662946
I don't think there is any. Check out this link:\nhttp://en.wikipedia.org/wiki/Vitiligo#Treatment
85994
Anemia is not a disease, its a condition. \nDiseases that cause anemia (Thalassemia, sickle cell) have support groups. There is no need for a support group for an acute condition.
1013268
If you are looking for a treatment for Hep B\n\ncheck this link\n\nhttp://www.hivandhepatitis.com/hep_b/treatment.html
387409
Hi there.. I'm a Licenced Dietetic Technician. The answer to your question....Anemia is a concern that can be fixed. Anemia is a condition in which the iron count in your blood is low meaning you have a hard time transmitting oxygen in the blood to other body parts. An easy solution is to take a simle daily supplement of 18mg/daily for 2 weeks. Then have your HGB and HCT checked again to see if the Anemia has improved. Make sure you also take a Vit C supplement with the Iron to help absorb the iron. Hope this helped. Your should have more energy because you'll have more oxygen flowing through your blood! Good luck and if you have anymore health questions feel free to ask. -Elizabeth Segler DTR/LDT
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Gentle exercise for your circulation, and iron tablets should help. But you should ask the doctor to try and find out the cause of your anaemia.
1110367
yeah it really works.. but u'd better consult with a doctor first because it's not recommended for people who have anemia or who are hypotensive
992303
talk to a doctor - there are prescription treatments out there
202994
Try:\nhttp://www.nlm.nih.gov/medlineplus/ency/article/001177.htm#Treatment
254574
Common signs include:\n\npaleness \nyellow eyes/skin \ngrowth retardation \nTests commonly performed to diagnose and monitor patients with sickle cell anemia include:\n\nComplete blood count (CBC) \nHemoglobin electrophoresis \nSickle cell test \nPatients with sickle cell may have abnormal results on certain tests, as follows:\n\nperipheral smear displaying sickle cells \nurinary casts or blood in the urine \nHemoglobin; serum decreased \nelevated bilirubin \nhigh white blood cell count \nelevated serum potassium \nelevated serum creatinine \nblood oxygen saturation may be decreased \nCT scan or MRI can display strokes in certain circumstances
923777
Sickle cell disease is an inherited disorder of the red blood cells characterized by abnormally shaped red cells. This abnormality can result in painful episodes, serious infections, chronic anemia, and damage to body organs. These effects can, however, vary from person to person depending on the type of sickle cell disease the person has.\n\nSome individuals are relatively healthy and others are hospitalized frequently. But thanks to advancements in early diagnosis and treatment, most children born with this disorder grow up to live relatively healthy and productive lives.\n\nA Closer Look at Sickle Cell Disease\nThe different forms of sickle cell disease are determined by the genes that are inherited from the person's parents.\n\nAn individual has the disease if he or she inherits a sickle cell gene from each parent (hemoglobin SS disease, also called sickle cell anemia).\n\nHe or she can also inherit a sickle cell gene from one parent and a different kind of abnormal gene from the other and end up with a different form of sickle cell disease, such as hemoglobin SC disease and hemoglobin S-thalassemia.\n\nIf an individual inherits only one sickle cell gene and a normal gene from the other parent, then he or she will have the sickle cell trait, but not the disease. A blood test can determine whether you have sickle cell disease or carry the sickle cell trait.\n\nPeople with sickle cell trait don't have sickle cell disease or exhibit any signs of the disorder, but they can pass the gene for the disease to their children. When both parents have the sickle cell trait, there's a 25% chance that a child will have sickle cell disease. But when one parent is carrying the trait and the other actually has the disease, the odds increase to 50% that they will pass the disorder on to their child.\n\nWho Does It Affect?\nIn the United States, hemoglobin SS disease (sickle cell anemia) affects mostly African Americans and some Latino Americans. Some forms of sickle cell disease may occur, although less frequently, in people with different ethnic backgrounds such as those whose ancestors came from Mediterranean countries (including Turkey, Greece, and Italy) or East India.\n\nWhat Causes It?\nA child with sickle cell disease has inherited a defective hemoglobin gene. In a child with sickle cell disease, the hemoglobin can take on an abnormal shape, distorting the shape of the red blood cell. The cells change from a normal round, doughnut shape to the elongated shape of a sickle, or the shape of a "C."\n\nUnlike normal red blood cells, which move easily through small blood vessels, sickle cells are hard and pointed. The sickle shape means that they have a tendency to get stuck in narrow blood vessels and block the flow of blood. This can cause episodes of pain and can also lead to damage of the body organs because they aren't getting enough oxygen.\n\nSickle cells have a shorter-than-normal life span, which leads to anemia (low red blood cell count). A normal red blood cell lives for about 120 days, whereas a sickle cell lives for only 10 to 20 days.\n\nHow Is It Diagnosed?\nUsually, sickle cell disease is diagnosed at birth with a blood test. This can be done in conjunction with other routine newborn screening tests. If your child tests positive on the screening test, a second blood test is usually performed to confirm the diagnosis.\n\nBecause children with sickle cell disease are at an increased risk of infection and other health complications, early diagnosis and treatment is important. Currently, more than 40 states have newborn screening programs for sickle cell disease.\n\nWhat Are the Signs and Symptoms?\nSymptoms of sickle cell disease vary and range from mild to severe, and symptoms may be less severe or different in children who have inherited a sickle cell gene from one parent and a different abnormal hemoglobin gene from the other.\n\nMost children with sickle cell disease have some degree of anemia, an
612994
I don't agree that the answer is "none" when in fact cord blood stem cells has already proven to be effective. Below is a link for a full list of diseases treated by cord blood stem cells.\n\nIt seems as if more of the successful stories from using cord blood stem cells are for leukemia, lymphoma and sickle cell anemia.
209856
No, there's no known cure.