What is myocardium disease?
142992
Diseases of the myocardium\nThere has been increasing recognition of a type of heart disease characterized as primary myocardial disease. The cardiomyopathies are diseases involving the myocardium (heart muscle) itself. They are unique in that they are not the result of hypertensive, congenital, valvular, or pericardial diseases and are rarely the result of ischemic heart disease
668577
It could be gas or air, or pleurisy - fluid around your heart.  It could also be more serious.  If it persists you should get it checked out.
803525
Dilated cardiomyopathy. This is the most common form of cardiomyopathy. In this disorder, the left ventricle — your heart's main pumping chamber — becomes enlarged (dilated) and its pumping ability becomes less forceful. Although this type can affect people of all ages, it occurs most often in middle-aged people, with an incidence greater among men than women. Some people with dilated cardiomyopathy may have inherited the condition. \n\nHypertrophic cardiomyopathy. This type involves abnormal growth or thickening of your heart muscle, particularly affecting the muscle of the left ventricle. As thickening occurs, the heart tends to stiffen and the size of the pumping chamber may shrink, interfering with your heart's ability to deliver blood to your body. Hypertrophic cardiomyopathy can develop at any age, but the condition tends to be more severe among those diagnosed during childhood. Researchers have identified abnormal genes that predispose people to hypertrophic cardiomyopathy. Most affected people have a family history of the disease. \n\nRestrictive cardiomyopathy. The heart muscle in people with restrictive cardiomyopathy becomes rigid and less elastic, interfering with the expansion and filling of the heart's ventricles with blood between heartbeats or contractions. While restrictive cardiomyopathy can occur at any age, it tends to affect older people most often. It's much less common than the other types of cardiomyopathy. Restrictive cardiomyopathy can occur for no known reason (idiopathic) or may result from diseases elsewhere in the body that affect the heart. One example is amyloidosis, a rare condition in which abnormal proteins present in the blood are deposited into the heart. \n\nIn most people, the cause of cardiomyopathy is unknown. In some people, however, doctors are able to identify a cause or contributing factors, including some that affect the heart and cardiovascular system. For example, any of the following conditions may cause or contribute to a dilated and poorly functioning heart:\n\nSustained high blood pressure \nHeart valve problems \nHeart tissue damage from a previous heart attack \nChronic rapid heart rate \nMetabolic disorders, such as thyroid disease or diabetes \nNutritional deficiencies of essential vitamins and minerals, such as thiamin (vitamin B-1), selenium, calcium and magnesium \nPregnancy \nExcessive use of alcohol over many years \nAbuse of cocaine or antidepressant medications, such as tricyclic antidepressants \nUse of some chemotherapy drugs to treat cancer \nCertain viral infections, which may injure the heart and trigger cardiomyopathy \nHemochromatosis is a disorder in which your body doesn't properly metabolize iron, causing the accumulation of iron in your heart muscle. This can lead to a weakening of the heart muscle, resulting in the appearance of dilated cardiomyopathy.\n\nI hope this helps.  Good luck.
880325
Sounds like "Holiday Heart Syndrome" to me.\n\nIt's a classic medical school scenario.  See the attached links.\n\nBTW, the guy has multiple diagnoses (CHF, Afib, ARF, Alcohol abuse and probably alcoholic cirrosis, CAD, portal hypertension due to the cirrosis, etc) and you basically list all of them in the description, but the one you seem to be looking for is the Holiday Heart.
43842
Angina is a symptom not a disease.  It simply means pain that is associated with heart disease.  Having angina does not indicate the amount of heart disease only it's presence.  \nMany people who have heart disease control their angina with medications while others have surgery, or a procedure called a cardiac catheterization and balloon angioplasty to relieve their angina.\nI think you need more information.  For example, what is the extent of her heart disease.  Has she had a heart attack?  Does she suffer from something called congestive heart failure. Has she had any procedures/surgeries on her heart?\n\nSummary:  Angina by itself does not indicate how long someone will live or how serious their condition is.
342534
the disease may be Angina Pectoris: Pain due to narrowing of coronary arteries secondary to aortic stenosis or low pressure; may also be due to sevgere anemia, hyperthyroidism, or excercise\n\nSymptoms: Crushing chest pain, palpitations, faintness, dyspnea, levine’s sign (fist to chest), diaphoresis, increased BP, abnormal EKG, evident cardiac disease\n\nconsult your doctor.
635530
I am sorry that I haven't the knowledge to recommend anything to you or give you a helpful diagnosis. However, if you feel that the way in which you have been treated has not been the most helpful for your condition, or that you need to resume your treatment, please visit a reliable Dr. \nHeart conditions need to be treated, particularly when they are as noticeable as yours. \nVisit the websites of Medicine Schools, and try to contact someone in the faculty who will be able to help you. Through the Internet, try to locate as many heart doctors as possible. I hope that they can help you. \nGood luck.
751027
if i knew what EF meant it would help. if you're still worried about your heart, ask about a myoview or cardiolite test.
349565
ISCHEMIA - Myocardium ( heart muscle) is alive but inefficient to do its work, because of narrowing of the artery that supply that particular area.\n\nINFARCTION: Death of the mycardium, due to lack of blood supply to that particular area. \n\nCertain type of Ischemia can be reversible, by proper perfusion therapy at proper TIME, but not the all ischemia, and infarction are reversible.
232173
Enlargement of the heart is a sign of serious disease of the muscle of the heart. When disease attacks the muscles of the heart, the ability of the heart to function properly as a pump is reduced. This causes the blood flow to slow and the heart to enlarge. When the heart becomes severely damaged, abnormalities in breathing, beating of the heart, and fluid retention may occur. This process known as heart failure commonly results in patients feeling dizzy and short of breath, with irregular heart beats and swelling of the legs and feet. My husband had an enlarged heart and another congenital defect.  It is serious.  He needs to get numerous opinions on how to handle it and find out the cause.  Good luck.  Be very proactive about his care.
481082
I have heart disease and my heart used to beat really fast, almost like I had a double heart beat (it's called a "flutter").  I would ask your doctor about it and see if they can send you to a heart specialist.  Heart disease can be a major problem if it's not treated right away.  \n\nYou might have arrythmia or tachycardia or it could be a panic attack, those aren't any fun either.  I would talk to your doctor.  He would know for sure.
713095
The heart IS a muscle, but a heart attack isn't a cramp like you would get in say a leg muscle. \n A heart attack, also called a myocardial infarction, refers to the death of a certain segment of the heart muscle (myocardium).  It's usually the result of a complete blockage in one of the main coronary arteries or a branch which supplies oxygentated blood to the heart muscle.  The main cause of myocardial infarction is atherosclerosis (the build up of plaque on the inside of the blood vessels due to fat substances, cholesterol, calcium and other substances)  in the coronary arteries which results in impaired contractility of the heart muscle within seconds, and is initially restricted to the affected segment.\n\nThe myocardial ischemia or infarction begins in the endocardium (the inner lining of the heart) and spreads to the epicardium (the outer lining of the heart). Irreversible heart damage will occur if the blockage is complete for at least 15-20 minutes. Irreversible damage occurs maximally in the area at risk, and when the occlusion is maintained for 4-6 hours. Most of the damage occurs in the first 2-3 hours.  Restoration of flow within the first 4-5 hours is associated with salvage of the heart muscle, but the salvage is greater if flow is restored in the first 1-2 hours.\n\nA major determinant of death and illness is the size of the infarct. Increasing the oxygen supply to the involved site of blockage by coronary reperfusion can be done through use of certain medications and angioplasty, stents or artherectomy.\n\nAddendum: \n(Impaired contractility means a muscle is unable to contract the way it should or can't contract at all.)\n\nCramps:\nA muscle cramp is an involuntary, painful CONTRACTION of the muscles which produce a hard, bulging muscle. Muscle twitching, also called fasciculation is the result of spontaneous local muscle contractions that are involuntary. The muscle cramp usually only affects the individual muscle groups connected to a particular motor neuron.  Some common causes of cramps are:\n\nMuscle fatigue\nHeavy exercise \nDehydration \nPregnancy \nHeat Cramps\n\nThere may be some temporary loss of circulation to the muscle group that is cramped because of muscle contraction, but there is NO death of tissue due to a clot or embolus.  There are no clots or emboli involved in a cramp therefore they aren't the same as an infarction (as explained in the answer below).     :-)
368620
Angina--which when used alone refers to angina pectoris--is a type of chest pain that is symptomatic of underlying heart problems. It is the direct result of a lack of blood to (or ischemia) an area of the heart muscle (or myocardium), but this ischemia is temporary. A heart attack--technically known as a myocardial infarction--is when a coronary artery in the heart becomes completely occluded and this cuts off the blood supply to a part of the heart muscle permanently. Thus, the essential difference is that angina is temporary and a heart attack is permanent. Both result from ischemia of some area of the myocardium.
745949
CPK levels rise in the blood in response to injury to muscle, any muscle. This could be due to myocardial infarction in the heart, skeletal muscle injury or trauma, or specific disorders of muscle such as autoimmune disorders and inflammatory disorders such as polymyositis. If they are elevated for multiple readings to a significant degree further examination is warranted. In the setting of chest pain or other cardiac associated symptomology, an immediate full work up is required for isozyme specific tests and for the presence of troponin.
664269
The motor neuron diseases (MND) are a group of progressive neurological disorders that destroy motor neurons, the cells that control voluntary muscle activity such as speaking, walking, breathing, and swallowing.  Characteristic symptoms of MND include gradual weakening, wasting away, and uncontrollable twitching of the muscles; spasticity or stiffness in the arms and legs; and overactive tendon reflexes. Sensation, intellect, memory, and personality are not affected in MND.  In some types of MND, such as amyotrophic lateral sclerosis (ALS, commonly called Lou Gehrig’s disease) muscle weakness is progressive and eventually leads to death when the muscles that control breathing no longer work.  Other types of MND progress slowly and last over a lifetime.  MND occur in adults and children, and are more common in men than in women.  In adults, symptoms usually appear after age 40, and may be similar to those of other diseases, making diagnosis difficult.  In children, particularly in inherited forms of the disease, symptoms are present from birth.  Inherited forms of MND are caused by genetic mutations or deletions that cause degeneration of motor neurons.  Hereditary MND include a group of childhood disorders known as the spinal muscular atrophies.  Nonhereditary (also called sporadic) MND are caused by unknown factors, although scientists think environmental toxins or viruses could act as disease triggers.  Nonhereditary MND include ALS, progressive bulbar palsy, pseudobulbar palsy, primary lateral sclerosis, progressive muscular atrophy, and post-polio syndrome. There are no specific tests to diagnose the MND.
32588
myo = major/main\ncardial/cardio = heart\nin = not/bad\nfarction = function\n\nbasically. your heart stopped and now you are struggling to stay alive.
810538
MI means myocardial infarction or damage to the heart muscle due to lack of oxygen.  You apparently had a heart attack at some point.  Women often manifest signs and symptoms of heart attack different from the conventional "left arm/elephant on the chest" type thing.  Some people have no symptoms at all.  Women can manifest angina (chest pain from lack of blood to the heart and ensuing muscle damage) as fatigue, jaw pain, neck pain, pain between the shoulder blades, shortness of breath or indigestion.  Because women often don't notice or pass off these symptoms as something else, heart attacks in women can result in a greater incidence of heart failure (where the heart pump doesn't work as efficiently to move blood).  \n\nBecause women's blood vessels to their hearts tend to be smaller then men's, women can have heart damage from vasospasm (spasmodic contraction of the artery) and doesn't have to be from plaque.  Heart disease in women affects more women than breast cancer by a large margin, yet it is not widely known.\n\nPlease see a good cardiologist right away and get another EKG (12 lead) and an exercise or nuclear stress test and get some clear diagnosis of what happened.  Check out these websites.