how do beta blockers cause hyperglycemia?
156858
They do not cause hyperglycemia -- infact they can facilitate hypoglycemia! Most of the major adverse effects of beta blocking drugs result from beta-adrenoreceptor blockade -- ie: blocking the effects of epinephrine. \n\nEpinephrine, acting via the beta-adrenergic receptors, has important effects on glucose metabolism. It increases glucose production by stimulating both glycogenolysis and gluconeogenesis from amino acids, glycerol, and pyruvate. It also increases the delivery of these gluconeogenic substrates from the periphery, inhibits glucose utilization by several tissues, and, via the alpha-2-receptors, inhibits insulin secretion.
260501
gas, constipation, diarrhea, and bad taste are the most common. Also can cause lactic acidosis if you drink alcohol with it. The best drugs to take are either Actos or Avandia because they do about the same thing but reduce fat in the middle body (vat Fat) which is so bad for type 2 diabetics. These two drugs are much more expensive though.  I'm a pharmacist.
1058108
Some patients have been helped with Beta Blockers\nSee URL\nhttp://www.mayoclinic.com/health/peyronies-disease/DS00427/DSECTION=3\nI hope this is helpful.\nPresident Clinton is apparently affected with this condition.
25152
Quick answer: maybe.  Current evidence suggests that caffine enhances the warning symptoms, rather than causing a drop in blood sugar levels.  \n\nAt this time, the evidence is not quite strong enough to be, _scientifically speaking_, considered causal -- especially in a non-diabetic person.  However, the preliminary evidence is very suggestive of such a link.\n\nEVIDENCE:\nIn a man with diabetes, "excessive coffee ingestion was associated with marked hyperglycaemia." (#1 - case report of only one individual)\n\n"In type 1 diabetes, caffeine has been reported to enhance the symptomatic and hormonal responses to hypoglycaemia."  (#1)\n\n"Ingestion of modest amounts of caffeine enhances the intensity of hypoglycemia warning symptoms in patients with type 1 diabetes without altering the prevailing standard of glycemic control or increasing the incidence of severe hypoglycemic episodes." (#2. Only 34 patients in test: not a large enough population for making a causal link.)\n\n"In healthy volunteers and patients with type 1 diabetes, acute ingestion of modest amounts of caffeine (250-400 mg, which is equivalent to 2-4 cups of drip-brewed coffee) markedly enhances the intensity of warning symptoms and the usual hormonal counterregulatory response to clamped hypoglycemia under laboratory conditions. However, although the perception of hypoglycemia is augmented by prior caffeine ingestion, whether low blood glucose levels will become associated with warning symptoms if caffeine is used on a daily basis is unclear." (#2.  The time frame was only three months, again, not sufficient for a conclusive connection.)
792732
Blod pressure- sodium ( causes fluid retention)\nCreatinine- overly excessive protein consumption and malnourishment\nGlucose- all nutrients that provide calories will ulimately raise blood sugar, some more than others.
928821
You need to see a doctor and get a glucose tolerance test.  That is a high fasting blood sugar and needs investigating...;
169086
In medical school, they never told us that eating too much sugar brings on diabetes, but I do think there is some truth to what you say, that eating alot of sugar contributes to causing Type II diabetes.  (Right, type I diabetes, which is very severe insulin dependent diabetes, is caused by failure of the insulin producing pancreatic beta cells.  it isn't dietary at all)  In type II diabetes, your pancreas is still producing insulin, it just gets "tired" and isn't producing enough insulin, and/or the cell receptors for the insulin, where the insulin has to do its thing, get resistant to the insulin.  In a sense, you wear the system out eating too much sugar.  Also, many type II diabetics are too fat.  Getting too fat does something to make the insulin receptors resistant, and also causes a huge demand for more and more insulin.  Certainly eating alot of "sweet" foods contributes to being fat.  Also, chromium is a known cofactor required for insulin to act correctly.  Refined sugar has no chromium in it, neither does refined flour.  (whole wheat flour does have chromium in it).  So you are eating sugar and refined flour, which requires insulin and chromium to metabolize, but you aren't getting any chromium with it.  \n\nWhat does "junk food" have in it?  It is all high in sugar and/or refined flour.  I think these are the modern foods, which contribute to poor health, including diabetes.  Also these junk foods are high in fat, esp. the evil trans fat.
883918
There are many types of Medications used to treat hyperlipidemia. You would need to be more specific,however OTC Niacin causes flushing of the skin of the head and neck. Statins (Lipitor, Crestor) can cause leg cramping and decreased Liver function. Bile Acid Sequestrant (Cholestyramine) can cause Diarrhea. The list goes on are you looking for one specifically?
1119101
And this is a question about diabetes how?
709322
ask your phsician to check "fasting glusose level"\n\nI'm not an m.d., but used to have "hypoglycemia"
1148393
Yes and increase ur blood pressure...not good...if its happening to u stop taking them and call ur doc. Otherwise ur a tickin time bomb...
361870
ANything over about 15 mg/dL is considered hyperglycemic- Under about 4 or 5 would be considered hypoglycemic, though that would be an exceedingly rare condition in the absence of exogenous insulin.  Our laboratory does work on diabetes and uses mice as models- in general, 15 mg/dl is hyperglycemic, but mildly-quite easily diabetic mice can reach glucose levels of over 30.  Normal levels are 7-12.
924122
provided you are in a research organisation and this is for research then you can obtain propranolol from Sigma aldritch.  if this is for self medication then you should see a doctor since, as with all drugs propranolol is toxic if incorrectly taken
193422
The beta cells in the pancreas produce insulin continuously, and in non-diabetics, there is always some insulin on hand in each cell. When blood glucose increases, non-diabetics (and some diabetics as well) this causes release of the already procuded insulin into the blood. \n\nThe actual amount stored in all the beta cells together is very small. Insulin, in non type 2 diabetics, is effective in very small quantities (micrograms is typical). So there will not be as much as a small drop ever on hand in all the beta cells together even when the most insulin has been stored. This is not very much like the gall bladder which does store gall (from the liver), or any of several other secretory organs.
519786
they risk for alcohol abuse, therefore they are at risk for diabetics... take goood care of your self :)
1160511
What Types of Oral Diabetes Medicine Are Available?\n\nDiabetes medications are grouped in categories based on medication type. There are several categories of oral diabetes medicine -- each works differently.\n\nSulfonylureas. These drugs lower blood glucose by stimulating the pancreas to release more insulin. The first drugs of this type that were developed -- Dymelor, Diabinese, Orinase and Tolinase -- are not as widely used since they tend to be less potent and shorter acting drugs than the newer sulfonylureas. They include Glucotrol, Glucotrol XL, DiaBeta, Micronase, Glynase PresTab and Amaryl. These drugs can cause a decrease in the hemoglobin A1c (/content/article/46/1667_50945?z=1667_50946_6504_00_09HbA1c) [link] of up to 1%-2%. \nBiguanides. These drugs improve insulin's ability to move glucose into cells especially into the muscle cells. They also prevent the liver from releasing stored glucose. Biguanides should not be used in people who have kidney damage or heart failure because of the risk of precipitating a severe build up of acid (called lactic acidosis) in these patients. Biguanides can decrease the HbA1c 1%-2%. Examples include metformin (Glucophage, Glucophage XR, Riomet, Fortamet and Glumetza). \nThiazolidinediones. These drugs improve insulin's effectiveness (improving insulin resistance) in muscle and in fat tissue. They lower the amount of glucose released by the liver and make fat cells more sensitive to the effects of insulin. Actos and Avandia are the two drugs of this class. A decrease in the HbA1c of 1%-2% can be seen with this class of medications. These drugs may take a few weeks before they have an effect in lowering blood glucose. They should be used with caution in people with heart failure. Your doctor will do periodic blood testing of your liver function when using this medication. \nAlpha-glucosidase inhibitors, including Precose and Glyset. These drugs block enzymes that help digest starches, slowing the rise in blood glucose. These drugs may cause diarrhea or gas. These drugs can result in the reduction of the level of HbA1c of 0.5%-1%. \nMeglitinides, including Prandin and Starlix. These medicines lower blood glucose by stimulating the pancreas to release more insulin. The effects of these medications depend on the level of glucose. They are said to be glucose dependant. High sugars make this class of medications release insulin. This is unlike the sulfonylureas that cause an increase in insulin release, regardless of glucose levels, and can lead to hypoglycemia.\nCombination therapy. There are several combination pills that combine two medications into one tablet. One example of this is Glucovance, which combines glyburide (a sulfonylurea) and metformin. Others include Metaglip, which combines glipizide (a sulfonylurea) and metformin, and Avandamet which utilizes both metformin and rosiglitazone (Avandia) in one pill.\n\nMetformin and glipizide are generic products which would be less costly that the brand counterparts.
260233
No.\n\nType 1 diabetes is an auto-immune disease, like some kinds of arthritis or lupus. It requires an inherited susceptibility (traced to certain kinds of HLA antigens), and a trigger. the trigger is usually infection from any of several things, commonly viruses. German measles is one, as are several members of the Coxsackie virus family. During such an infection, the immune system 'learns' to attack cells with certain surface markers, and beta cells have such markers. At least that's the current best idea of what ususally happens. The beta cells get killed (having the wrong markers) and that ends your internal insulin production. Food has nothing to do with it. \n\nType 2 diabetes is more complex. It is strongly inherited, but is also influenced by environment. Those carrying more weight than they should, and the elderly, are more likely to get it. Hence its older names: obesity-related and adult-onset diabetes. It has recently been discovered that fat tissue, especially fat tissue around the abdominal organs (but not on thighs or hips or chins or ...) produce several hormones, just like the endocrine glands. Some of those hormones affect other cells, and one of them makes them  more resistant to insulin. That is, when insulin is present, it's harder for those cells to take up glucose from the blood. This is insulin resistance and it's at the heart of Type 2 diabetes. Some can be treated with diet, exercise, and supervision; some require all that and oral medications as well; some require insulin, perhaps in addition to an oral medication. \n\nObiously, it's not only sugar which can cause fat like this. Overeating anything, perhaps especially fats, can cause weight gain. Ther's nothing special about weight gain from sugar as opposed to weight from the fat in meats. \nAnyone who eats enough to become a little thick here and there has increased their chance of getting Type 2. Lack of exercise seems to be significant as well. So, whether it's sugar or anything else that you eat too much of, you can increase your chance of getting Type 2. \n\nThe only direct effect of sugar is a speculative one. In the extreme pure sugar (or sugar in solution, like a soft drink) will go into the bloodstream very quickly. This will cause a rapid rise in blood glucose level, and perhaps to a high spike in that level. There is some speculation that too many cases of such rapid spikes causes damage to beta cells in the pancreas or to other cells, in such a way as to increase the chance of developing Type 2 diabetes. Thus far, there only suspicions along this line as an experimental study to test this prediction would be difficult to design and carry out. Not to mention expensive. No drug company will finance such a study as there is no prospect of a salable drug resulting. \n\nIt's probably best to avoid 'empty calories' in any case. The classic examples are soft drinks which are basically nothing but flavored sugar water. Check the nutrient label the next time you see a soft drink bottle.