How is acetylcholine released?
146369
If you're talking about the human body, acetylcholine is a neurotransmitter that causes muscle contractions.  Essentially, its released by the axons of a presynaptic neuron.  This release occurs bc of a graded hyperpolarization or depolarization of the plasmalemma causing vesicles containing ACh to be released by means of exocytosis.  Hope that helps.
38377
It comes from the muscle having a spasm.
239811
exergonic releases heat and endergonic take in heat...they both use ATP or some form of energy. They are both chemical reactions dealing with heat
1095780
Contain it in a tube.
647652
Muscles contract when nerves signal them to do so. Nerve transmission is by electicity traveling down them to junctions, where they connect to each other with chemical messengers. So, without the electricity, no nerve signal, no transmission to the muscles, no heartbeat, no you.
373625
stimulation from other nerver endings that are not "feeling pain" cause Ca++ to be released, allowing for analesic neurotransmitters to be released to interneurons, competng with pain causing effects from other places.
693680
mostly with the folloiwing drugs\n- anti cholinergic ,\n- anti parkinsonians,\n- analgesic\n-cimetidine\n-digoxin\n-benzodiaziapines\n-antidepressants\n -illicit substances\n and also...with drawal from alcohol
314995
Within the cells, small-molecule neurotransmitter molecules are usually packaged in vesicles. When an action potential travels to the synapse, the rapid depolarization causes calcium ion channels to open. Calcium then stimulates the transport of vesicles to the synaptic membrane; the vesicle and cell membrane fuse, leading to the release of the packaged neurotransmitter, a mechanism called exocytosis.\n\nThe neurotransmitters then diffuse across the synaptic cleft to bind to receptors. The receptors are broadly classified into ionotropic and metabotropic receptors. Ionotropic receptors are ligand-gated ion channels that open or close through neurotransmitter binding. Metabotropic receptors, which can have a diverse range of effects on a cell, transduct the signal by secondary messenger systems, or G-proteins.
321619
Botulism is caused by a neurotoxin produced by a microbe, Clostridium botulinum. The toxin acts at the neuromuscular junction, i.e. the point at which nerves arrive in the muscles. The nerve endings use a neurotransmitter called acetylcholine to communicate with the muscles and stimulate them to contract. Botulinum toxin blocks the release of acetylcholine.\n\nThe muscle itself is therefore not affected directly, but can't receive any messages from the brain/nervous system. The blocking of acetylcholine also produces other effects, often seen first in botulism poisoning, which include dry mouth, double vision, droopy eyelids, difficulty swallowing or speaking (due to weakness of throat muscles), and a blank facial expression. Muscles of the limbs and trunk, and of respiration, progressively weaken in a descending pattern. However, people usually remain alert throughout and don't have any disturbance of sensation.\n\nThere are actually seven different botulinum neurotoxins, four of which affect humans--type A, B, or E toxin or rarely type F toxin.
375941
Because it's like a release of tension.
183769
Endorphins released in your system is a natural painkiller, released from your brain. Lots of runners have high levels of endorphins which is why they can run with cramps and everything. They don't rid the body of anything, they are a molecular receptor in your brain that releases something like opiates. Hope that helps you
1159021
It's much harder to control your bowel movement than your urinary release because in your rectum you have the external sphincter (which is voluntary) and the internal sphincter (involuntary), the space in between them is not alot of space and therefore the need to empty it is greater due to the involuntary sphincter.  in the urinary tract, the release is almost completely voluntary!
413341
Nicotine works on one of the two types of acetylcholine (ACh) receptors.  The two types of acetylcholine receptors are nicotinic (responds to nicotine as well as ACh) and muscarine (responds to muscarine as well as ACh).  The two different types of receptors have different roles in the body.\n\nNicotinic receptors are found in the all the neuromuscular junctions and the pre-ganglionic Autonomic Nervous System.  So nicotine causes all of these receptors to react.
637350
INFORMATION IS CONDUCTED ELETROCHEMICALLY BY THE NEURONS THROUGH VARYING CONCENTRATIONAS OF Na and K ions across the myelin seath of the neurons.in a neuron at rest there is more conc. of k ions on the inner side(this is the polarised region) and more conc. of Na ions on the outer side(depolarised region) .thus the inner side is at a greater potential than the outer side .when a stimulas is received the conc. in a particular regin of the neuron begins to change. K IONS move to the outer side and Na ions move to the inner side.now in that part of the neuron the outer membrane is at a higher potential than the the inner membrane. in the outer region the Kions are surrounded by large number of Na ions due to this a current sink develops and the K ion move back to the inner side while Na ions move back to the outer region and this region gets repolarised.but the region next to this gets deolarised in the process.thus the nerve impulse travels across the neuron as a wave of polarisation, depolarisation and repolarisation.\n           as the nerve impulse reaches the terminals of the neuron called dendrites the vesicles present there release ascetylcholine and this neurotransmitter transfers messages from one neuron to another and the above process continues till the message reaches the concerned organ.
522406
It is because the urethra (the pee tube) exspans to release the bladder.
670939
No, that's not right.  The movement of sodium and potassium across the membrane carries the action potential along the neurite, but not between neurons.  Neurotransmitters-released by one neurons (usually at an axon terminal) crosses the synapse, and binds with specific receptors (usually on the dendrite or axon hillock). Once it binds, it usually opens ligand-gated sodium channels.  \n\nYou asked the name of the chemical, but it's actually a class of chemicals, not one, called neurotransmitters.  Common ones are glutamate, acetylcholine, seretonin, epinephrine and dopamine, but there are literally dozens of them.
56694
We suspect that there was expression of the 1 subunit in HT 29 cell line after being exposed to nicotine at a concentration of 5 µM, 10 µM and 20 µM