what is Hepatitis Be antibody blood test?
155469
i had the disease too and i had the test. i dont know the medical explanation but the idea is that this test shows if the disease is now chronic or if u r completely recovered.
1038409
There are several antigens besides A, B and O that make up blood types, The E antigen is just one of these.  Having the Anti-E antibody means you are negative for the E antigen and at some time in the past been exposed to that antigen.  This is what caused you to produce the antibody.  Healthwise, all it really means to you is that if you ever need blood, it will be screened to make sure it does not have the E antigen so as not to cause any kind of transfusion reaction.  The blood banker who does your type and cross will have to work a little harder but that's all right, belive me, as a blood banker for nearly 20 years, we don't mind at all, we really do want to do every thing in our power to keep our patients safe.
249456
It is an obsolete term for hepatitis type B virus. It is a virus that causes inflammation of the liver
644538
ANA Anti-nucleus antibodies) is a parameter for a group of Auto -antibodies that go against the cell core (nucleus) what results in the disturbances of the cell-functions. it is usually done when you (the doctor) suspects rheumatoid diseases, but there are usually more antibodies to be determined to tell what you are suffering from. \nA titer of the indirect Immunofluoroscence assay of 1:80 to 1:160 is considered positive. roughly sid the put your blood(serum) in a tube, added some auto-antibody-antibodies and looked for a reaction which shows itself in agglutination of the antibodies ( made visible with the before mentioned Fluorescence). \na positive result COULD mean systematic Lupus erythematodes, Lupus erythematodes of the skin, systematic sclerosis, Poly or Dermatomyositis, Sjoergen Syndrome, Myasthenia gravis, chronic-active Hepatitis, rheumatoid Arthritis and many other rheumatic Diseases. \n\nresults in this Assay vary strongly between laboratories, many healthy people especially over 60 years old show in up to 30% of the cases a titer in their blood without being sick. \ni hope i could help you to understand a little better. But understand that this parameter by itself is not strong enough of an indicator what is wrong and where your symptoms come from.
980950
Hey Hep carrier, let's not get into a tizzy. Hep C is NOT the only strain of viral hep in existence.\n\nAs it so happens, Hepatitis B sometimes does cross react with HIV on the ELISA test. btw, the previous asker gave a FABULOUS explanation of how the tests work. Before we had HIV testing, CDC officials debated using the Hep B test as preliminary test for HIV, despite lower sensitivity to HIV.\n\nThe root of cross-reactivity is in the concepts of sensitivity and specificity. These are measures of test accuracy.\n\nA highly sensitive test picks up a lot of positives, including false positives. A highly specific test eliminates a lot of negatives, including a lot of false negatives.\n\nSo ideally, you want a test that is highly specific and sensitive, but balanced enough so that you pick up all the true positives and rule out all the true negatives.\n\nThe HIV ELISA test is very sensitive, so it picks up a lot of false positives. Because the immunoblot is expensive and time-consuming to run (though highly accurate), it's used as a back up to the quick and easy ELISA.
842991
it depends if its a r n a virus or d n a like hep e  cant do ,it without hcb{hep b}to produce the envolope.
969225
Hepatitis A\n\nFormerly called infectious hepatitis, hepatitis A is most common  in developing countries, but is being seen more frequently in people of all ages and in the developed world. Hepatitis A is thought to be spread by a virus from an infected person's feces directly or indirectly contaminating food, raw shellfish, drinking water, cooking utensils or someone else's fingers. The incubation period is two to six weeks after infection. Hepatitis A is considered an acute condition.\n\nHepatitis B\n\nFormerly called serum hepatitis, hepatitis B is the most serious form of hepatitis.\n\nHepatitis B is caused by the hepatitis B virus and is spread through sexual contact, blood transfusion or exposure to an infected person's blood via cuts, open sores, needle sharing, razor sharing or ear piercing tools. Additionally, hepatitis B can be spread from mother to child at birth. The incubation period is four to 25 weeks.\n\nTherefore in answer to your question, my medical textbook says Hep A can be contracted through contaminated food sources!\n\nHope this helps
452686
Alanine transaminase (ALT), also called Serum Glutamic Pyruvic Transaminase (SGPT) or Alanine aminotransferrase (ALAT) is an enzyme present in hepatocytes (liver cells). When a cell is damaged, it leaks this enzyme into the blood, where it is measured. ALT rises dramatically in acute liver damage, such as viral hepatitis or paracetamol (acetaminophen) overdose. Elevations are often measured in multiples of the upper limit of normal (ULN). The reference range is 15-45 U/L in most laboratories.\n\n\n\nAspartate transaminase (AST) also called Serum Glutamic Oxaloacetic Transaminase (SGOT) or aspartate aminotransferase (ASAT) is similar to ALT in that it is another enzyme associated with liver parenchymal cells. It is raised in acute liver damage. It is also present in red cells, and cardiac and skeletal muscle. The ratio of AST:ALT is useful in differentiating between causes of acute hepatitis.
329131
I'm guessing this is an abbreviation for Hepatitis B surface antigen +. This means that a person has made antibodies against one of the proteins on the surface of Hepatitis B virus. This can result from hepatitis B infection or vaccination with the hepatitis B vaccine. This does not necesarily indicate that a person has an infection it just means that they have made an antibody that recognizes one of the viral proteins. It would be best to talk to a doctor for further clarification about the meaning of any lab test.
681280
I'm not 100% sure, but I think it only checks enzymes...a different blood test is performed to diagnose hepatitis.
570097
Treatment\nAlpha-interferons were the first drugs approved in the United States for the treatment of chronic hepatitis B. Interferon treatment is recommended for individuals who have "replicative disease" (HBeAg positive). About 40% of such individuals will lose serum HBeAg after 16 weeks of treatment with interferon-alpha. Loss of HBeAg is correlated with an improved prognosis. A few treated patients (less than 10%) may even be cured as assessed by the loss of HBsAg.\nPatients treated with interferon-alpha should have evidence of infection with hepatitis B virus, documented by the presence of hepatitis B surface antigen in the blood, for six months. The patients should also have evidence of virus replication, documented by the presence of hepatitis B e antigen in the blood. Ongoing inflammation of the liver should also be present as documented by an elevation in serum aminotransferase activities. A liver biopsy should also be performed prior to treatment. Patients with severe, decompensated liver disease (eg. encephalopathy, ascites, very high serum bilirubin, prolonged prothrombin time, etc.) should not generally be treated with interferon alfa except in the setting of an approved clinical study. \n\nThe recommended dose of interferon alfa-2b for the treatment of chronic hepatitis B is 5,000,000 units daily, administered by subcutaneous or intramuscular injection, for a total of 16 weeks. The patient must be monitored carefully during the treatment period for side effects including flu-like symptoms, depression, rashes, other reactions and abnormal blood counts. \n\nA meta-analysis of several randomized trials of interferon alfa-2b in the treatment of patients with chronic hepatitis B showed such treatment to be cost-effective (Wong et al. Annals Intern. Med. 1995;122:664-675). This analysis showed that treatment with interferon alfa-2b decreased viral replication, documented by loss of serum hepatitis B e antigen, in about 45% of patients compared to less than 10 % of untreated patients. About 8% of patients also lost hepatitis B virus surface antigen (cured) within one year of treatment compared to a rate of about 1% a year for untreated patients. \n\nOther treatment options for chronic hepatitis B include nucleoside analogues. In December, 1998, the United States Food and Drug Administration approved lamivudine , also known as 3TC and is also effective against HIV, for the treatment of chronic hepatitis B (patients who are HBeAg positive). Lamivudine is taken orally at 100 mg/day for chronic hepatitis B. In studies where they were compared, lamivudine was equally effective to interferon-alpha in inducing a loss of serum HBeAg. It also has been shown to improve liver biopsy results in patients treated for one year. In September 2002, the United States Food and Drug Administration approved adevofir dipivoxil, another nucleoside analogue also effective against HIV, for the treatment of hepatitis B. The dose is 10 mg/day for chronic hepatitis B. At the present time, other nucleoside analogues are being studied in clinical trials. The combination of interferon-alpha and a nucleodide analogue, two nucleoside analogues together (such as lamivudine and adefovir) are also under investigation.
770411
They maybe checking for the following: \nSickle cell anemia, Iron dificency, Bata thalassemia trait, aplastic anemia.   Add....'HaemoglobinA1C'  to the list of test and see what you find.\n\nGOOD  LUCK.
443419
Prolonged HBS Ag positives indicate that you have a chronic infection with hepatitis B. There are treatment options available for chronic hep B; many of the same drugs are used as are used for hep C. The treatments tend to last a long time, are expensive, and many have unpleasant side effects. It is best to discuss the treatment options with your doctor.
1068520
Clinical assay is determining the concentration of either a antigen or antibody or microorgannisms or drugs.\n\nIt is very useful in the diagnosis of diseases.
91801
What are they testing for?
675453
There is only A, B and C. No D and E that I'm aware of...\n\nGo to this site for more information. I guess there is Hepatitis D & E: http://digestive.niddk.nih.gov/ddiseases/pubs/viralhepatitis/\n\nThat I decided to research with one of the handy internet search engines everybody has access to by putting "hepatitis d and e" in the search term input box.
279797
Hepatitis B: A Brief History:\n\nIn 1965, Dr. Blumberg who was studying haemophilia, found an antibody in two patients which reacted against an antigen from an Australian Aborigine. Later the antigen was found in patients with serum type hepatitis and was initially designated "Australian Antigen". Subsequent study has shown the Australia Antigen to be the hepatitis B surface antigen. Dr. Blumberg was subsequently awarded the Nobel Prize for his discovery. Initially there appeared to be three particles associated with hepatitis B infection: a large "complete" particle called the "Dane particle", a small circular 20nm particle and an oblong 40nm particle. Further research identified the Dane particle as the hepatitis B viron and the other two particles as excess surface protein. This former terminology is no longer used and the virus is referred to according to its structure.