Bone Paget´s disease!? Child osteoporosis?
115120
What is the question?  My daughter has fibrous dysplasia, similar to Paget's disease, but not at all to osteoporosis. She is treated by a osteoncologist, although she doesn't have cancer.  You can get a lot of information off the 'Net by Google or Yahoo search engines.
179508
Please, don't put your child's health in the hands of an amateur forum. Respect your child's mind and body. Consult your doctor. (I am a physician myself and this is the best advice I can possibly give you.)
542084
Bob wasn't very nice. What a crappy comment.  You should be able to get a referral to an orthodontist for your child through her dental office. Lots of orthodontic offices offer free consultations/estimates which can be beneficial especially if you don't have dental insurance. Good luck and don't pay attn. to Bob. Always consult your dentist for the best advice concerning yoru dental health.
7926
Hi Visu !!!\n\n"Osteoporosis is a disease in which bones become fragile and more likely to break. If not prevented or if left untreated, osteoporosis can progress painlessly until a bone breaks. These broken bones, also known as fractures, occur typically in the hip, spine, and wrist.\n\nAny bone can be affected, but of special concern are fractures of the hip and spine. A hip fracture almost always requires hospitalization and major surgery. It can impair a person's ability to walk unassisted and may cause prolonged or permanent disability or even death. Spinal or vertebral fractures also have serious consequences, including loss of height, severe back pain, and deformity."\n\n----------------------------------------------------\n\n"Reduction in the quantity of bone or atrophy of skeletal tissue; an age-related disorder characterized by decreased bone mass and increased susceptibility to fractures." - From Stedman's Medical Dictionary. \n\n\nPlease go through the below articles for valuable data about osteoporosis.\n\n>>> Naveen Kumar
1094720
There have been some cases of osteonecrosis of the jaw in patients, but the numbers are small.  Everything is a risk, there are no free lunches.  Actonel and Fosamax both increase bone mass, and studies have shown that the bone mass is as good as if it were never not there.  Fractures of the hand, spine etc., (Dowager's Hump) are less in patients taking these medications.  Caucasion women, and Asians are particularly prone to osteoporosis, since they are naturally lighter boned than say black women---i.e. they begin menapause with less bone mass.  You probably now are producing no estrogen, therefore are open to bone resorption at a faster level than if you were on HRT.  Estrogen keeps a woman "younger" than she would otherwise be -- estrogen keeps collegen in place, slows the process of wrinkles, allows vaginal tissues to remain thick and strong, reduced constipation by keeping the lower bowel fit,  keeps mental accuity, and on and on and on.  But estrogen dependent cancers tend to kill, so women with estrogen dependent  cancers generally have a hysterectomy with ovary removal.   The interesting thing about Actonel and Fosamax is that even if you acquire osteonecrosis of the jaw, even if you stop, the drug remains in your system for years.....that's why it is no big deal to miss a week once in a while, or to not take the pill every day of the same week.   In your place, the threat of osteoporosis is a greater one than osteonecrosis of the jaw.  I take Fosamax, and plan to do so for the rest of my life....
413684
This is an excellent site for explaining the results of a DEXA scan.\n\nhttp://courses.washington.edu/bonephys/opbmd.html
467298
mostly  gene can do  long  bone ... \nbut calcium is good for growth ..
760525
Excess protein can causr osteoporosis.\n\nCultural Propaganda on Protein Consumption\nhttp://dgwa1.fortunecity.com/body/protien.html
771634
do you mean Paget's disease \n\nI looked it up via Internet & found this site for you:\n\nwww.healthatoz.com/healthatoz/Atoz/ency/pagets_disease_of_bone.jsp  \n\nclick on the site I typed to get the entire address\n\nhope this helps
253494
Osteogenesis Imperfecta is a congenital disease (blue sclera disease like the person above said). this is a condition resulting from abnormality in the type I collagen, and commonly manifests as brittle easily breakable bones. \n\nLike the people above said osteoporosis is a systemic skeletal disorder people have decreased bone mass and deterioration of bone architecture. Usually people with this have fragile bones and an increased risk for fracture with even minimal trauma. Good luck with your report.
472282
http://www.webmd.com/hw/raising_a_family/tp22217.asp\nhttp://www.orthoseek.com/articles/congenmt.html\ngo to this website
101908
Thank you very much for writing your question in English.  \n\nOne of the rules in Yahoo! Answers is: "USE ENGLISH ONLY: Please do not use other languages or symbols". \n\nThere are many places to learn about Paget's disease. You could begin by reading at these sites:\n\nhttp://www.arthritis.ca/types%20of%20arthritis/pagets/default.asp?s=1\nhttp://www.paget.org/\nhttp://www.nlm.nih.gov/medlineplus/pagetsdiseaseofbone.html\nhttp://www.paget.org.uk/\nhttp://www.mayoclinic.com/health/pagets-disease-of-bone/DS00485\n\nI hope some of these are helpful to you, and I wish you and your daughter the very best.
956538
Talk more with your doctor and ask him to be more specific.  Tell him you are VERY concerned.\n\nMy daughter, when she was around 7 had osteomyelitis in her heel.  She was put in the hospital, given antibiotics through an IV.  She was in the hospital for around a week and every thing worked out fine.  She got over it.   Medicine was near as good as it is today.  Good luck.  Pops
305892
I don't like doing this, but here you go.\n\nWhat is Tay-Sachs Disease?\n\nTay-Sachs disease is a fatal genetic lipid storage disorder in which harmful quantities of a fatty substance called ganglioside GM2 build up in tissues and nerve cells in the brain. The condition is caused by insufficient activity of an enzyme called beta-hexosaminidase A that catalyzes the biodegradation of acidic fatty materials known as gangliosides. Gangliosides are made and biodegraded rapidly in early life as the brain develops.\n\nInfants with Tay-Sachs disease appear to develop normally for the first few months of life. Then, as nerve cells become distended with fatty material, a relentless deterioration of mental and physical abilities occurs. The child becomes blind, deaf, and unable to swallow. Muscles begin to atrophy and paralysis sets in. Other neurological symptoms include dementia, seizures, and an increased startle reflex to noise. A much rarer form of the disorder occurs in patients in their twenties and early thirties and is characterized by an unsteady gait and progressive neurological deterioration. Persons with Tay-Sachs also have "cherry-red" spots in their eyes. The incidence of Tay-Sachs is particularly high among people of Eastern European and Askhenazi Jewish descent. Patients and carriers of Tay-Sachs disease can be identified by a simple blood test that measures beta-hexosaminidase A activity. Both parents must carry the mutated gene in order to have an affected child. In these instances, there is a 25 percent chance with each pregnancy that the child will be affected with Tay-Sachs disease. Prenatal diagnosis is available if desired.\n\nIs there any treatment?\nPresently there is no treatment for Tay-Sachs disease. Anticonvulsant medicine may initially control seizures. Other supportive treatment includes proper nutrition and hydration and techniques to keep the airway open. Children may eventually need a feeding tube.\n\nWhat is the prognosis?\nEven with the best of care, children with Tay-Sachs disease usually die by age 4, from recurring infection.\n\nWhat research is being done?\nThe National Institute of Neurological Disorders and Stroke (NINDS), a part of the National Institutes of Health (NIH), conducts research about Tay-Sachs disease in laboratories at the NIH and also supports additional research through grants to major medical institutions across the country.
357998
calcium intake\nsoda can affect the break down of bones \nwhen your at the age that your bones do not grown anymore osteoporosis can affect it \n-scrolosis\n-genetics\n-Nutrition\n-exercise\n-diease\n-disorders\nmany things can affect bones\n-
692131
PLEASE I URGE YOU TO TAKE HER TO HER PEDO DR, POSSIBLY HAVE AN ULTRSOUND OR CHEST XRAY DONE, DOES SHE HAVE A RATTELY BREATHING SEQUENSE?? ARE THERE ANY ABNORMALITIES IN YOUR FAMILY, THINK HARD....GOOD LUCK... HOPE ALL GOES WELL!
420217
Osteopenia refers to bone mineral density (BMD) that is lower than normal peak BMD but not low enough to be classified as osteoporosis. Bone mineral density is a measurement of the level of minerals in the bones, which indicates how dense and strong they are. If your BMD is low compared to normal peak BMD, you are said to have osteopenia. Having osteopenia means there is a greater risk that, as time passes, you may develop BMD that is very low compared to normal, known as osteoporosis.\n\nSome people who have osteopenia may not have bone loss; they may just naturally have a lower bone density. Osteopenia may also be the result of a wide variety of other conditions, disease processes, or treatments. Women are far more likely to develop osteopenia and osteoporosis than men. This is because women have a lower peak BMD and because the loss of bone mass speeds up as hormonal changes take place at the time of menopause. In both men and women, the following factors can all contribute to osteopenia: \n\nEating disorders or metabolism problems that do not allow the body to take in and use enough vitamins and minerals \nChemotherapy, or medications such as steroids used to treat a number of conditions, including asthma \nExposure to radiation \nHaving a family history of osteoporosis, being thin, being white or Asian, getting limited physical activity, smoking, regularly drinking cola drinks, and drinking excessive amounts of alcohol also increase the risk of osteopenia and, eventually, osteoporosis.\nWhether you will tend to develop osteopenia is, in part, already determined. Things like whether you have any family members who have had osteoporosis or osteopenia, whether you have chronic asthma that requires you to take steroids, and how much calcium and vitamin D you got while you were growing up are beyond your control now. However, if you are a young adult or if you are raising children, there are things you can do to help develop strong bones and help slow down osteopenia and prevent osteoporosis.\n\nYour bones don't reach their greatest density until you are about 30 years old, so for children and people younger than 30, anything that helps increase bone density will have long-term benefits. To maximize bone density, make sure you get plenty of calcium and vitamin D through your diet and by spending a little time in the sun, get weight-bearing exercise on a regular basis, don't smoke, and avoid excessive alcohol or cola drinks. If you have children, teach them to make a nutritious diet and exercise part of their normal routine, make sure they play a little in the sunshine to help their bodies make more vitamin D, and help them avoid smoking and alcohol as they get older.\n\nIf you're older than 30, it's still not too late to make these lifestyle changes. A balanced diet and regular exercise will help slow the loss of bone density, delay osteopenia, and delay or prevent osteoporosis.