what advantage gained by staining cells?
101757
Details are seen easier and with more clearity.
794268
To learn how to make media. To learn to culture and split cells. To know contamination when you see it. Microscopy. Cell counting; by Coulter and hemocytometer. And a whole lot more, but these will do for a start.
928880
More importantly, large multicellular organisms have "specialized" cells. This means that if you took the average cell from the body it would simply die since most cells have outsourced various things to cells that can do it more efficiently. Blood for transporting oxygen, muscle cells for movement, etc.
840819
Gram positive cells stain gram positive because of teichoic acid that binds the peptidoglycan layers of the cell walls together. Gram positive cells have 25 to 30 layers of peptidoglycan in their walls, whereas gram negative cells only have one or two layers. As the cells age  their walls start to break down and cannot retain the stain. They lose layers of their peptioglycan or the teichoic acid isn't as effective. I hope that helps.
529880
Advantages - fast creation, easily modified and saves money\n\nDisadvantages - data can be lost or corrupted, needs backing up all the time.
667592
C\n\nThe ribosomes are far to small to see with a ligh microscope. Under an electron microscope, they look like tiny dots (of whatever color you stained them).\n\nAlso, you won't be able to make out the details of the cell membrane either, only that it exists.
760868
Organelles don't have much of an effect on cell function--mostly it's its shape. More active cells are smaller than less active cells so information can quickly pass through the cytoplasm and into the nucleus. This explains why muscle cells are long, small and slender, and fat cells are large and bulky. \n\nThe membrane of smaller cells has many exterior folds to increase its surface area and ablitily to collect nutrients necessary for cell function.
1144237
The above response addressed positive advantages; as for disadvantages:  When defective apoptotic processes occur, an extensive variety of diseases can result. Too *much* apoptosis causes cell loss disorders, whereas too little results in uncontrolled cell proliferation [read: cancerous tumors].  As outlined on Wikipedia -"Apoptosis can occur, for instance, when a cell is damaged beyond repair, or infected with a virus. The "decision" for apoptosis can come from the cell itself, from its surrounding tissue or from a cell that is part of the immune system.  If a cell's capability of apoptosis is damaged (for example, by mutation), or if the initiation of apoptosis is blocked (by a virus), a damaged cell can continue dividing without restrictions, developing into cancer. For example, as part of the hijacking of the cell's genetic system carried out by human papillomaviruses (HPV), a gene called E6 is expressed in a product that degrades p53 protein, which is a vital piece of the apoptotic pathway. This severe interference in the apoptotic capability of cells plays a critical role in the fact that persistent infection by oncogenic HPVs can result in the development of cervical cancer."  Also, the Hgg protein, which conveys an anti-apoptotic signal, has been found to be overexpressed in pancreatic adenocarcinoma tissues.
310214
That's a rather broad question...  You would probably have to specify it a little more to get the answer you are looking for.  But that said, there are numerous reasons as to why animal cells may develop an "abnormal" morphology.  Basically, there are perhaps two categories that can roughly be differentiated, even though - again - you have to be careful there.  One would be genetic reasons, another would be environmental reasons.  Genetic causes could be numerous as well, but take certain cancers for example, that have a genetic predisposition.  Certain environmental toxins can likewise lead to malformation of cells.  Please note that I was talking of a genetic "predisposition".  Often in case of cancer ebnvironmental and genetic effects are both responsible for the adverse effects observed.  So, the two categories are not ontological absolutes.  \n\nPerhaps what would help you to envision the problem is the basics.  First, cells divide mitotically in unicellular organisms.  consequently, a central question would be the default state of the cell.  There are three possibilities: (a) quiescence (i.e., the cell does not divide and needs an initiation of the mitotic process), (b) proliferation (i.e., cells default state is division and it needs particular factors to asure quiesence as needed in many multicellular organs), and (c) neither, since cell division is contextual.  The most common assumption in biology (and cancer biology, for that matter) is (a).  (b) has been suggested by  Carlos Sonnenschein and Ana Soto ("The Society of Cells. Cancer and Control of Cell Proliferation." New York: Springer, 1999).  Personally, I tend towards (c).  \n\nSecond, cells that proliferate during organogenesis have to further differentiate into the morphology and functionality they are to have in a specific organ.  \n\nThird, certain organs have cells that proliferate, whereas other organs don't.  Consequently, once everything goes well intially in organs of the second type, malformation may not occur as easily.  However, that said there are of course cancers of organs that normally do not have proliferating cells.  \n\nThese three basics may help you to ask your question a little more specifically and then seek out the literature that could give you answers accordingly.\n\nFinally, just remember that what is considered normal in science is the mean of a population.  But what determines this mean is often unclear.  I am thinking here for example of norms of reaction where cells may be shaped quite differently in response to environmental factors, and yet function wonderfully - in fact just exactly as they should given the particular environment.  Now, what is "deformed" would have to be probably understood functionally (i.e., a morphology associated with a functional break down).  And it seems to me that this is what you are looking for anyway.\n\nHope this helps.
439570
Increase Co2 result in the more production of sucrose in the guard cells.That increases its osmotic potential.Water from adjoining cells rush in.Resulting increase in it's opening.
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Cells have different shape to allow them to work better. For example, the alveoli cells are flat and big in surface, these features helps in gas exchange.
1002689
advantage:  money, special privileges\ndisadvantage: no privacy
954310
The main reason is to get better results.
951092
a group of cells form a tissue
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IT absorbes more light,and at the same time magnifies the color of the stained section.
203558
cells that have the potential to differentiate into any kind of cell type
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Advantages:  quickly and clearly demonstrate concepts, tabulate data and compare differences, track trends and predict outcomes with impact. \n\nDisadvantages: If not prepared properly, too complicated or if the wrong type is used, everyone will be confused and the impact will be lost.