Is anyone familiar with Pheochromocytoma,a tumor located on the adrenal gland?
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Pheochromocytomas are tumors of the adrenal gland which produce excess adrenaline. Pheochromocytomas arise from the central portion of the adrenal gland which is called the adrenal medulla. The adrenal medulla is responsible for the normal production of adrenaline which our body requires to help maintain blood pressure and to help cope with stressful situations. A tumor which arises from the adrenal medulla and overproduces adrenaline can be a deadly tumor because of the severe elevation in blood pressure it causes.\n\n\nSymptoms of Pheochromocytomas\n\nThe classical symptoms of pheos are those attributable to excess adrenaline production. Often these patients will have recurring episodes of sweating, headache, and a feeling of high anxiety. The following symptoms are listed from the most common to the least common:\n\nHeadaches (severe) \nExcess sweating (generalized) \nRacing heart (tachycardia and palpitations) \nAnxiety / nervousness (feelings of impending death) \nNervous shaking (tremors) \nPain in the lower chest or upper abdomen \nNausea (with or without nausea) \nWeight loss \nHeat intolerance
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Do you possibly mean a Wilms' Tumor?  If so, this is a pediatric cancer of the kidney.  With proper treatment it has an excellent cure rate, approximately 90%.  I used to work on a pediatric oncology unit, and we took care of many Wilms' tumor patients.  All of our patients are St. Jude kids, but both the site for St. Jude and Memorial Sloan-Kettering have good information for you about Wilms' tumor, both what it is, types, and forms of treatment.  I hope this information is of help to you.
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http://www.ajtmh.org/cgi/content/abstract/72/4/478\n\nthis article may interest you.
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Adenocarcionoma of the cervix is a neoplasia that has not cross the basal membrane (INVASION) and is an early stage) it has good survival percentage if treated right. DO NOT WAIT UNTILL GOES ABOVE THIS STAGE
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Try this link. It appears to be a benign tumor-like growth, and depending on the area is usually a surgical removal. It is actually spelles neurofibroma. Good Luck!!http://www.thedoctorsdoctor.com/diseases/neurofibroma.htm
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I think you're thinking of liposarcoma, which is defined as a malignancy of fat cells. See the link below.
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It is not cancer but something else kindly consult your medical consultant. You may, I repeat may have a problem with the blood vessel in that area or the nerve.\n Get your heart also checked.\n \nPlease do not waste your time seeking advice on the Internet from ignorant people like me.
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The incidence of vonRecklinghausen's disease becoming neurofibrosarcomas is very rare. In reviewing the literature for an article I wrote, I found the possibility of malignant transformation far down on the possible serious side effects. A tumor suddenly growing could be biopsied.
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this is what you want...\nhttp://www.rarediseases.org/nord/search/rdbdetail_abstract.html?disname=Pseudotumor%20Cerebri
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Cavernous angioma, also known as cerebral cavernous malformation (CCM), cavernous haemangioma, and cavernoma, is a vascular disorder of the central nervous system that may appear either sporadically or exhibit autosomal dominant inheritance.
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i am sorry about ur grief. i will try my best to give the best information i can. but u wouldnt mind if its long enough? do u?\nA brain tumor is any intracranial mass created by an abnormal and uncontrolled growth of cells either normally found in the brain itself: neurons, glial cells (astrocytes, oligodendrocytes, ependymal cells), lymphatic tissue, blood vessels), in the cranial nerves (myelin-producing Schwann cells), in the brain envelopes (meninges), skull, pituitary and pineal gland, or spread from cancers primarily located in other organs (metastatic tumors).\n\nPrimary (true) brain tumors are commonly located in the posterior cranial fossa in children and in the anterior two-thirds of the cerebral hemispheres in adults, although they can affect any part of the brain.\n\nIn the United States in the year 2000, it was estimated that there were 16,500 new cases of brain tumors[1], which accounted for 1.4% of all cancers, 2.4% of all cancer deaths[2], and 20%–25% of pediatric cancers[2],[3]. Ultimately, it is estimated that there are 13,000 deaths/year as a result of brain tumors[1].\n\nAside from exposure to vinyl chloride or ionizing radiation, there are no known environmental factors associated with brain tumors. Mutations and deletions of so-called tumor supressor genes are incriminated in some forms of brain tumors. Patients with various inherited diseases, such as Von Hippel-Lindau syndrome, multiple endocrine neoplasia, neurofibromatosis type 2 are at high risk of developing brain tumors.In contrast to tumors originating elsewhere in the body, differentiating primary "brain tumors"—these are the true brain tumors, arising exclusively from cells normally present in the brain itself—into benign and malignant is of relative and limited clinical value, since even histologically-benign tumors grow by infiltration of healthy brain tissue and, in time, tend to transform into malignant forms (anaplastic degeneration). True benign intracranial tumors arise mainly from the meninges (meningiomas; about 95% are benign), pituitary gland (pituitary adenomas) and the myelin sheath of cranial nerves (neuromas or Schwanomas, e.g. acoustic neuroma).\n\nMost primary brain tumors (gliomas) originate from glia: astrocytes (astrocytomas), oligodendrocytes (oligodendrogliomas). There are also mixed forms, with both an astrocytic and an oligodendroglial cell component. These are called mixed gliomas or oligoastrocytomas. Additionally, mixed glio-neuronal tumors (tumors displaying a neuronal, as well as a glial component, e.g. gangliogliomas, disembryoplastic neuroepithelial tumors) and tumors originating from neuronal cells (e.g. gangliocytoma, central gangliocytoma) can also be encountered.\n\nOther varieties of primary brain tumors include: primitive neuroectodermal tumors (PNET, e.g. medulloblastoma, medulloepithelioma, neuroblastoma, retinoblastoma, ependymoblastoma), tumors of the pineal parenchyma (e.g. pineocytoma, pineoblastoma), ependymal cell tumors, choroid plexus tumors, neuroepithelial tumors of uncertain origin (e.g. gliomatosis cerebri, astroblastoma), etc.\n\nFrom a histological perspective, astrocytomas, oligondedrogliomas, and oligoastrocytomas may be benign or malignant. Glioblastoma multiforme represents the most aggressive variety of malignant glioma. At the opposite end of the spectrum, there are so-called pilocytic astrocytomas, a distinct variety of astrocytic tumors. The majority of them are located in the posterior cranial fossa, affect mainly children and young adults, and have a clinically favorable course and prognosis.\n\nIn contrast to other types of cancer, primary brain tumors rarely metastasize, and in this rare event, the tumor cells spread within the skull and spinal canal through the cerebrospinal fluid, rather than via bloodstream to other organs.\n\nThere are various classification systems currently in use for primary brain tumors, the most common being the World Health Organization (W
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The adrenal gland in the endocrine system actually works in co-ordination with the autonomic nervous system of the peripheral nervous system.
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Epinephrine (INN) (IPA: [ˌɛpiˈnɛfrən]) or adrenaline (BAN) (IPA: [əˈdrɛnələn]), sometimes spelled "epinephrin" or "adrenalin" respectively, is a hormone and a neurotransmitter. Epinephrine is a catecholamine, a sympathomimetic monoamine derived from the amino acids phenylalanine and tyrosine. The Latin roots ad-+renes and the Greek roots epi-+nephros both literally mean "on/to the kidney" (referring to the adrenal gland, which secretes epinephrine). Epinephrine is sometimes shortened to epi in medical jargon.\n\nIn May 1886, William Bates reported the discovery of a substance produced by the adrenal gland in the New York Medical Journal. Epinephrine was isolated and identified in 1895 by Napoleon Cybulski, a Polish physiologist. The discovery was repeated in 1897 by John Jacob Abel. Jokichi Takamine discovered the same hormone in 1900, without knowing about the previous discovery; but, in later years, counterevidence is shown from the experiment note that Kaminaka leaves that the Takamine team is the discoverer of first adrenaline. It was first artificially synthesized in 1904 by Friedrich Stolz.\n\nContents [hide]\n1 Actions in the body \n2 Pharmacology \n3 Terminology \n4 See also \n5 References \n \n\n\n[edit]\nActions in the body\nEpinephrine plays a central role in the short-term stress reaction—the physiological response to threatening, exciting or environmental stressor conditions such as high noise levels or bright light (see Fight-or-flight response). It is secreted by the adrenal medulla. When released into the bloodstream, epinephrine binds to multiple receptors and has numerous effects throughout the body. It increases heart rate and stroke volume, dilates the pupils, and constricts arterioles in the skin and gut while dilating arterioles in leg muscles. It elevates the blood sugar level by increasing hydrolysis of glycogen to glucose in the liver, and at the same time begins the breakdown of lipids in adipocytes. Epinephrine has a suppressive effect on the immune system.\n\nEpinephrine is used as a drug to promote peripheral vascular resistance via alpha-stimulated vasoconstriction in cardiac arrest and other cardiac dysrhythmias resulting in diminished or absent cardiac output, such that blood is shunted to the body's core. This beneficial action comes with a significant negative consequence—increased cardiac irritability—which may lead to additional complications immediately following an otherwise successful resuscitation. Alternatives to this treatment include vasopressin, a powerful antidiuretic which also increases peripheral vascular resistance leading to blood shunting via vasoconstriction, but without the attendant increase to myocardial irritability.\n\nBecause of its suppressive effect on the immune system, epinephrine is used to treat anaphylaxis and sepsis. Allergy patients undergoing immunotherapy may receive an epinephrine rinse before the allergen extract is administered, thus reducing the immune response to the administered allergen. It is also used as a bronchodilator for asthma if specific beta-2-adrenergic agonists are unavailable or ineffective. Adverse reactions to epinephrine include palpitations, tachycardia, anxiety, headache, tremor, hypertension, and acute pulmonary edema.\n\nA pheochromocytoma is a tumor of the adrenal gland (or, rarely, the ganglia of the sympathetic nervous system), which secretes excessive amounts of catecholamines, usually epinephrine.\n\n[edit]\nPharmacology\nEpinephrine's actions are mediated through adrenergic receptors (sometimes referred to as adrenoceptors).\n\nIt binds to α1 receptors of liver cells, which activate inositol-phospholipid signaling pathway, signaling the phosphorylation of insulin, leading to reduced ability of insulin to bind to its receptors.\n\nEpinephrine also activates β-adrenergic receptors of the liver and muscle cells, thereby activating the adenylate cyclase signaling pathway, whic
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Maybe lipomas?  A benign tumor composed chiefly of fat cells.
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they are not always affected. Let me explain. Metastasis is the proliferation and spread of cancer cells throughout the body and it just so happens that the adrenal glands were affected by the spreading cancer cells.
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Pheochromocytoma is a possibility, as well as other neoplasias.  You need to see a doctor ASAP.  40 pounds of weight loss in a 16 year old is very, very abnormal.  Best wishes and good luck.
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you may want to get that checked by a doctor, as it could be the sign of an adrenal gland tumor, but it can also be a general overactivity problem (cushings syndrome).  \n\ni believe beta blockers are used for this, but im not sure.