cord blood is an insurance for genetic disorders like thalassemia, cancers, etc. A newly born baby's umblical cord blood is drawn within 10 mins of the baby being born. Thereafter this blood is banked in special blood banks to facilitate the collection of properly matched bone marrow, etc.
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I haven't experienced first hand a case of someone who was able to use the cord blood but I have read several stories of children or families with a sick child that was given the transplant as part of their treatment and were "cured."
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I dont think there's any evidence to claim that they are at this point. Umbilical cord stem cells do NOT have more capability then embryonic stem cells EVER. Embryonic stem cells are totipotent and umbilical cord cells, which are adult stem cells, are only pluripotent. While they cannot become any cell in the human body as ES cells can, they can become several specialized cells of the cell type they are. since the umbilical cord is used for blood and immunological cells and such, its stem cells can be used to produce more of any of those. This is why umbilical stem cells is commongly used in bone marrow transplants (bone marrow is where the cell components of your blood are created).\n\nThey are also of more use now than embryonic stem cells because we have not yet been able to produce a viable embryonic stem cell line from a cloned embryo. We can easily do it with an embryo which has been donated to research, but the problem there is that the genetic make-up of the embryonic stem cells will be foreign and the same fear of rejection and search for a close-enough match will occur. If stem cells are taken from one's own umbilical cord, it will be of their own genetic make up, thus their own cells.\n\n...This was a very long and possibly confusing answer. I hope it helps anyway. If you have any further questions you can contact me.
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Since only one of you carries the Thalassemia gene, your child has a 25% chance of getting thalassemia minor, regardless of its sex. It's usually unnoticed at birth, but becomes apparent in the first year of life (child becomes anemic).\n\nThe hemophilia gene is carried on the X chromosome. For a father who has hemophilia, all sons will be unaffected and all daughters will have the condition (since the daughters will inherit the father's X chromosome for sure).
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no, the only time that blood type will be a concern is when the mother is Rh negative, and the father, Rh positive. The baby will be Rh positive like the father, because positive is dominant.\nThe problem with this is that if the baby's Rh positive blood mixes with the mother's Rh negative blood, the mother's blood will attack the baby's blood.\nThis looks bad, but doctors give Rh negative mothers shots before labor (and in any other situation where the blood might mix) to keep the mother and baby's blood from mixing.\nthat is the only genetic disadvantage that blood type can present.\nare congratulations in order?\nif so, CONGRATULATIONS!!!
489860
I've never heard of a foetus.  What is that exactly?  You're quite the genius.
2936
Most blood transfusions are of packed red cells and not serum/platelets or white cells.  Red cells have no nuclei/genetic material.  Blood type and rH factor are important and mismatch can yield a type of rejection.  The white cells are ultimately "rejected"   In someone with an inactive immune system (chemo patient) the blood can attack the recepient!
568267
Well, gallstones are relatively unusual in children and unborn babies,but have been noted in the following conditions:\n1.neonatal cholestasis- caused by sepsis (infection), hypoxia (usually in mothers who smoke or have lung conditions like asthma),or poor maternal nutrition,or mothers with high cholesterol levels.\n2.hereditary spherocytosis/sickle cell disease\nThese are both inherited blood disorders\n3.Problems with enterohepatic (liver)  circulation\nSometimes,even in the absence of these,some unborn children have gallstones.\nlt is not life-threatening.Elective  C-section at 39 weeks is okay.\nUltrasound can be used to break up the stones without surgrey after birth,thank God!\nWish you a safe delivery!
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Normal red blood cells have a life of about 120 days, and do not have a nucleus, so DNA is extremely minimal. White blood cells, which do have a nucleus, last about 10 days. Transfused blood has had most of the white cells taken out, and does not last as long as your own blood when transfused. So the amount of time it would take for the donor DNA to be cleared from the body would be days, not years. I believe that a swab from the inside of the mouth would circumvent any problems involving blood transfusion and DNA, anyway.
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Sickle cell disease is an inherited disorder of the red blood cells characterized by abnormally shaped red cells. This abnormality can result in painful episodes, serious infections, chronic anemia, and damage to body organs. These effects can, however, vary from person to person depending on the type of sickle cell disease the person has.\n\nSome individuals are relatively healthy and others are hospitalized frequently. But thanks to advancements in early diagnosis and treatment, most children born with this disorder grow up to live relatively healthy and productive lives.\n\nA Closer Look at Sickle Cell Disease\nThe different forms of sickle cell disease are determined by the genes that are inherited from the person's parents.\n\nAn individual has the disease if he or she inherits a sickle cell gene from each parent (hemoglobin SS disease, also called sickle cell anemia).\n\nHe or she can also inherit a sickle cell gene from one parent and a different kind of abnormal gene from the other and end up with a different form of sickle cell disease, such as hemoglobin SC disease and hemoglobin S-thalassemia.\n\nIf an individual inherits only one sickle cell gene and a normal gene from the other parent, then he or she will have the sickle cell trait, but not the disease. A blood test can determine whether you have sickle cell disease or carry the sickle cell trait.\n\nPeople with sickle cell trait don't have sickle cell disease or exhibit any signs of the disorder, but they can pass the gene for the disease to their children. When both parents have the sickle cell trait, there's a 25% chance that a child will have sickle cell disease. But when one parent is carrying the trait and the other actually has the disease, the odds increase to 50% that they will pass the disorder on to their child.\n\nWho Does It Affect?\nIn the United States, hemoglobin SS disease (sickle cell anemia) affects mostly African Americans and some Latino Americans. Some forms of sickle cell disease may occur, although less frequently, in people with different ethnic backgrounds such as those whose ancestors came from Mediterranean countries (including Turkey, Greece, and Italy) or East India.\n\nWhat Causes It?\nA child with sickle cell disease has inherited a defective hemoglobin gene. In a child with sickle cell disease, the hemoglobin can take on an abnormal shape, distorting the shape of the red blood cell. The cells change from a normal round, doughnut shape to the elongated shape of a sickle, or the shape of a "C."\n\nUnlike normal red blood cells, which move easily through small blood vessels, sickle cells are hard and pointed. The sickle shape means that they have a tendency to get stuck in narrow blood vessels and block the flow of blood. This can cause episodes of pain and can also lead to damage of the body organs because they aren't getting enough oxygen.\n\nSickle cells have a shorter-than-normal life span, which leads to anemia (low red blood cell count). A normal red blood cell lives for about 120 days, whereas a sickle cell lives for only 10 to 20 days.\n\nHow Is It Diagnosed?\nUsually, sickle cell disease is diagnosed at birth with a blood test. This can be done in conjunction with other routine newborn screening tests. If your child tests positive on the screening test, a second blood test is usually performed to confirm the diagnosis.\n\nBecause children with sickle cell disease are at an increased risk of infection and other health complications, early diagnosis and treatment is important. Currently, more than 40 states have newborn screening programs for sickle cell disease.\n\nWhat Are the Signs and Symptoms?\nSymptoms of sickle cell disease vary and range from mild to severe, and symptoms may be less severe or different in children who have inherited a sickle cell gene from one parent and a different abnormal hemoglobin gene from the other.\n\nMost children with sickle cell disease have some degree of anemia, an
370075
Right after the donation the blood components are separated into Platelets ( good for 5 days), Plasma ( frozen good for 1year) and red blood cells ( good for 30 days). The testing is done on the extra tubes taking during donation. Testing is done for some chemistry, infectious disease ( HIV, west Nile, Hepatitis B, C, and many more). The blood is typed (A,B,AB,O and RH+/-). It is also tested for poss. bacteria contamination. A complete blood count is performed. After all the test pass acceptability, the components are released to be given. All components are constantly monitored regarding the storage temp.\nBefore blood components are given the recipient is tested to be compatible to the product. ( same blood type and compatible cross match with the donor).\nThe is a lot of checking and double checking involved in blood banking. If you need more or specific info just e-mail me.\nbeen working in blood bank for 20+ years.
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It's a genetic variation like eye colour or skin tone.  \nSome specific diseases do not affect certain blood types and thus it seems like differnt blood types have evolved to combat these diseases.\n\n(I am A positive by the way)
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Rh Blood Types\n\n\nRh click this icon to hear the preceding term pronounced blood types were discovered in 1940 by Karl Landsteiner and Alexander Wiener.  This was 40 years after Landsteiner had discovered the ABO blood groups.  Over the last half century, we have learned far more about the processes responsible for Rh types.  This blood group may be the most complex genetically of all blood type systems since it involves 45 different antigens on the surface of red cells that are controlled by 2 closely linked genes on chromosome 1.\n\nThe Rh system was named after rhesus click this icon to hear the preceding term pronounced  monkeys, since they were initially used in the research to make the antiserum for typing blood samples.  If the antiserum agglutinates your red cells, you are Rh+ click this icon to hear the preceding term pronounced.  If it doesn't, you are Rh- click this icon to hear the preceding term pronounced.  Despite its actual genetic complexity, the inheritance of this trait usually can be predicted by a simple conceptual model in which there are two alleles, D and d.  Individuals who are homozygous dominant (DD) or heterozygous (Dd) are Rh+.  Those who are homozygous recessive (dd) are Rh- (i.e., they do not have the key Rh antigens).\n\nClinically, the Rh factor, like ABO factors, can lead to serious medical complications. The greatest problem with the Rh group is not so much incompatibilities following transfusions (though they can occur) as those between a mother and her developing fetus.  Mother-fetus incompatibility occurs when the mother is Rh- (dd) and the father is Rh+ (DD or Dd).  Maternal antibodies can cross the placenta and destroy fetal red blood cells.  The risk increases with each pregnancy.  Europeans are the most likely to have this problem--13% of their newborn babies are at risk.  Actually only about ½ of these babies (6% of all European births have complications.  With preventive treatment, this number can be cut down even further.  Less than 1% of those treated have trouble.  However, Rh blood type incompatibility is still the leading cause of potentially fatal blood related problems of the newborn.  In the United States, 1 out of 1000 babies are born with this condition.\n\nRh type mother-fetus incompatibility occurs only when an Rh+ man fathers a child with an Rh- mother.  Since an Rh+ father can have either a DD or Dd genotype, there are 2 mating combinations possible:\n\ndrawings of two Punnett squares showing the possible Rh positive mates of an Rh negative woman and the probability of their children being Rh positive--100% if the father is homozygous dominant and 50% if he is heterozygous for this trait\n\nOnly the Rh+ children (Dd) are likely to have medical complications.  When both the mother and her fetus are Rh- (dd), the birth will be normal.\n  \n\ndrawing of a fetus in its mother's uterus; the placenta and the umbilical cord are highlighted\n\nHuman fetus in a mother's uterus\n(the umbilical cord and placenta\nconnect the fetus to its mother)\n \n\nThe first time an Rh- woman becomes pregnant, there usually are not incompatibility difficulties for her Rh+ fetus.  However, the second and subsequent births are likely to have life-threatening problems.  The risk increases with each birth.  In order to understand why first born are normally safe and later children are not, it is necessary to understand some of the placenta's functions.  Nutrients and the mother's antibodies regularly transfer across the placental boundary into the fetus, but her red blood cells usually do not (except in the case of an accidental rupture).  Normally, anti-Rh+ antibodies do not exist in the first-time mother unless she has previously come in contact with Rh+ blood.  Therefore, her antibodies are not likely to agglutinate the red blood cells of her Rh+ fetus.  \n\nPlacental ruptures do occur normally at birth so that some fetal blood gets into the mother's system, stimulatin
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you really need to ask your doc about this one. afp tests are done to check the possibility of neural tube defects in unborn children.A slightly abnormal test does not mean 100% chance the baby will have a birth defect. It is also used to help diagnose problems in people of any age. You need to ask the doc for some solid info on why the test was ordered and what all the possible explanations are for the test results!\n Get a notepad and write down every question you have for the doc and take it with you to the next appointment- pull it out and go over the list with the doc. If doc brushes you off and doesn't take the time- find a doc who will take the time!!
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the baby could become Detached from the umbilical cord
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Blood is everything!! You do not want children with problems, do you?\n\nFor your blood type, your best match is: \n\nType O\n\nA+ and B+ can cause hemogoblinia, which causes small stature in offspring and can cause clawlike hands to form\n\nA+ and A can cause 'Vampirism' which results in offspring that tends to stay out late, wear dark clothes, and engage in orgiastic bloodletting parties.\n\nO is the 'black' of blood. It goes with everything.
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Stem cells are little cells that babies have to form their organs. They can be made into whatever cell you need basically. Stem cell implantation is a handy little thing that transfers embryonic cells (like the kind you had when you were first convieved) into basically whatever kind of adult cell that you need. (It could be a kidney, heart, lung etc..) Personally, I don't think there is anything wrong with that, but there are people why believe that it is inhumane. Stem cells can be taken out of the mothers umbilical cord, and also when women get their tubes tied we want to use the rest of their eggs for stem cell research. Some people say that this is murder in itself and should be banned, but they probably know nothing about it. Stem cells could save lifes of many people. (people that have diseases such as Parkinsons, Alzheimers, Diabetes, etc..)
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store information about the cell. its mostly hereditary.