Death by alcohol induced liver disease - what happens in the final moments?
960670
actually, in most cases end stage liver disease causes the person to enter into a coma like stage. they usually are not aware of what is happening after this stage is present.\n\nas for convulsing....sometimes this happens as the body has already died. it is simply the nerves reacting.\n\n\n\nas far as the disease progression -with liver disease is that the toxins the liver is unable to filter will cause kidney failure and in turn the chemical buildup in the blood will cause severe bloating-so much that the skin seeps fluid. this is when the body enters the coma stage. during the release of fluids, the person is alternating between the coma like state and awareness stages.\n\nyour friend did not feel a thing.\n\ni hope this puts your mind at ease.
167274
The answer to this question is entirely dependant upon the persons metabolism and body weight. The amount of alcohol it takes to lose consciousness won't be the same for a 98lb person as it is for a 250lb person.\n\nAlcohol is metabolized by the liver, where enzymes break down the alcohol. Understanding the rate of metabolism is critical to understanding the effects of alcohol. In general, the liver can process one ounce of liquor (or one standard drink) in one hour. If you consume more than this, your system becomes saturated, and the additional alcohol will accumulate in the blood and body tissues until it can be metabolized. This is why pounding shots or playing drinking games can result in high blood alcohol concentrations that last for several hours.\n\nThe Blood Alcohol Educator is an interactive way to look at BAC levels. Once you input your gender and weight, you can choose the time of drink and how quickly you drink it. You can compare yourself to friends of different weights and gender:\n\nhttp://www.baecdrom.org/\n\nNow that you know how to calculate BAC, see how alcohol affects your body at different levels. The effects of blood alcohol content on thinking, feeling and behavior:\n\n0.02 - 0.03 Legal definition of intoxication in R.I. for people under 21 years of age. Few obvious effects; slight intensification of mood.\n\n0.05 - 0.06 Feeling of warmth, relaxation, mild sedation; exaggeration of emotion and behavior; slight decrease in reaction time and in fine-muscle coordination; impaired judgment about continued drinking.\n\n0.07 - 0.09 More noticeable speech impairment and disturbance of balance; impaired motor coordination, hearing and vision; feeling of elation or depression; increased confidence; may not recognize impairment.\n\n0.08 Legal definition of intoxication in R.I. for people 21 years and older.\n\n0.11 - 0.12 Coordination and balance becoming difficult; distinct impairment of mental faculties and judgment.\n\n0.14 - 0.15 Major impairment of mental and physical control; slurred speech, blurred vision and lack of motor skills; needs medical evaluation.\n\n0.20 Loss of motor control; must have assistance moving about; mental confusion; needs medical assistance.\n\n0.30 Severe intoxication; minimum conscious control of mind and body; needs hospitalization.\n\n0.30 - 0.60 This level of alcohol has been measured in people who have died of alcohol intoxication.\n\n0.40 Unconsciousness; coma; needs hospitalization.
922933
Assuming that the person is otherwise healthy (no medical problems, no kidney problems, no ADH regulation problems, etc):\n\nWell, most people can pee about 15 liters or urine in a day.  This is around 4 gallons!\n\nSo, it would HAVE to be much more than this to cause death by hyponatremia!\n\nIF you drank much more over a VERY short period of time (several gallons in an hour), you would probably vomit some back up, too!\n\nFurthermore, as you started to become hyponatremic, you would become nauseated, feel realy sick, get a bad headache, become confused, and lethargic...\n\nThis, in a sense, would save most people's lives...and prevent death.  In other words, they would get too sick to continue to drink enough to die....even in fraternity hazing.\n\nHowever, if someone was forced to drink enough water over a short enough period of time, they could theoretically drink enough to pass out, have seizures, go into a coma...and yes, even die.\n\nIt is hard (at least for me) to even put a rough guess at this number, as I believe every person would be quite a bit different...and the range of amount to death would likely be VERY variable, depending on weight of the person, etc.  Furthermore, it would depend more on the RATE of water consumption than the actual AMOUNT.\n\nSome would NOT be able to drink enough water to kill themselves.  They would feel so awful, they would stop.\n\nMost people that become hyponatremic from drinking "too much" water either have a physical disease (i.e. impaired ADH regulation, etc) and/or they have a psychiatric disease (i.e. pyshogenic polydypsia).\n\nIn psychiatric disease of drinking too much water, it is usually a chronic thing...not an acute thing (i.e. not a "in one sitting" situation).\n\nHowever, if there were an underlying psychological or physiological problem to begin with, then the hazing thing could kill such a person.\n-\n\nFurthermore, most otherwise healthy people would not die from their "water drinking hazing" hyponatremia...they would pass out, maybe have a seizure, go into a coma.  Then, 911 would be called, they'd be taken to the hospital...given medical care...and gradually come out of it with appropriate treatment.\n\nSo, I know you wanted an exact number...but I don't think this can be answered.\n\nA better fraternity haze is to have people drink a gallon of whole milk in 30 minutes!\n\nNO one can hold down a gallon of milk, if they drink it within 30 minutes....and projectile milk vomiting is quite a sight.
266461
Toxic levels of Tylenol will deplete the liver of an major anti-oxidant, called glutathione.  The accumulation of oxidative radicals will destroy the liver.  The first sign will be jaundice and fatigue-Unfortunately, by this time its too late to save the liver, the damage is already done.  The mortality rate from this level of damage nears 100%.
610341
Alcohol is a central nervous system depressant. It is rapidly absorbed from the stomach and small intestine, passes into the bloodstream, and is then widely distributed throughout the body. The effects of alcohol on the body are directly related to the amount consumed. In small amounts, alcohol can have a relaxing effect. Adverse effects of alcohol, can include impaired judgment, reduced reaction time, slurred speech, and unsteady gait (i.e., difficulty walking); When consumed rapidly and in large amounts, alcohol can also result in coma and death. In addition, alcohol can interact with a number of prescription and non-prescription medications in ways that can intensify the effect of alcohol, of the medications themselves, or both. Furthermore, alcohol use by pregnant women, can cause serious damage to the developing fetus.\nSO YES IT IS A DRUG.
707335
Cirrhosis is a consequence of chronic liver disease characterized by replacement of liver tissue by fibrotic scar tissue as well as regenerative nodules, leading to progressive loss of liver function. Cirrhosis is most commonly caused by alcoholism and hepatitis C, and was the 12th leading cause of death in the United States in 2000. Ascites is the most common complication of cirrhosis and is associated with a poor quality of life, increased risk of infections, and a poor long term outcome. In advanced stages of cirrhosis, the condition is irreversible and the only option would be a liver transplant.\n\nFor any other details you may require please visit website-http://en.wikipedia.org/wiki/Cirhosis-
157522
He should go and see a doctor.......could have an inflammation of the liver or something else. Might be /or not related.\nAlcohol abuse can cause loads of problems.
1118098
Yeah as far as I know, a liver problem. Go to the doc!
805706
If you take a lot of aspirin it just turns to poison inside you and your liver starts frantically trying to get rid of everything before it causes damage to other organs. Unfortunately your liver takes a beating from this and might just decide that it's all too much and give up. If that happens, you will experience acute liver failure and you will very likely die if you do not immediatly receive a liver transplant.\n\nThis is caused by a chemical called acetaminophen which is found in Panadol and Aspirin.\n\nAlso, if you drink alchol or eat foods with a lot of sodium in them (Think salty, savoury foods) then you will likely experience extreme brain damage or death.\n\nI knew somebody who took a lot of painkillers that had  acetaminophen in it and she was moment away from a liver failure. She is now unable to drink alcohol for a year, if not longer.\n\nPS I don't think you will get very sick from just 7 aspirins but you may get a bad headache and feel queasy. If you did take them, don't drink any alcohol because that will make it a lot worse and may cause damage to your body.
1056303
It's so going to be very awful for all of you and very sad. Usually one of the things is boils breaking out all over their body, usually legs. Their liver and kidneys begin to shut down, jaundice develops and they will need dialysis. They will be in alot of pain, may hallucinate and lose weight rapidly.\nI am sorry you are in this position and my thoughts are with you.
1130428
(m)\n\nWhen red blood cells die, the heme in their hemoglobin is converted to bilirubin in the spleen. The bilirubin is processed by the liver, enters bile and is eventually excreted through feces. Once hemoglobin is in the red cells of the blood, it circulates for the life span of those cells. The hemoglobin that is released when the cells die is turned into bilirubin. If for any reason  \nthe RBCs die at a faster rate than usual, bilirubin can accumulate in the blood and cause jaundice. \n\nOnce hemoglobin is in the red cells of the blood, it circulates for the life span of those cells. The hemoglobin that is released when the cells die is turned into bilirubin. If for any reason the RBCs die at a faster rate than usual, bilirubin can accumulate in the blood and cause jaundice.\n\nPre-hepatic (or hemolytic) jaundice is caused by anything which causes an increased rate of haemolysis (breakdown of red blood cells). In tropical countries, malaria can cause jaundice in this manner. Certain genetic diseases, such as glucose 6-phosphate dehydrogenase deficiency can lead to increase red cell lysis and therefore hemolyic jaundice. Defects in bilirubin metabolism also present as jaundice.\n\nLiver diseases of all kinds threaten the organ's ability to keep up with bilirubin processing. Starvation, circulating infections, certain medications, hepatitis, and cirrhosis can all cause hepatic jaundice, as can certain hereditary defects of liver chemistry, including Gilbert's syndrome and Crigler-Najjar syndrome. Hepatic causes include acute hepatitis, hepatotoxicity and alcoholic liver disease. Less common causes include primary biliary cirrhosis, Gilbert's syndrome and metastatic carcinoma. Jaundice commonly seen in the newborn baby is another example of hepatic jaundice.\n\nPost-hepatic forms of jaundice include the jaundices caused by failure of soluble bilirubin to reach the intestines after it has left the liver. These disorders are called obstructive jaundices. The most common cause of obstructive jaundice is the presence of gallstones in the ducts of the biliary system. Other causes have to do with birth defects and infections that damage the bile ducts; drugs; infections; cancers; and physical injury. Some drugs--and pregnancy on rare occasions--simply cause the bile in the ducts to stop flowing. Post-hepatic (or obstructive) jaundice, also called cholestasis, is caused by an interruption to the drainage of bile in the biliary system. The most common causes are gallstones in the common bile duct and pancreatic cancer in the head of the pancreas. Other causes include strictures of the common bile duct, ductal carcinoma, pancreatitis and pancreatic pseudocysts. A rare cause of obstructive jaundice is Mirizzi's syndrome. The presence of pale stools suggests an obstructive or post-hepatic cause as normal feces get their colour from bile pigments.
84610
no, but overdosing in pain relievers will cause liver damage in the future.
388977
yes, its called alcohol poisoning and people die from it all the time.
991052
Yes it's Haram (Or forbidden):\n-No drinking\n-No selling\n-No working in any place where it's sold.\n\nWhy? Alcohol causes temporary insanity (disgrace to a human), and can lead to liver damage.
520851
your body is trying to compensate for the loss of nutrients caused by the alcohol
154446
Symptoms of liver failure are usually vague initially, and can include nausea or fatigue. The progression to jaundice (yellow skin) and confusion or coma can be very rapid. When a patient has the first signs of confusion and disorientation (hepatic encephalopathy), when liver enzymes increase dramatically, or when coagulation factors begin to change - a quick referral to a liver transplant center is highly recommended.\n\nHepatic encephalopathy always accompanies fulminant liver failure. It can manifest as irritability, drowsiness, sluggishness, insomnia, confusion, and coma. With hepatic encephalopathy, the patient is suffering an impairment of proper brain function, which is related to the inability of the liver to metabolize toxic products such ammonia. These toxic products are normally broken down or eliminated by the healthy liver. Treatment is directed at preventing ammonia production in the intestine. This is done with the use of a liquid sugar (lactulose) and neomycin (antibiotic). The neomycin is poorly absorbed and kills the bacteria that promote ammonia production.
790404
Not necessarily. Drinking until you are unconscious can lead to death. When the stomach is pumped, not all the alcohol is removed from the bloodstream, so it can still be in quite lethal levels even if the stomach is emptied.\n\nOther factors such as height, weight, sex, age and many other things also factor in.