Regarding the aging process of your body...?
920673
Take Another Little Piece of My Heart...and my memory, and my joints, and my eyesight.
1045140
Immortality is a tough one.  There is, however, plenty of scientific evidence that aging is in large part genetically programmed. Individuals with certain genetic mutations for example succumb to a premature aging disorder called progeria. Teenagers with this disorder have grey hair, wrinkles, bone degeneration and many other symptoms associated with old age. The fact that these events are genetically controlled, indicates that drugs or genetic therapies can be designed to regulate either slow down or completely inhibit these processes. We would first need to identify the genes that are important.  Studies from a number of groups working in the fruit fly system have used old school breeding strategies to accomplish this.  They raised a colony of fly's separating males and females.  They then allow the last remaining, oldest male and female to breed. Repeat this process 10,000 times over 15 years and what you get is a fly with a life span >3x that of a normal fly (that's 400 years to you and me).  You can then compare the genes that are expressed in your super-fly to that of a normal fruit fly and identify candidate longevity  genes.  This has been done and the human versions of these genes have been identified. The results are fascinating and certainly have the potential to guide therapeutic development.  All of this will not help you if you walk in front of a bus at the spry, young age of 184. Alas immortality is just a dream, rooted in selfishness and a fear of the unknown. Live the life you have to the fullest and try to make a difference in the lives of others.
714196
water. though some foods have enough preserveative to make them last forever or at least until you are old and grey.  j/k
897100
No it is not true. Ageing is a natural process of decay. We live in a decaying world and as with all equipment our bodies decay. If we were to even slow the proces of aging the world would be a mess and over populate at a rate that could not be substrained thus the balance of the natural order would cause even greater poverty and hunger in the world. So why would we want to even try and slow the ageing process.
741661
Yes, like almost every other bodily system, the nervous system 1st gains effectiveness as we go from infantry to childhood, then loses effectiveness as we go from middle-age adulthood to elderly adulthood.
437871
Aging is a broad term that refers to each part of the body getting old. But each part of the body gets old in its own way. Your eye has two main parts that are necessary for vision, the front which is a lens for focusing and adjusting the amount of light and the back which is like the film in a 35mm camera to receive the light and translate it into a message for the brain. When we age, for the most part, the changes to the front of the eye are the most noticeable. The front of the eye has a lens which is attached to little muscles which bend it so you can focus on things far away and things up close like the lens of a camera. This lens is a big piece of transparent protein that is created during development and doesn't really change much for the rest of your life, so as you get older, day to day, you bend the lens to read, and then to see far away and everything in between and it works, but because it's not perfect it gets gradually stiffer as you get older, sometimes it will get cloudy as in cataracts and sometimes the muscles that bend it will get weak, all contributing to the perceived aging of the eye.
457891
I thought it was just that the body slows down the production of all cells around age 30,,  I know you generally reproduce your entire body every 7 years until it slows at  30 ,, an old dude,,  with old brain cells,,,,,,,
115747
Our brain functions tell our heart to pump, it pumps blood throughout your body, when your heart stops pumping, it cuts off circulation to the brain and to the tissues in your body.  Without blood circulation, your cells in your body degenerate.  Your degeneration will start with rigamortis (makes your body turn cold and stiff).  After day 3, your body starts to swell up and deteriorate. The soul is not a physical part of your body but a mental part of your mind.
116499
DNA, tells you're body how you're be when you get older.
875389
When and how will someone learn to cure cancer ?\nWhen and how will someone learn go control human aging ?\nThese are both equally unaswerable questions...\nProbably in the next 25 - 50 yrs ?? Or sooner ?? Or later ??
1163502
There is less hormone prodution in an old body.
342281
Ever wonder what happens to your\nbody the moment you stop smoking? \nWithin 20 minutes of smoking that last cigarette, the body begins a series of changes that continues for years. \n20 MINUTES \nBlood pressure drops to normal. \nPulse rate drops to normal. \nBody temperature of hands and feet increases to normal. \n8 HOURS \nCarbon monoxide level in blood drops to normal. \nOxygen level in blood increases to normal. \n24 HOURS \nChance of heart attack decreases. \n48 HOURS \nNerve endings start regrowing. \nAbility to smell and taste is enhanced. \n2 WEEKS TO 3 MONTHS \nCirculation improves. \nWalking becomes easier. \nLung function increases up to 30%. \n1 TO 9 MONTHS \nCoughing, sinus congestion, fatigue, and shortness of breath decrease. \nCilia regrow in lungs, increasing ability to handle mucus, clean the lungs, and reduce infection. \nBody's overall energy increases. \n1 YEAR \nExcess risk of coronary heart disease is half that of a smoker. \n5 YEARS \nLung cancer death rate for average smoker (one pack a day) decreases by almost half. \nStroke risk is reduced to that of a nonsmoker 5-15 years after quitting. \nRisk of cancer of the mouth, throat and esophagus is half that of a smoker's. \n10 YEARS \nLung cancer death rate similar to that of nonsmokers. \nPrecancerous cells are replaced. \nRisk of cancer of the mouth, throat, esophagus, bladder, kidney and pancreas decreases. \n15 YEARS \nRisk of coronary heart disease is that of a nonsmoker.
705800
Damage-Based Theories of Aging\nOne class of theories of aging is based on the concept that damage, either due to normal toxic by-products of metabolism or inefficient repair/defensive systems, accumulates throughout the entire lifespan and causes aging. In this essay, I present and review the most important of these theories.\n--------------------------------------------------------------------------------\n\nOrgel's Hypothesis\nAging has long been seen as a result of errors of many kinds. An early attempt to build a theory engulfing the genetic and protein machineries was Orgel's hypothesis (Orgel, 1963). His argument was that errors in transcription led to errors in the DNA creating an amplifying loop that eventually kills the cell and leads to aging. Indeed, damaged proteins accumulate with age, and enzymes lose catalytic activity with age (Gershon and Gershon, 1970). This can lead to cellular dysfunctions and accumulation of other forms of damage and senescence. Proteasome is a protein that degrades other proteins; its expression decreases with age (Lee et al., 1999) and this has been implicated as a factor contributing to aging (Friguet et al., 2000). Also, the half-life of proteins is longer in older animals (Friguet et al., 2000). On the other hand, this theory has been regarded as unlikely: feeding abnormal amino acids to animals does not result in a shorter lifespan (Strehler, 1999, p. 293); errors in macromolecular synthesis also does not appear to increase with age (Rabinovitch and Martin, 1982); cultured fibroblasts do not have increased protein errors (Harley et al., 1980); and cellular senescence appears to derive from the telomeres. Presently, Orgel's hypothesis is largely discarded. It is useful to point out, however, that some authors (e.g., Olson, 1987; Holliday, 2004; Kirkwood, 2005) defend that aging is a result of many forms of damage accumulation, and hence that aging is due to an overlap of the mechanistic theories of aging described next.\n\nEnergy Metabolism and Aging\nIn 1908, physiologist Max Rubner discovered a relationship between metabolic rate, body size, and longevity. In brief, long-lived animal species are on average bigger--as detailed before--and spend fewer calories per gram of body mass than smaller, short-lived species. The energy consumption hypothesis states that animals are born with a limited amount of some substance, potential energy, or physiological capacity and the faster they use it, the faster they will die (Hayflick, 1994). Later, this hypothesis evolved into the rate of living theory: the faster the metabolic rate, the faster the biochemical activity, the faster an organism will age. In other words, aging results from the pace at which life is lived (Pearl, 1928). This hypothesis is in accordance with the life history traits of mammals in which a long lifespan is associated with delayed development and slow reproductive rates (reviewed in Austad, 1997a & 1997bb).\n\nAs previously mentioned, caloric restriction (CR) is one of the most important discoveries in aging research. Although the mechanisms behind CR remain a subject of discussion (see below), since it involves a decrease in calories, one obvious hypothesis is that maybe CR works by delaying metabolic rates, in accordance with the energy consumption hypothesis (reviewed in Masoro, 2005). Body temperature is crucial to determine metabolic rate since the rate of chemical reactions rises with temperature. One common feature of animals, such as mice, rats, and monkeys, under CR is a lower body temperature (Weindruch and Walford, 1988; Ramsey et al., 2000), which is consistent with the energy consumption hypothesis. On the other hand, some evidence indicates that mice under CR burn the same amount of energy as controls, suggesting they have similar metabolic rates. These studies, however, remain controversial in the way metabolic rate is normalized to metabolic mass (McCarter and Palmer, 1992). One hypothesis is
301189
Yes.\nBut some - no, make that a lot of - scientists are working on aging, and some believe that if you can eliminate aging, then the natural lifespan of a human will be on the order of 1500-2000 years.\n*Then* we run into some major, new social problems (read John Wyndham's 'Trouble with Lichen' for a slightly dated but still unsurpassed exploration of some of the issues), but I imagine they're not insurmountable.\nNew Scientist ran an article on this in January (link below.) If you want it free you can download it in podcast form. It's pretty neat.
627882
Exercise, eat well, and live a full life! You can't stop aging but you can delay it!
785398
Yours is an intriguing question to put out there to millions of people.  How on Earth, can one intelligently or accurately answer this question even if I knew your life style and I were in the medical profession.\n\nThere are wonderful anti-aging and life extending foods and health practices anyone can utilize to prolong their life.  \n\nObviously proper diet and exercise are key.  Anti-oxidant foods such as blueberries are helpful.  Eating lots of green foods, raw and cooked, is the most advised.\n\nPositive thinking is a major key to health and longevity.  Lots of really good water.  Forgiveness and compassion are the best ways to let go of past negative emotions.\n\nHow long will you live?  You can put yourself into the statistical norm of your mid seventies.  \n\nBut I am not a psychic yet at this moment I get the feeling of living into your eighties.\n\nHave a happy, wonderful rest of your life.  Good Luck\nGod Bless.
86834
Menopause, or premenopause, whichever stage you are in is very unpredictable.  There is a book I love called "What Your Doctor May not Tell You About Premenopause" by dr. John Lee.  That man changed my life and answered questions my regular ob/gyn glared at me for asking.