what is carcinogenic?
915581
A carcinogen is a substance that is capable of causing cancer in humans or animals. If a substance is known to promote or aggravate cancer, but not necessarily cause cancer,may act on deoxyribonucleic acid (DNA), causing dangerous changes, or it may work to increase the rate of cell division. This change in cell division may work to increase the probability of DNA changes. Some carcinogens promote the development of cancer in other ways as well.
168346
Second hand smoke is a known carcinogen.  It is also known to have other effects (increased risk of asthama, reduced immune system, etc.).  And it stinks.\n\nThat said, it is not a higher risk than smoking.  Still, I would try and find a way to deal with it.
764824
It has a stronger potential to cause cancer than cigs. More tar = carcinogens.
918524
These are typically chemicals which do not occur naturally which increase a body's chances of developing cancer. These chemicals in some cases interfere chemically with your body's DNA and allow certain errors in transcription (which cause cancer) to occur.\n\nFiberglass for instance is a man made mineral which is considered to be a carcinogen.
686938
causes cancer\ni guess
88034
The risk of cancer increases if people are exposed to carcinogens (factors causing cancer,ex radiation)
533580
cancer is the most common cause of death of all animals.  cancer has been here since the dinosaurs.  there are cancer tumors in the dinosaur bones.  cancer is more a process than a disease.  After saying all that stomach cancer is a terrible condition and i hope you are just curious.
881950
There are many different forms of cancer and many different causes. Cancer develops from mutations in the genes that regulate cell division. The mutations silence the genes that ordinarily monitor replicating DNA for chemical errors. The affected cells have no built in brakes to halt cell division. As the abnormal mass of clees,called malignant tumor, grows, blood bessel form to supply the tumor with the nutrients it needs to support its growth.Eventually, the tumor invades healthy tissue and may metastasize.\n\nBladder cancer is associated with smoking cigs and alcohol.So is mouth, throat and esophagus cancers. Lung-smoking.Liver-infection with hepatitis virus,alcohol,iron overload,toxicity.Kidney and Pancreatic and lung cancer-high intakes of red meat(pan), cig smoking and air pollution.Prostate-high intakes of fats,esp.saturated fats from red meats and possibly milk products.Ovarian-high lactose intake from milk products and oral contraceptive use. Stomach-high intakes of smoke or salt preserved foods, smoking, infection with ulcer causing bacteria or refined flour or starch.Colorectal-high intakes of fat(saturated), red meat, alcohol, and supplemental iron.Low intakes of fiber, folate,viatmin D and veggies.
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Colorectal cancer (CRC) is a common and lethal disease. The risk of developing CRC is influenced by both environmental and genetic factors.\n\nApproximately 145,290 new cases of large bowel cancer are diagnosed each year in the United States, of which 104,950 are colon and the remainder rectal cancers.\n\nIn 2005, more than 56,000 Americans will die of CRC, accounting for approximately 10% of all cancer deaths. In the United States, CRC ranks second to lung cancer as a cause of cancer death, and it is third both in frequency and cause of cancer death, in men and women separately.\n\nAge is a major risk factor for sporadic CRC. It is a rare diagnosis before the age of 40, the incidence begins to increase significantly between the ages of 40 and 50, and age-specific incidence rates increase in each succeeding decade thereafter.\n\nThe lifetime incidence of CRC in patients at average risk is about 5%, with 90% of cases occurring after age 50. The incidence is higher in patients with specific inherited conditions that predispose them to the development of CRC.\n\nEnvironmental and genetic factors can increase the likelihood of developing CRC. Although inherited susceptibility results in the most striking increases in risk, the majority of CRCs are sporadic rather than familial. At least some data supports the view that family history (as well as physical activity level) are stronger contributors to colon cancer as opposed to rectal cancer risk.\n\nAssociated risk factors:\n1. Familial adenomatous polyposis\n2. Hereditary nonpolyposis colorectal cancer \n3. Personal or family history of sporadic cancers or adenomatous polyps\n4. Inflammatory bowel disease\n5. Diabetes mellitus and insulin resistance\n6. Alcohol \n7. Cigarette smoking\n\nHope this helped ;)\n\n-random physician
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Cancer is not strictly hereditary, but genetics does play a part, and the size of the part it plays varies from one kind of cancer to another.\nCancer is in fact body cells growing out of control, which is triggered when enough mutations of the cells have accumulated. Mutations may be caused by carcinogens, which are various things that can lead to cancer, for example tobacco, radioactive substances, hepatitis B virus. Mutations occur in our bodies every day, and that is why our body cells die every day to lower the possibility that they accumulate enough mutations to become cancerous.\nHowever mutations in germ cells (the reproductive cells like ova and sperms) may be given to offsprings. This means that offsprings now have more mutations to start with, and so easier to accumulate enough mutations to have cancer. Hence, we can see clustering of various types of cancers in families, such as colorectal cancer, cervical cancer, lung cancer. (but lung cancer may also be due to second hand smoke from one family member that smokes)\nEven though cancer is not classified as a hereditary disease (contrary to cystic fibrosis, colour blindness etc), genetics does play a part in the development of cancer.
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Although some carcinogens may be cancer causing, I believe a carcinogen to be any harmful substance that can be introduced into the human body.......
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let's define a few terms first... ;p\n\nan oncogene is a modified gene that increases the malignancy of a tumor cell. some oncogenes usually involved in cancer development increase the chance that a normal cell develops into a tumor cell, possibly resulting in cancer.\n\na proto-oncogene is a normal gene that can become an oncogene, either after mutation or increased expression. proto-oncogenes code for proteins that help to regulate cell growth and differentiation. proto-oncogenes are also often involved in signal transduction and execution of mitogenic signals, usually through its protein product. upon activation, it or its product becomes a tumor inducing agent, an oncogene.\n\nthus... since the activation of the proto-oncogenes either by mutation or increased expression could both occur naturally, then i guess inducing or manipulating these proto-oncogenes to become oncogenes in laboratories is done solely for further studies and research purposes. i think these endeavors more or less aim to equip us with the knowledge and means to better understand how to deal, treat and maybe even prevent it as well as its effects... and definitely not with the intent of killing people...
1158775
you can do something related to human body or effects of environmental degradation on human body such as cancer etc. caused by UV rays.
205680
Chemotherapy is the treatment of cancer using specific chemical agents or drugs that are selectively destructive to malignant cells and tissues. \n\nBasically, it's poison to kill the cancer.  Chemotherapy is usually a combination of drugs to specifically target certain kinds of cancer.  Unfortunately, science doesn't always allow the chemicals to be a specific as we want them to be...so there are some side effects as a result.
589734
A growing field of cancer prevention research is chemoprevention, or the use of natural or synthetic compounds to decrease the number of mutations that may lead to cancer. Chemoprevention research seeks to identify those compounds that reduce risk and use them in pills or food additives as a prevention measure for those who are at high risk for cancer. More than two dozen chemopreventive compounds are currently being studied for how well they work in humans.
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Environment is easily the primary cause of lung cancer.  I'm not even aware of any genetic disorders that are associated with lung cancer.\n\nSmoking is the primary cause of lung cancer.  Before smoking was common, lung cancer was exceedingly rare.  Back in the early 1900's (1904 I believe), a doctor in the US found a man with lung cancer, and he wrote to all of his doctor friends to gather them so that they could have a part in this "once in a lifetime" chance to see a lung cancer patient.\n\nI wish I could photograph some of my textbooks to show you the correlation between smoking and lung cancer.  \n\nThe main causes of lung cancer are smoking, asbestos inhalation, and radon inhalation.  Each of these works via a completely different mechanism.  Smoking works by causing mutations in DNA with chemical carcinogens.  Asbestos works by causing excessive proliferation of certain lung cells, which increases the chance of a transcription error.  Radon works by emmitting alpha radiation, which causes DNA mutations by breaking DNA strands.  Radon then half-lifes into polonium, which adheres to the cells in your lungs and continues to emit radiation, causing more damage.  All three of these mechanisms have only become important within the last century.  Smoking was not popular until the 1940's or so, asbestos was not used prior to the 1950-60's or so, and radon accumulation in homes was not a problem before a similar timeframe.\n\nThere is really no contest between environmental and heredity effects on lung cancer.  Before smoking, asbestos, and radon, there was virtually no lung cancer at all.
161554
the kind that causes cancer