what is morton neuron desies?
919564
I believe you are asking about Motor Neuron Disease.\nMotor Neuron Disease includes many severe disorders of the nervous system characterized by progressive degeneration of motor neurons. \n\nMotor neurons which originate in the spinal cord or brain stem and synapse with muscle fibers are nerves that  carry nerve impulses away from the central nervous system. It facilitates muscle contraction.\n\nSpasms and exaggerated reflexes indicate damage to the upper motor neurons. A progressive wasting (atrophy) and weakness of muscles that have lost their nerve supply indicate damage to the lower motor neurons.♥
928951
There is a relatively new medication called Lyrica that is approved for peripheral neuropathy.\n\nAsk your doctor about it.
215204
yes the brain cells can regenerate.. by process called WALLERIAN DEGENERATION(here degenerate means the dead cll has to gone off first later new one will regenrate... firt of all u have to knw wht r the cells in brain , most of are neurons..and other supporting cells like .neuroglial cells,ependymal cells,schwann cells. satellite cells(ofcourse they r named as they surround peripheral ganglia(ganglia means bunch of neurons )).\nhere wallerian degeration is happend in axon(stem) of neuron.  but it depends of type of injury to axon(whether whole or partial axon is involved).
804499
In its most general terms, neuropathy refers to a disorder of the peripheral nerves (the nerves in our legs, arms, etc.).  Neuropathy can be a chronic, painful, and often debilitating condition where the nerve experiences chronic inflammation and/or dysfunctions.  There are serveral types/causes of neuropathy including:\n1. Diabetes\n2. Alcoholism\n3. Chronic nerve compression in the spine\n4. History of crush injury\n5. ...or even without a known cause, or idiopathic.\n\nStandard treatments usually include management of diabetes (in diabetic neuropathy) and use of medications which depress excitation of the nervous system such as neurontin or Lyrica. Unfortunately, these medications usually come with some hefty side effects.\n\nAnodyne therapy is a realtively new therapy, but there is still much to be learned about this.  It's a treatment usually performed by a physical therapist that uses monochormatic infrared light waves to stimulate blood flow to the nerves.  It is usually used as part of a comprehensive program of Anodyne, gait and balance training in order to reduce pain and increase function/quality of life.  \n\nFirst step is for her to consult with her primary physician or a neurologist.
105755
I suspect it is a biochemical process. I skimmed the attached reference, and here is what I was able to pull out: \n\nThere is a complicated process that takes place in the neurons. Chemical action separates positive sodium and potassium ions from anions across a membrane, creating an electric field. Neurotransmitters open ion gateways in the membrane when the field is built up to a certain level. This allows an electromagnetic wave to travel down the nerve. \n\nIt is MUCH more complicated than that, but that is a simplified cartoon version of the events I think.
460561
The numbness could very well be due to sciatica, but the memory loss and slurred speech intimate a more serious condition.  Forget the neurologist, talk to your family doctor about getting you a referral to a psychiatrist who should order immediate EEG/MRI of the brain.  Or, go directly to emergency.\nI wish you well!!!
576668
The polio virus affects nerve called the sciatic nerve. This nerve innfervates the muscles of the hamstrings, leg and feet. So when this nerve is paralysed, all the muscles it controls atrophy. This is because, for a muscle to not atrophy, it needs to receive tonic impulses from the nerve. If not, it will atrophy.
499693
Peripheral Neuropathy refers to loss of function of the peripheral nervous system (which provides motor and sensory input to the skin and extremities) - as opposed to the central nervous system (brain and spoinal cord).\n\nCauses of peripheral neuropathy can include diabetes, vascular diseases, and toxins among others.  It often begins with the longest nerves (which tend to be the ones that innervate your toes), which is why people with diabetic peripheral neuropathy have so much foot trouble.\n\nYou can learn more here:\nhttp://en.wikipedia.org/wiki/Peripheral_neuropathy
466739
Guillain-Barre Syndrome     proper name and spelling\nWhat is Guillain-Barre Syndrome?\n\nGuillain-Barre Syndrome is a disease of the nervous system due to damage to the myelin sheath around nerves. The myelin sheath acts as an insulator the same as rubber or plastic around electrical wires.\n\nGuillain-Barre Syndrome is the most frequent acquired (as opposed to inherited) nerve disease (neuropathy). In many cases it follows shortly after a virus infection. It is also rarely associated with immunizations, surgery, and childbirth.\n\nWhat causes Guillain-Barre Syndrome?\n\nThe cause of Guillain-Barre Syndrome is unknown, but it appears that there is an autoimmune (one's own immune system attacks the body) reaction.\n\nWhat are symptoms of Guillain-Barre Syndrome?\n\nSymptoms of Guillain-Barre Syndrome include weakness, typically beginning in the legs and progressing upward. The weakness is accompanied by decreased feeling (paresthesia). Reflexes are lost, for example, the hammer to the front of the knee will not induce a kick. In severe cases breathing can be affected enough to require a ventilator and rarely the heart can be affected. The maximal degree of weakness usually occurs within the first 2-3 weeks.\n\nHow is Guillain-Barre Syndrome treated?\n\nThe majority of patients can expect a slow but progressive recovery over several months. In severe cases the primary concern is maintaining vital functions and passively exercising the muscles. This sometimes requires temporary artificial breathing machines for severely affected patients.\n\nPlasmapheresis is a method to remove toxic substances from the blood and has been shown to improve outcome and shorten the disease. Intravenous immunoglobulin is also beneficial
377305
I don't know about nerve damage in the body, but I imagine if nerves recover at all, it is slow. I had a brain injury a few years ago and had serious nerve damage. Your brain cells don't regenerate. The nerves have to find a new pathway. \nTherapy for me, was intense therapies every day. Progress was slow and, eight years later, have nervous reactions at times. Treatment is a good diet, exercise, and avoid harmful things like anything harmful for the body.
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Neurotrophic keratopathy is a degenerative disease characterized by decreased corneal sensitivity and poor corneal healing. This disease leaves the cornea susceptible to injury and decreases reflex tearing. Epithelial breakdown can lead to ulceration, infection, melting, and perforation secondary to poor healing.\n\n\nPathophysiology: The common factor in all cases of neurotrophic keratopathy is corneal hypesthesia. Sensory nerves exert a trophic influence on the corneal epithelium. The sensory neuromediators, acetylcholine, substance P, and calcitonin gene-related peptide, have been shown to increase epithelial cell proliferation in vitro. \n\nDenervation results in decreased cell metabolism, increased permeability, decreased levels of acetylcholine, and decreased cell mitosis. Because a continuous turnover of corneal epithelial cells occurs, this can lead to an epithelial defect even in the absence of injury. Sympathetic neuromediators and prostaglandins decrease epithelial cell mitosis. In fact, ipsilateral sympathetic denervation appears to mitigate the effects of corneal sensory denervation.\n\n\nFrequency: \n\n\nIn the US: Of anesthetic corneas, 15% will develop serious complications. Of the 40,000-60,000 cases of herpes zoster ophthalmicus occurring each year, 50% will have ocular involvement. Of these, 16% will demonstrate some form of neurotrophic keratopathy. \nMortality/Morbidity: \n\nBlurred vision secondary to epithelial irregularity, neovascularization, or corneal scarring \nSecondary infection of nonhealing epithelial defects \nCorneal perforation following stromal melting \nRace: No racial differences exist. \n\nSex: No gender differences exist. \n\nAge: Incidence of neurotrophic keratopathy increases with age.
23899
Polyneuropathy is a neurological disorder that occurs when many peripheral nerves throughout the body malfunction simultaneously. It may be acute and appear without warning, or chronic and develop gradually over a longer period of time. Many polyneuropathies have both motor and sensory involvement and some have autonomic dysfunction. These disorders are often symmetric and frequently involve distal extremities. There is a very large differential for polyneuropathy.
59287
For decades it was thought that there was no regeneration of neurons in the brain or spinal cord.  Relatively recent research has shown that there is some regeneration of neurons. To my knowledge it has only been demonstrated in the hippocampus, a structure primarily responsible for the formation of new memories.
244157
If the foot "beats", an upper motor neuron lesion is suspected.  Usually don't get too worried about 1 or 2 but more is suspicious of increased tone/ UMN lesion
367494
In many cases, prompt diagnosis and treatment of the underlying cause can reduce the risk for permanent nerve damage. For example, controlling diabetes may reduce diabetic neuropathy and renal dialysis often improves neuropathy that develops as a result of chronic renal failure. Treatment options for reducing pain include medication, injection therapy, and physical therapy. Surgery may be needed to treat some causes of neuropathy (e.g., carpal tunnel syndrome, radiculopathy). You can read all about the current treatments here http://www.neurologychannel.com/neuropathy/treatment.shtml
413039
ONJ or “dead jaw” is a rare bone disease in which the jaw bone deteriorates and dies
227285
I've treated dozens of people that have suffered from it. Its not the same in all people. Some get it worse than others. Treatment includes IVIG or plasma pheresis or a combination of both. \n\nSymptoms are progressive weakness that usually starts in the feet and progresses upwards and ends in paralysis that can last from days to weeks.\n\nThe severity of the illness is highly variable.