What is the relationship between aspergillus's and amoxicillin?
913703
They both start with the letter "A" ??
630870
Not likely, but you might be real lucky. Do you feel lucky? Well, do you? \n\nEatwood aside, there are several issues here: \n\nIs the mold one of those which produces penicillin? Can you distinguish among all the various possibilities?\n\nEven if it is, how much penicillin will you be ingesting? Likely not much, and almost certainly not enough. The mold has to protect only a small volume of cells from nasty bacteria, you have to protect a whole lot more. Perhaps 150 pounds of you versus maybe a miligram of mold. \n\nAnd even if there's enough penicillin, it's probably not in the proper chemical form to be ingested and remain effective. One of the things that pharma companies do, aside from producing, purifying, and concentrating the drug, is to find ways to convert it to forms which can survive passage through the stomach (acid and enzymes) and intestines (alkali and enzymes) and still be absorbed into the blood in condition to work. Or to be absorbed in a form which, when processed by the body (usually in the liver), will work. \n\nAnd still further, even if all of that somehow works out, what bacteria do you need killed? Penicillin doesn't work against all bacteria, and qutie a few have learned how to survive in a penicillin rich environment. If the one you need killed is one of those, there won't be much benefit. And anyway, are you sure it isn't a virus? Neither penicillin nor any other bacteriahave any effect on them. The only anti-viral drugs we have work very differently indeed. \n\nAll things considered, I'd suggest you suck it up and give in to artifical corporate culture (pun intended) and do it the pharma way. With all of the unfortunate accompaniements. Odd that a single industry can have earned so much disdain from its customers. Getting all of it right is not something you want to do (unless you are, perhaps, a medicinal chemist?) and certainly not when you've a bacterial infection spreading in your tissues. Not a race you want to lose, surely. Plus there's all that lab culturing business to find out what it is that you need to kill, and whether it's one of the ever so much more common drug resistant strains, ...
667534
I deal a lot with people with gallstones and to be honest with you I've never heard of the connection.  If you are on antibiotics they might be making you itch and whatnot.
985948
There's penicillin, the first antibiotic ever, it was discovered from a fungus that grew in a loaf of bread. I've never heard of any plant or extract that acts as an antibiotic. They may act as antiseptics and kill bacteria, but they aren't antibiotics.
1061288
In case of penicillin based drugs: they inhibit bacterial capacity to build a bacterial wall, so bacteria get killed by lisis. Eukariotic cells don't have a cell wall so they are not affected by penicillin.\n\nSimilarly happens with other kinds of antibiotics
438419
What most physicians prescribe Amoxicillin for most infections - PO (Adults and Children >20 kg): 250–500 mg q 8 hr or 500–875 mg q 12 hr. The drug's usual onset via blood levels is 30 min. Peak lasting for 1–2 hrs and at a duration of 8–12 hr.\n\nThe best way is to follow your physicians prescribed regimen. Follow the directions given, take medication around the clock and to finish the drug completely as directed, even if feeling better.
973241
I believe an antifungal
1103647
Alamethicin is a peptide antibiotic, produced by the fungus Trichoderma viride. It contains the non-proteinogenic amino acid 2-aminoisobutyric acid (Aib), which strongly induces helical peptide structures. The peptide sequence is:\n\nAc-Aib-Pro-Aib-Ala-Aib-Ala-Gln-Aib-Val-Aib-Gly-Leu-Aib-Pro-Val-Aib-Aib-Glu-Gln-Phl\n\n(Ac = acetyl, Phl = phenylalaninol)\n\nCAS number: 27061-78-5\n\nIn cell membranes, it forms voltage-dependent ion channels by aggregation of four to six molecules.
680560
Amoxicillin is an aminopenicillin, has a Betalactam ring and it has not phenylalanin.  It can be used with no trouble.
200382
Penicillin is one of the earliest discovered and widely used antibiotic agents, derived from the Penicillium mold. Antibiotics are natural substances that are released by bacteria and fungi into the their environment, as a means of inhibiting other organisms - it is chemical warfare on a microscopic scale. \n\n\nFollow below link for more info and a picture of the mold!
245029
Penicillium, the genus of the organism which secretes penicillin, is bread mold. It is a type of fungus.\n\nPenicillin was first discovered in Penicillium notatum -- however, that is not the only type of fungus that secretes it. Commercially, the fungus /Penicillium chrysogenum/ is usually used. It isn't as though they grow it on large pieces of bread; instead, the use the process of fermentation to manufacture large quantities.\n\nSee this website:\nhttp://www.chemsoc.org/exemplarchem/entries/2002/stanley/02a_Manufacture/Manufacture.htm
719074
Penicillin refers to a group of β-lactam antibiotics used in the treatment of bacterial infections caused by susceptible, usually Gram-positive, organisms. The name “penicillin” can also be used in reference to a specific member of the penicillin group. Penicillins have the molecular formula R-C9H11N2O4S, where R is a variable side chain.\n\nThe serendipitous discovery of penicillin is usually attributed to Scottish scientist Alexander Fleming in 1928, though others had earlier noted the antibacterial effects of Penicillium. He noticed a halo of inhibition of bacterial growth around a contaminant blue-green mould on a Staphylococcus plate culture. Fleming concluded that the mould was releasing a substance that was inhibiting bacterial growth. He grew a pure culture of the mould and discovered that it was Penicillium notatum (now known as Penicillium chrysogenum) – he later named the bacterial inhibiting substance “penicillin” after the Penicillium notatum that released it. Fleming was convinced after conducting some more experiments that penicillin could not last long enough in the human body to kill pathogenic bacteria and stopped studying penicillin after 1931, but restarted some clinical trials in 1934 and continued to try to get someone to purify it until 1940.\n\nIn 1939, Australian scientist Howard Walter Florey and a team of researchers (Ernst Boris Chain, A. D. Gardner, Norman Heatley, M. Jennings, J.Orr-Ewing and G.Sanders) at the Sir William Dunn School of Pathology, University of Oxford made significant progress in showing the in vivo bactericidal action of penicillin. Their attempts to treat humans failed due to insufficient volumes of penicillin, but they proved its harmlessness and effect in mice. On 1942-03-14 John Bumstead and Orvan Hess became the first in the world to successfully treat a patient using penicillin\n\nDuring World War II, penicillin made a major difference in the number of deaths and amputations caused by infected wounds amongst Allied forces. Availability was severely limited, however, by the difficulty of manufacturing large quantities of penicillin and by the rapid renal clearance of the drug necessitating frequent dosing. Penicillins are actively secreted and about 80% of a penicillin dose is cleared within three to four hours of administration. During those times it became common procedure to collect the urine from patients being treated so that the penicillin could be isolated and reused. (Silverthorn, 2004)\n\nThis was not a satisfactory solution, however, so researchers looked for a way to slow penicillin secretion. They hoped to find a molecule that could compete with penicillin for the organic acid transporter responsible for secretion such that the transporter would preferentially secrete the competitive inhibitor. The uricosuric agent probenecid proved to be suitable. When probenecid and penicillin are concomitantly administered, probenecid competitively inhibits the secretion of penicillin, increasing its concentration and prolonging its activity. The advent of mass-production techniques and semi-synthetic penicillins solved supply issues, and this use of probenecid declined. (Silverthorn, 2004) Probenecid is still clinically useful, however, for certain infections requiring particularly high concentrations of penicillins. (Rossi, 2004)\n\nThe chemical structure of penicillin was determined by Dorothy Crowfoot Hodgkin in the early 1940s, enabling synthetic production. A team of Oxford research scientists led by Australian Howard Walter Florey and including Ernst Boris Chain and Norman Heatley discovered a method of mass producing the drug. Florey and Chain shared the 1945 Nobel prize in medicine with Fleming for this work. Penicillin has since become the most widely used antibiotic to date and is still used for many Gram-positive bacterial infections.\n\nAntibiotic resistance to penicillin was first observed in 1947, shortly after its introduction. Resistance to penicill
605165
Simple...ALL antibiotics that we use to treat bacterial infections and ifestations come from fungi themselves. It's just natural that fungi will have a resistance to these antibiotics that they release.\n\nA rather different approach must be used to stop fungal infection and proliferation not because they have a "tougher" cell wall or because they are eukaryotes--these things are more trivial than what people think. The difficulty in treating them is how to disintegrate their nucleic acid property (DNA) using a particular chemical without compromising the infected individual (i.e. you cannot just use acid to treat fungus in your skin or else you'll damage your skin also).
766505
coccidiomycocis, aspergillosis, and blastomycosis
207586
There are plenty of reasons for being cautious about alcohol, but taking penicillin isn't one of them. In fact, it's OK to drink alcohol with nearly all antibiotics, though just a few might make you nauseous.\n\nAlcohol won't interact with penicillin, but it will decrease the effectiveness of the antibiotic, and make healing time longer and the chance to spread whatever you have may increase.
1016584
Antibiotics that have been shown to interact with birth control pills include rifampin (brand name Rifadin), penicillin (Veetids), amoxicillin (Amoxil), ampicillin (Omnipen), cotrimoxazole (Septra or Bactrim), tetracycline (Sumycin), minocycline (Minocin), metronidazole (Flagyl), and nitrofurantoin (Macrobid or Macrodantin). For at least one week afterward doctors generally recommend that you use a condom or spermicide as a back-up method of birth control. If you are taking both antibiotics and birth control pills, be sure to check with your doctor about how long you should continue to use a back-up method.
269157
Fusarium comes from the latin for spindle, referring to the general shape of the spores of this fungus.  oxy means sharp, or saw-toothed, and sporum refers to the spore itself.  So I would assume that the spores of this fungus are spindle shaped, with sharp or pointy bits sticking out from it.\n\nFusarium oxysporum is a common soil fungus, and can become a pathogen causing a wide variety of wilt diseases in plants (usually called Fusarium wilts).  It is also sometimes known as Agent Green.  no idea why.  Fusarium oxysporum can also sometimes infect people (and I presume other animals too).  usually it manifests as toenail or fingernail fungus, making them deformed and icky looking, but can also become a more generalized infection, especially in people with weakened or compromised immune systems.  some of these strains have become resistant to conventional antibiotics.