is it true that eating lots of cheese can develop alzheimers disease?
343015
Very little is known at this point as to what causes Alzheimers.  Family history is a known risk factor.  Cheese consumption is not.
1037007
You'd have to add up all the muscle (red meat) and the organs. Then did you want to factor in the bones & skin? The number depends on the make up of the tissue, there is no set number/pound. Sorry See_n_stars 03 but that is not true.\n\nI have no idea. I would advise against eating the organs - high cholesteral - and nervous tissue has different types of diseases on top of that (Mad COw disease is spread by eating brain and spinal column tissue). That means no more head cheese (thank God!!!!!!).\n\nThe Calorie (not calorie) count is determined by the amount of energy released when an object is reduced to ash.
1075114
If you eat too much of these, yes, to be sure.\n\nHowever, if you eat them in moderation, and mix in vegetables, and some exercise, you should be just fine.
21798
cows and goats can carry some diseases and it can be spread through the milk. T.B. can be spread that way and dairy animals are regularly tested for T.B. If an animal gets mastitis the milk can become harmful. In the late 50's a young girl who lived next to my grandmother and drank fresh cow milk got milk sickness and nearly died. It left her with some brain damage and shaky hands. I don't know if it was caused by something the cow ate or an actual disease the cow had. I think the milk would need to be tested to find out for sure. Also her 4 brothers and her parents drank the milk, as did several other people in the neighberhood and as far as I know she is the only one who got sick.
86911
Alzheimer's disease (AD), a neurodegenerative disease, is the most common cause of dementia and characterised clinically by progressive intellectual deterioration together with declining activities of daily living and neuropsychiatric symptoms or behavioral changes. The most striking early symptom is memory loss (amnesia), usually manifest as minor forgetfulness that becomes steadily denser with illness progression, with relative preservation of older memories. As the disorder progresses, cognitive (intellectual) impairment extends to the domains of language (aphasia), coordinated movement (apraxia), recognition (agnosia) and those functions (such as decision-making and planning) closely related to the frontal lobe of the brain, reflecting extension of the underlying pathological process. This consists principally of neuronal (cell) loss (or atrophy), together with deposition of amyloid plaques and neurofibrillary tangles. Genetic factors are known to be important, and polymorphisms (variations) in three different autosomal dominant genes - Presenilin 1, Presenilin 2, and Amyloid Precursor Protein - \n\nDiagnosis\n\nThe diagnosis is made primarily by clinical observation and tests of memory and intellectual functioning over a series of weeks or months, with various physical tests (blood tests and neuroimaging) being performed to rule out alternative diagnoses. No medical tests are available to conclusively diagnose Alzheimer's disease pre-mortem, however.\n\nInterviews with family members and/or caregivers can be extremely important in the early phases as well, as the sufferer him/herself may tend to minimize his symptomatology or may be being observed on a day when his/her symptoms are in temporary dormancy.\n\nInitial suspicion of dementia may be strengthened by performing the mini mental state examination, after excluding clinical depression. Psychological testing generally focuses on memory, attention, abstract thinking, the ability to name objects, and other cognitive functions. Results of psychological tests do not easily distinguish between Alzheimer's disease and other types of dementia but can be helpful in establishing the presence of and severity of dementia. They can also be useful in distinguishing true dementia from temporary (and more treatable) cognitive impairment due to depression or psychosis, which has sometimes been termed "pseudodementia".\n\nWhile expert clinicians who specialize in memory disorders can now diagnose AD with an accuracy of 85-90%, a definitive diagnosis of Alzheimer's disease must await the autopsy.
22511
I have heard that using your brain more can help delay or prevent Alzheimers, though not scientifically proven. Puzzles like Mensa, Soduku, brain teasers may help.\n\nEfforts to find effective treatments for Alzheimer's after-the-fact have so far been disappointing. Age is the primary risk factor for Alzheimer's. The baby boom is approaching its golden years. Indeed, much of the concern about the solvency of governmental social safety nets is founded on estimates of the costs of caring for baby boomers, assuming that they develop Alzheimer's in the same proportions as earlier generations.\n\nOne study ("Leisure Activities and the Risk of Dementia in the Elderly," New England Journal of Medicine [8]) found that people who played chess on a regular basis went on to get Alzheimer's at a substantially lower rate than the general population. The chess relationship was stronger than any other factor, including dancing and solving crossword puzzles, both of which were also shown to be inversely proportional to getting Alzheimer's disease.\n\nIn a number of retrospective studies, regular physical exercise has appeared to be inversely related to the development of Alzheimer’s. The Alzheimer's risk of those exercising regularly was half that of the least active. This research is consistent with the observation that virtually all measures designed to promote cardiac fitness and reduce stroke risk also seem to reduce Alzheimer's risk. However in one study, dance appeared to be the only exercise effective in reducing risk. One explanation is that dancing requires the use of complex mental skills such as performing correct steps while at the same time keeping track of the music. The presence of cardiovascular risk factors -- diabetes, hypertension, high cholesterol and smoking -- in middle age (ages 40 to 44) was found very strongly associated with late-life dementia (Neurology 2005;64:277-281. PMID 15668425).
458663
Yes , eating mutton or lamb will cause heart diseases. it is true
77631
Although it says 'low-fat' I believe the fat content of cheese is higher than the yogurt.  Cottage cheese also has more protein in it (on average) and may actually be more filling.  I personally don't see a problem with substituting those 2 items althought I am no dietition.
550467
yogurts and dairy foods definitely do not.  They are healthy foods in moderation, but they actually make people gain more weight.  They are good sources of calcium however.\n\n\nFoods with antioxidants are great, though, as well as certain citrus fruits.\n\nPommegranites, cranberries, and blueberries are the best.  These foods are also great for giving energy, loosing weight, and they help with slowing down the aging process.
35151
not all the time, it could be bad cheese. but you can go to an allergy doctor (i'm not sure what they're called) and get tested. all they do ispoke you witha piece of plastic and see if your allargic and if ur skin swells up.
532852
At this point no- I have spent about 10 years researching the causes and potential treatments of AD.  We know a fair amount about risk factors- about 10% of AD is caused by known mutations in specific genes, the majority to the gene that is the precursor to amyloid beta (APP), or a gene that is involved in producting amyloid beta from APP, called presenilin.  These people get AD very young.  However, even in these cases, where we know the genetic cause, we still have no cure or prevention.  In the majority of cases, we don't even have any clear genetic cause-But, we do have a lot of risk factors, and suppose that if a person avoided them it would delay or in some cases possibly prevent the disease.  It is an extraordinarily complex disease, in the most complex organ of the body, and no, we can't cure or prevent it with drugs, but possibly delay it with changes in lifestyle.
794579
In addition to throwing off the body's homeostasis, excess sugar may result in a number of other significant consequences. The following is a listing of some of sugar's metabolic consequences from a variety of medical journals and other scientific publications.\n\nSugar can suppress your immune system and impair your defenses against infectious disease.1,2\n\nSugar upsets the mineral relationships in your body: causes chromium and copper deficiencies and interferes with absorption of calcium and magnesium. 3,4,5,6\n\nSugar can cause can cause a rapid rise of adrenaline, hyperactivity, anxiety, difficulty concentrating, and crankiness in children.7,8\n\nSugar can produce a significant rise in total cholesterol, triglycerides and bad cholesterol and a decrease in good cholesterol.9,10,11,12\n\nSugar causes a loss of tissue elasticity and function.13\n\nSugar feeds cancer cells and has been connected with the development of cancer of the breast, ovaries, prostate, rectum, pancreas, biliary tract, lung, gallbladder and stomach.14,15,16,17,18,19,20\n\nSugar can increase fasting levels of glucose and can cause reactive hypoglycemia.21,22\n\nSugar can weaken eyesight.23\n\nSugar can cause many problems with the gastrointestinal tract including: an acidic digestive tract, indigestion, malabsorption in patients with functional bowel disease, increased risk of Crohn's disease, and ulcerative colitis.24,25,26,27,28\n\nSugar can cause premature aging.29
97221
I participated in a health research study a few years back. At the end of the study, they did a MRI scan on my brain.  They found some white spots all over the brain.  \n\nIt was determined that the sheaf, (Myelin) which is the outer coating of the nerves in the brain, had been apparently eaten away for some unknown reason. That was the white spots.  I was told it could have been some kind of virus.  Still don't know. I was also told that I might get Dementia in my old age.  So far, I'm doing good.  \n\nThere is a whole lot of information on the Internet.  But by all means, keep in contact with your husband's doctor so he/she can run additional tests from time to time TO SEE IF THERE IS ANY CHANGE.  Its better knowing I think.   Good luck.  (sorry for yelling)
194063
Has it been this way all your life, or did it seem to just come about?  How old are you?  Cognitive decline in the later years, combined with memory problems, might warrant a visit to the doctor to rule out Alzheimer's.  The headaches are not likely related, but it wouldn't be smart to rule it out.  Meningitis can cause confusion and headaches, but it's the type of thing that doesn't run a very long course.  \n\nIn the meantime, it couldn't hurt to make sure your nutrition is in line.  Take a vitamin, and make sure you're not starving yourself.  Caffeine has been shown to temporarily improve mental acuity, so you might consider this for a critical activity.  Then again, too much coffee will probably harm you physically more than it will help you mentally.\n\nGood luck, and try not to worry.  If you're really concerned about it, just have a visit to your primary care doc.
44062
Lack of Gluclose has not been shown to cause dementia although\nDeficiencies of certain nutrients, especially B vitamins, can cause dementia if not corrected.
173948
Alois Alzheimer's, a doctor and academic, presented a key paper to the meeting of the South West German Society of Alienists on the 3rd November 1906. It was the first paper describing a clinical case of dementia and was an examination of the behavior of Frau Auguste D.\n\nFrau Auguste had been admitted to the Municipal Mental Asylum in Frankfurt-am-main, Germany on 25th November 1901 at the age of 51 years. The senior physician at the hospital was Dr Alois Alzheimer.\n\nBy the time she died in April 1906, only 5 years later, Dr Alzheimer had moved to the Anatomical Laboratory of the Royal Psychiatric Clinic, Munich University as a co-worker to Dr Kraepelin. Frau Auguste D's brain was sent to Alzheimer and his findings of plaques and fibrules formed his hypothesis that there was an organic cause for her condition. The translated paper, only three pages long, describes symptoms that included, disorientation, aphasia, auditory hallucinations, paranoia, unpredicable behavior and pronounced psycho-social impairment. It is interesting to note that Alzheimer made no attempt to place the case in any sort of social or environmental context. Frau Auguste D's past life and institutional incarceration seem to have been ignored completely as a possible explanation of Frau D's mental state.\n\nDr Alzheimer's description of organic changes, atrophy, neurofibrils, and plaques linked to dementia was not a new discovery, yet it is his name that we link to dementia in most cases. Why should this be?\n\nAt the time, the first attempts were being made to classify illness and disease both organic and psychiatric. Kraepelin, probably better known for his classification of schizophrenia, is the pivotal figure. It was he that incorporated the study by Alzheimer on Frau Auguste D in his 1910 eighth edition textbook 'Psychiatrie'. Kraepelin stressed the role of organic changes occuring in the brain in his theories of mental illness. He described 'Alzheimer's disease' both as a distinctive disease that resulted from the action of a single cause and a distinct subcategory of senile dementia. In so doing Kraeplin describes the clinical behaviors that formed a set of distinct symptoms resulting from a specific organic cause.\n\nOther exponents in the field could just as easily have been the figure head for a disease category, and instead of Alzheimer's disease we could easily be talking of Bonfiglio's disease, Fischer's disease, Fuller's or Perusini's dementia. Alzheimer just seems to have been in the right place at the right time. Kraepelin was making a name for himself as the head of the large Munich academic institution, he needed to increase its prestige and influence, what better way to do it than by using someone from the same department.\n\nAlois Alzheimer died aged 51 of tonsillitis which had resulted in nephritis. In his life time he published 19 papers on subjects as diverse as eplilepsy, the classification of psychiatry, general paresis as well as on dementia. He could have little idea that his name would become an internationally understood description of a disease suffered by millions of people.\n\nAlois Alzheimer born June 14 1864 in Marktbreit, Bavaria. Died December 19 1915 in Breslau.
1160031
The evidence is not clear. Cases where several members of a single family have had autopsy-confirmed diagnoses of Alzheimer's disease are rare. Much more common is the situation where a single family member is diagnosed as having probably Alzheimer's (meaning that physicians are 80 to 90 percent certain that it is Alzheimer's).\n\nA person's risk of developing the disease seems to be slightly higher if a first-degree relative (brother, sister, parent) has the disease. This situation is called "familial," which means there could be a genetic factor involved, or perhaps family members were exposed to something in the environment that caused the disease.\n\nAnybody can develop Alzheimer's disease. However, a person who has a parent or sibling with the disease carries a greater risk of developing Alzheimer's disease than people who have no family history of the disease. Yet, it can also strike only one member of a family. Since many factors affect the development of the disease, having Alzheimer's disease in the family is a risk factor, but it doesn't mean you will develop the disease.