If the transverse tubules were blocked.what would happen to muscle contraction and why?
353806
T-tubules transmit nerve impulses throughout the muscle.  Think of it this way.  Your biceps muscle is composed of many different muscle cells working together.  When your brain tells your biceps to contract, does it send individual nerves to every single muscle cell?  No it doesn't (I guess that really isn't an obvious answer).  This is where the T-tubules come in.  They transmit electrical impulses from one muscle cell to another.  This way, only some muscle cells need to be activated by a nerve--the rest will be signaled through the T-tubule network, so that all of the muscle cells will contract together.\n\nSo what happens when you block the T-tubules?  Your muscle doesn't work so well anymore.  You might still get cell-to-cell communication (I'm not sure), but even if you did, it would be slow so that the muscle fibers would not contract synchronously.\n\nAddendum: It's been 6 or 7 years since I last studied physiology, so I think you should listen to the professor, not me!
626833
No matter how far you go or you fancy to go, there is no way a male organ in intercourse can get stuck. The forced sex can result into a spasmodic contraction of vaginal muscles and extreme dryness on the part of an unwilling female but even then you don't get stuck. Nature has provided its own technological miracles to avoid situations like this.
932403
Animal muscle tissue is fascinating, when you learn how it works or "clenches". The process by which muscle clenche and release seems rather complicated to learn about.  But, it's worth it.  I believe there's a possibility for alternative energy stemming from the principles for how muscles work.  I wish all engineers had to learn about anatomy and physiology.  Here are the three easiest sites I could find. You might buy a used entry level human anatomy and physiology textbook and just start at the beginning, don't try to remember all the names of everything.  But start with cells and move up to muscle tissue early in the text. It's amazing stuff. Excellent question and sense of curiosity:\n\n\nhttp://en.wikipedia.org/wiki/Muscle_contraction\nhttp://users.rcn.com/jkimball.ma.ultranet/BiologyPages/M/Muscles.html
416619
C) By interfering with the electron transport chain, energy production (in the form of ATP) would be significantly reduced.  Neurons use a great amount of energy to maintain the ionic gradient that allows synaptic transmission.  In he absence of ATP, the ionic pumps, primarily sodium/potassium, would not function, the ionic gradient would break down, and it would halt neuronal transmission.
898301
According to my anatomy textbook, it's so that once urine comes out of the ureter into the bladder, the ureter is automatically pinched shut so that the urine doesn't flow back up the ureter toward the kidneys.  Having problems with this mechanism can cause one to have to urinate a lot more often (because when the bladder squeezes to empty itself, some of the urine goes back the wrong way), and if serious or untreated for a long time, can lead to kidney problems.\n\nOut of curiosity, why did you want to know?
688875
Ignore the first comment; the person didn't realize that's a nerve bundle block and not an artery blockage.\n\nBasically the depolarization/repolarization of the left ventrical will be slightly slower because the impulse has to use an alternate path around the ventricle. The reason for the blockage is usually unknown. This slight delay in the depolarization results in the widening of the QRS. Fortunately it's not normally something to worry about. I know someone who's lived with it for years and hasn't had any complications, just a slightly different looking ECG.
1096425
on a cellular level you have electrolytes such as calcium, potassium, and magnesium that affect muscle contrations.
579789
I think you actually mean tetrodotoxin.  It's a neurotoxin that binds the pores of sodium channels, thus inhibiting potentiaion.  If the toxin is ingested (usually happens by accidental ingestion of certain fish) it can cause paralysis of the individual's diaphragm followed by death due to respiratory failure (i.e. you suffocate).
1062790
Muscle contraction.  Much like when you stretch, then pull all your muscles together.
941883
Wow - tricky question because it goes at so many levels. I'll just keep increasing the detail and you read till you have enough.\n\nOkay - at the most obvious level:\n\nyour brain makes a decision to move (and that one is "why does the body move?") for whatever reason\n\nyour brain passes the message down through parts of the brain the relate to coordination and movement, memory etc and then the message goes down the spinal cord and winds up in the nerves to the sets of muscles. Then the muscles do it.\n\nIt's actually a little more complicated than just "bend my elbow" or "move your big toe" (Kill Bill fans - anyone?) because your brain sends the message to the biceps (in the front of your arm) to bend your elbow and also to the triceps (in the back of your arm) to pay out slack and let the elbow bend. You CAN send messages for both to contract and the elbow to stay rigid (isometric exercises).\n\nCertain parts of our brain are dedicated to coordination of these push/pull effects and allow one group of muscles to relax while the opposing group pull.\n\nThe muscles then pull on their attachment to the bones (the tendons) and then the bones move at the joints. This causes the body parts to move.\n\nOkay - let's learn about the muscle fibres and see how they actually do the movement. There's a good wiki page. Specifically, it has a good close up picture showing the actin and myosin proteins that make up the contractile elements of the muscle fibre.\n\nSLIDING FILAMENT MODEL\n\nIf you look at the close up picture, you will see that each contractile element is made up of long chains of ACTIN and MYOSIN. The Myosin being made up of a long chain of tropomyosin and a little troponin head.\n\nThis is, if you will imagine, a sort of a ratchet mechanism.\n\nIt forms a couple of sliding chains with lots of little troponin heads connecting the tropomyosin to the actin. In the picture of the sarcomere showing actin, myosin and titin you can see that the myosin is sort of double ended and pulls on two sets of actin.\n\nWhen stimulated by the nerve impulse (we'll get to that later) the myosin troponin heads waggle and "pull" the actin in and shorten the whole apparatus (the sarcomere).\n\nIn the normal resting state - the actin and the troponin heads are not actually bound together. It requires the presence of molecules of calcium to cause a change in the shape of the myosin head. This change in shape causes it to bind the actin filament and then "bend" thus pulling the actin and the myosin along each other. Remember that there are millions of the little blighters all binding at slightly different spots and all pulling along. The more of these you activate (again, controlled by the nerves) the more bind and pull you have and the stronger the contraction of your muscle.\n\nOkay - so how do you trigger it and where does the calcium come in? The calcium is stored in the muscle fibre in a spot called the "sarcoplasmic reticulum". These are arrange at each end of the sarcomere in conjunction with the transverse tubules ("T-Tubules") which penetrate the spaces between sarcomeres and allow electrical signal to flow rapidly all over the muscle fibre.\n\nThe trigger mechanism to cause the muscle to move comes from the NERVES. So let's take a look here to find out how this comes about.\n\nThe nerves which carry the signal from the brain, via the spinal cord, to the muscles all have a long process called the AXON which joins on to the muscle at a place called the Neuromuscular junction.\n\nThe axon makes and stores special chemicals called Neurotransmitters (usually Acetylcholine). When they get the signal to say "make this muscle go" they release the neurotransmitters which float across the teenie tiny space and land on the muscle fibre part (called an End Plate) and pass the signal on to the muscle.\n\nThe presence of the neurotransmitter molecules
1128885
Yes mostly. Its kind of an ongoing process. So the bringing in of water will then diffuse which is basically osmosis (water moving from a higher to a lower concentration). Which would take it to the Xylem were transport would occur. Turgidity and flaccidity are states of the cell and are not really processes. In a well watered root there should be no flaccid cells as this only occurs in the absence of water or the presence of excessive solutes. Most cells should be turgid most of the time.
121237
It could be a muscle cramp\nwho knows
1169292
Tough question.  By the time rhabdomyolysis occurs there is a lot of damage to the extremity.  The damaged muscle crumbles up and gets taken into the blood stream.  It circulates around and gets trapped by the kidneys.  If there has been a lot of damaged muscle the circulating remnants can clog up the kidneys and you can get kidney failure.  This can take a while to cure.  I hope things are getting better for your friend.  Did they figure out why she got the rhabdomyolysis in the first place?
311533
I believe the external stimulation would trigger a contraction.  I am basing that on the fact that the muscle fibers are stimulated directly by the electrical current\nfrom the voltage source.\n\nRecruitment of fibers occurs opposite the standard.  Type II, fast twitch fibers, are recruited first and Type I, slow twitch, are second.  Normally the pattern is Type I then Type II when needed.\n\nThe change in charge at the muscle, which is a great conductor of electricity, causes the Ca++ release normally triggered by ACh or another neurotransmitter.\n\nCouldn't find a source to back me up, try an advanced physiology textbook. I did find this link below that has similar information.
878047
Cardiac remodeling in response to arterial hypertension is a result of myocyte hypertrophy, hyperplasia, and hypertrophy of the nonmuscular cells and interstitial cell growth. (1)  \n\nIn short the muscle gets larger and the other structures such as endothelium and collagen divide and grow.  Some of the cardiac muscle eventually will actually die if the condition continues.
773260
I'd go see a doctor.  Could be an involuntary response with muscles tightening up.  Could be infection, yeast or others.
383529
Spasticity is like a “wicked charley horse.”  Brain injury from stroke sometimes causes muscles to involuntarily contract (shorten or flex) when you try to move your limb, creating stiffness and tightness.\n\nWhen a muscle can't complete its full range of motion, the tendons and soft tissue surrounding it can become tight.  This makes stretching the muscle much more difficult.   If left untreated, the muscle can freeze permanently into an abnormal and often painful position.Spasticity in the leg may cause a stiff knee, pointed foot and curling of the toes\nyou should go to the doctor and get their opinion.. check web md. with your symptoms too..