what you know about lsmg disease?
350093
I have never heard of it.  What kind of disease is it?  Is Ismg an acronym?
87673
MS is diagnosed with protein electrophoresis of lumbar punction spinal fluid - it yields stripes caused by monoclonal antibodies. It is an autoimmune disease, treated with immunosuppression, however slowly progressive in most cases. \n\nLyme disease is a bacterial infection by Borrelia burgdorferi. The diagnose is established after antibody detection tests are positive in blood / synovial fluid / spinal fluid according to the presentation. The treatment is generally easier in bacterial infections (antibiotics), however if left untreated for a long time, the neurological sequels of lyme disease can be unreparable. \n\nGreets
521528
Look, my cousin almost died several years ago of this disease.  Yes, to those who live with it, it IS a disease.  I thought she had it under control until my aunt just recently told me that she checked herself into a rehab in California for it.\nTalk to their parents.  Tell them that it should be taken seriously.  If they won't act on it, go to your guidance counselor.  If your counselor thinks it's a serious enough issue, he or she will go to their parents.\nIf worse comes to worst, they get checked into some facility to be evaluated and/or treated.\nUnfortunately, it's not your problem, so there's not really anything you can do about it aside from being supportive.  It sucks, but it's the truth.\nIf you open your mouth, be prepared to lose your friends.  Best case scenario:  they'll thank you for it later.
1049943
Systemic lupus erythematosus (SLE or lupus) is a chronic, potentially debilitating or fatal autoimmune disease in which the immune system attacks the body’s cells and tissue, resulting in inflammation and tissue damage. SLE can affect any part of the body, but often harms the heart, joints (rheumatological), skin, lungs, blood vessels and brain/nervous system. Lupus is treatable, mainly with immunosuppression, though there is currently no cure for it.\n\nThe standard treatment, for decades, has been a limited group of drugs (primarily corticosteroids and chemotherapy drugs). Research into more modern treatments has recently begun and is accelerated by genetic discoveries, especially mapping of the human genome. SLE is known as "the great imitator", as its symptoms often mimic other illnesses and because they come and go unpredictably. Diagnosis can be elusive, with patients sometimes suffering unexplained symptoms and untreated SLE for years. Increased awareness and education about lupus since the 1960s has helped many more patients get an accurate diagnosis and made it possible to estimate the number of people with lupus. Lupus was previously believed to be a rare disease. In the United States alone, an estimated 270,000 to 1.5 million people have lupus, making it more common than cystic fibrosis or cerebral palsy. The disease primarily affects young women, although men can be diagnosed with lupus. World-wide, a conservative estimate states that over 5 million people have lupus.\n\nSLE was called lupus (Latin for "wolf"), perhaps due to a crude similarity between the facial rash that some lupus patients develop, and a wolf's face, although various explanations have been proposed.\n\nI hope this helps and good luck!! you're in my prayers
481274
Do a Yahoo search on Lou Gherig disease.
1064717
The cause of Loug Gherig's disease, or ALS (amyotrophic latteral sclerosis), is still unknown.\nOnly 5-10 percent of ALS cases are inherited. \nALS scares the hell out of me, because I watched my mother die an agonizing death. I have no idea what variation of ALS she had, but I would rather be shot than die like she did.
393286
I think you would have an idea that GBS is actually the body attacking its own nervous system. Therefore it is autoimmune in nature (polyradiculoneuropathy). \n\nGBS manifests as rapidly evolving areflexic motor paralysis with or without sensory disturbance. The usual pattern is an ascending paralysis that may be first noticed as rubbery legs. Weakness typically evolves over hours to a few days and is frequently accompanied by tingling dysesthesias in the extremities. The legs are usually more affected than the arms, and facial diparesis is present in 50% of affected individuals. The lower cranial nerves are also frequently involved, causing bulbar weakness and difficulty with handling secretions and maintaining an airway. Most patients require hospitalization, and almost 30% require ventilatory assistance at some time during the illness. Fever and constitutional symptoms are absent at the onset, and, if present, cast doubt on the diagnosis. Deep tendon reflexes usually disappear within the first few days of onset. Cutaneous sensory deficits, e.g., loss of pain and temperature sensation, are usually relatively mild, but functions subserved by large sensory fibers, such as deep tendon reflexes and proprioception, are more severely affected. Bladder dysfunction may occur in severe cases but is usually transient. If bladder dysfunction is a prominent feature and comes early in the course, possibilities other than GBS should be considered, particularly spinal cord disease. Once clinical worsening stops and the patient reaches a plateau, further progression is unlikely.\n\nIn severe cases of GBS requiring critical care management, autonomic involvement is common. Usual features are loss of vasomotor control with wide fluctuation in blood pressure, postural hypotension, and cardiac dysrhythmias. These features require close monitoring and management and can be fatal. Pain is another common feature of GBS; several types are encountered. Most common is deep aching pain in weakened muscles, which patients liken to having overexercised the previous day. Other pains in GBS include back pain involving the entire spine and sometimes dysesthetic pain in the extremities as a manifestation of sensory nerve fiber involvement.
894027
I have had this disease for over 30 years... it was a gift from my mom who has had it for 65+ years.  The only complication that i recently have is weight gain.(due to taking insulin which is a protein for so long)  This is not the worse disease to have.  When I was first diagnosed the only sugar free item was TAB soda..... so i did stand out alot being "different"\nPlease go to the doctors with your grand child and learn as much as possible.  If she eats 5 small meals a day... tests her blood 4 or more times, exercises, visits her dr. every 3 months.... she will live a long healthy life.  Make sure she stays away from splenda items... this is a sugar.  Eating right and exercising is not a curse... get the whole family to eat the diabetic diet.....and play together..... let her teach you how to test the blood...have her test your blood.. see who has the best #\nThere are support groups for parents and children..... talk to her dr about where and when..... so she won't feel so "different" \nYou are a great grand parent.  I understand your worry... the main thing is to accept her.... go out for a s/f ice cream one night and talk to her about things... but keep an eye on her blood sugar levels... that is the most important...... You will be fine....
1066729
specialists are trying to cure musclular dystrophy. there might be the chance of healing it with the help of molecular biology and stem cell research. but so far there's no date set where it can be cured. they are trying but nobody knows when they figure it out, could be tomorrow, in 10 years or never.\nhuman genetics just involve soo much that stuff like that is unpredictable, unfortunately.
224693
I don't know if there is a cure but there are treatments this site had a buch of info\nallrefer.com   -  von gierke disease\nalternitive name for diseasa is  - Type 1 Glycogen Storage Disease\nalso check webmd site
500730
Did you mean Multiple Sclerosis?\nNo cure yet...\n\nFYI : Syrosis is related to the liver. (Too much drinking. Alcohol/beer etc...).
208526
Most of the information in here is correct. Please do not self diagnose yourself with this disease however; it will make it hard to get an offical diagnosis. The only way to have it offically diagnosed is via Endoscopy with Duodenal Biopsy to confirm via villi. There are lab titers that can be drawn such as, TTG, IGA, IGG and Serum IGA to determine if you are in danger of having this disease. The TTG is the most common indicator of the disease. The IGG can be falsely positive in up to 3/4 of the population and even higher in other races. Prometheus Labs out of CA does a celiac panel that is pretty popular amongst providers; but can be spendy for the patient. Prometheus will also do a celiac gene test, to see if the pt has the gene present to have the disease. The Gliadin antibodies are the strongest lab markers today for tracing the possibility of having the disease. Also, Celiac Disease is also VERY common in pt's with Down Syndrome.
372868
****Go to the website, there's alot more info. there. Good luck with you're project.\n\n\nIntroduction\n\nSmith-Magenis syndrome (SMS) is a distinct and clinically recognizable genetic disorder characterized by a specific pattern of physical, behavioral, and developmental features. SMS, which was first described in the early 1980's by Ann C.M. Smith, MA (a genetic counselor) and Ellen Magenis, MD (a cutogeneticist), is the result of a deletion of chromosome 17 (17p11.2). The chromosomal deletion occurs from a spontaneous genetic change (mutation) that happens for unknown reasons, therefore, it is not a familial disorder. SMS is considered a rare disorder and is estimated to occur in 1 out of every 25,000 live births. Currently there are over 100 cases reported, however, it is believed that SMS is widely underdiagnosed because clinical features may be subtle. It is expected that with increased awareness, the number of those identified as having SMS will increase.
888935
Def: chronic disease of the central nervous system, in which there is destruction of myelin (covering of the nerves) and nerves axon within several regions of the brain and spinal cord at different times ( episodic and progressive)\nSigns & Symptoms:\nParesthesia (numbness & tiggling) in different places\nDouble vision, blurred vision, dimness vision and sometimes partial or total blidness\nweakness in the extremities\nMuscle paralysis\ntremors\nGait disturbance (walking weird)\nSpasticity \nDifficulty controlling your bladder\nConstipation (a common problem)\nVertigo\nEmotional Disturbance (anger, irrational behavior, deprssion)\nPatient seem to get tired all the time.\nCheck everything you can, read, look it up on the web if you can, prepare yourself, take everything in, and always question the Dr. i know the know it ALL, but at times they can MISDIAGNOSE\ngood luck, & good health to you
202574
Ok... what do you want know in particular?\nI hope you don't want to know every disease!!\nToo many to discuss on this short space.\nAIDS/HIV@ \nChlamydia@ \nConsumer Products@ \nGenital Warts@ \nGonorrhea@ \nHepatitis@ \nHerpes@ \nLymphogranuloma Venereum (LGV)@ \nResources for Teens@ \nSafer Sex@ \nSyphilis@ \nTrichomoniasis@  \nStill,\nAIDS/HIV@ \nChlamydia@ \nConsumer Products@ \nGenital Warts@ \nGonorrhea@ \nHepatitis@ \n\nHerpes@ \nLymphogranuloma Venereum (LGV)@ \nResources for Teens@ \nSafer Sex@ \nSyphilis@ \nTrichomoniasis@  \netc. etc....
279769
Hummm are you sure you have the disease spelled correctly?  I am not finding anything on my net searches and I haven't heard of it as a trained Medical Assistant.  I'm sorry :/  GL!  Maybe someone else here give you a better answer!
254453
Wernig Hoffman Disease, also termed hereditary spinal muscular atrophy, is a disease of infants and young children that resembles amyotrophic lateral sclerosis. (spinal disease)Pathological changes are confined to the anterior horn cells of the cord and the cranial nerve motor nuclei. The disease affects all muscles to a varying degree. \n\nInfants with Werdnig Hoffmann disease may lack head control, may be unable to roll over or support their weight, and tend to lie relatively still, with little or no movement (flaccid paralysis). In addition, they may develop difficulties sucking, swallowing, and breathing; have an increased susceptibility to respiratory infections; or develop other complications that may lead to potentially life-threatening abnormalities within the first months or years of life. Intelligence is normal. Most affected children die before 2 years of age but survival may be dependent on the degree of respiratory function. \n\nFor infants who appear to have normal development for several months prior to the onset of muscle weakness, the disorder may tend to have a more slowly progressive course. Muscles of the pelvic, trunk, and shoulder areas may initially appear to be disproportionately affected. All muscles, with the exception of the eyes, are affected