differential diagnosis of desaturation during inhalational induction espcially with assisted ventilation?
350569
A Doctor of Medicine is your best bet.
995876
The answer is D. oxygen\n\n"Oxygen is given with a small amount of continuous positive airway pressure ("CPAP"), and intravenous fluids are administered to stabilize the blood sugar, blood salts, and blood pressure. If the baby's condition worsens, an endotracheal tube (breathing tube) is inserted into the trachea and intermittent breaths are given by a mechanical device."
897315
For a very short time, yes. People tend to forget about what's called "dead air spaces". Yes, you're exhaling some co2, but a good deal of that exhalation is still o2 ( it's not all used in a breath) and even more so in dead air spaces. These are areas in your body from the top of your lungs to the tip of your lips, that don't do any gas transfer to or from the blood stream. It's still normal, oxygen rich air in there.\n I also disagree with what one person has stated here, that artificial respiration is only a method to get the lungs moving. \n It IS in fact a method of oxygenating the blood stream and keeping a victim viable in the absence of their breathing on their own.
249172
i think that is oxygen depravation
96888
I need to know what medical history, Medications , allergies. What were you doing at the time, did you experience Chest pn with this, or shortness of breath. did you seel medical attention, did you have flu like symptoms.\n\nI am a paramedic , not a doctor but I may be able to answer your questions.
307122
Combivent is definitely NOT an expectorant. It is a combination of albuterol ( a beta 2 specific adrenergic bronchodilator) and atrovent ( an anticholinergic bronchodilator). I would reccommend going back to your Doc & talking to him. Be careful of the advice you get on here! LOL
978419
alkalosis.  Hyperventilation is breathing off the acid.
531899
Anxiety, fever, stimulant drugs, pain, sepsis(a toxic condition resulting from infection), hypobarism (high altitude), and any other condition in which ventilatory elimination of C02 exceeds it's production. Basically you are hyperventilating! These are all causes of respiratory alkalosis, so if you can manage to avoid all of these you should be okay.
5048
PNEUMONOULTRAMICROSCOPICSILICOVOLCANOCONIOSIS- LUNG DISEASE CAUSED BY BREATHI IN CERTAIN PARTICLES
1128009
I served in the USAF from 1994-1998. I wasn't a pilot, I was a poor little enlisted  scrub. But, I know that that the AF suggests weight training, with cardio, endurance activities. Now, I think your question revelovles around the positive pressure breathing technique? That would be rapid inhalation, followed by rapid exhalation while tensing all the major muscle groups. Now, most pilots do the 1-2 hour of pre-breathing of pure 100% oxygen. Personally, I think you should look into yoga.
392665
acute respiratory distress syndrome is basicly lung failure... and very deadly. The clinical findings to diagnose ards, is po2/fio2 of less than 200 (which in laymans terms inablity to exchange gases in the lungs), double pnemonia (xray will be completly whited out. This is caused by many things, pnemonia gone bad, chf, aspiration of stomach contents, septic shock, trauma to the body,high % of o2 given for too long of a time and so on... by the way it is either adult respiratory distress syndrome or respiratory distress syndrom (the name for the pediactric patients....... \n\nthe mortality rate is very high.. however recent studies have shown with low tidal volumes on the ventilator and high rates.... limiting pressures in the lungs for a certain period of time does lower the mortality rate... at the last hospital i worked at i managed ventilators on ards patients... we used ards.net for our patients... it lowered the mortality rate to 28% in one year in our hospital... thats down from 60%\n\nthere are also other methods of treating it.. such as using flolan or nitric oxide... both help lower the co2 in the body and help with oxgenating the body with out using high levels of o2 (which high levels causes further damage to the lung tissue)\n\nI could go on all night about ards......
535994
Ineffective airway clearance, evidenced by rales,/rhonchi. Interventions can included airway suction, percussion and postural drainage, breathing treatments to stimulate cough.\n\nIneffective gas exchange, evidenced by increased resp rate, and decreased oxygen saturation. Interventions can include cough and deep breathe to open airways, use of the incentive spirometer, supplemental oxygen. \n \nThese are just off the top of my head, but I hope they help.
323137
Increased Bronchovascular Markings\n\nThis always means either infection in respiratory passages or fluid overload. Infection may be acute or chronic. If radiologist reports increased bronchovascular markings but clinical situation is not supporting any thing it can be ignored and film should be taken as normal.\n\nIn cardiac failure cases so called reversal of pattern of markings occur. There is increase in markings in midzones due to fluid overload. Actually this is not reversal. This is only increase in midzones.\n\nIn chronic bronchitis, markings are increased. In emphysema, these are decreased in hyperinflated areas. In collapse of the lungs beyond the line-demarcating lung, these must be absent.
1080581
okay.. here goes\nI am a Respiratory therapist.. have been for 6 years and just in the past 6 years plus when i was in respiratory there has been many changes in clincial practice. \nThere are now different forms of ventliation from invasive and non-invasive. The invasive forms have went from just setting a tidal volume which while i was in respiratory school we figured tidal volume 10ml/kg to no more than 8ml/kg. They have found that the lower volumes have a lower chance of trauma to the lungs. Another change is the different modes on ventilators. Before there was only one pressure control ventilation setting. You set a pressure limit, and a rate and that patient go that no matter what... you would in turn have many patients unable to tolerate this mod of ventilation without being heavily sedated and even paralysed the whole time of treatment. Now we have modes like pressure limited volume guarenteed settings, bilevel is teh one that i am most familar with its the one that we used at my hospital, it is on the puritan bennet 840 ventilator it actually has a pressure support to be set, and a rate.. made up much like simv in voulme control mode. The patient is able to breathe in sync with the ventilator, instead of fighting the breaths. There are many other modes that the 840 has such as the volume support mode, basicly you set a volume and let the patient breathe and no matter what they get at least that volume with each breath.. it is a mode you only use with patients who cannot be weaned using pressure support modes. \n\nAnother big change has been the use of bipap/cpap it is now the first step to keep a patient from being intubated, and with the new machines coming out into market, all hospital are well equiped with them, no longer using the downs flow meter cpap. \n\nWe also have had an increase in using lower tidal volumes in all patients, espeacialy ones with ARDS net.. great choice of ventilation for acute lung injury and ards.. we used it as a protocol whree i worked at and actually reduced the mortality rate from 70% to only 30% with our ards patients. Great leaps and bounds!!!!\n\nWhich goes to my next change in clinical practice, Respiratory Therapist Driven protocols. The doctors only see the patient a couple times a day if that much. We see them ongoing throughout the whole shift, meaning we can make small adjustments as needed for the patient, catching just the right window to wean patients, etc.  THere is a huge reduction on days on a vent for patient using these protocols. Also reducing  mortality, and ventilator associated pnemonias.\n\nThe changes in the past few years have been huge, I remember setting up a vent in a mri unit, and the lady I was working with (a respiratory dinasour) telling me that the settings on that vent were all they had just about 10 years ago. Meaning a volume, pressure, and rate set... no inbetweens to be more comfortable for the patient, and theraputic.
457919
90-100% sat is normal, if you used a saline soulution, did they call it a challange test? if they did they wre checking to se how reactive your airways were, the gold standard in diagnosing astham...\nyour doctor will determine the results and give you a regimin to help your breathing
294687
I you mean Impaired Gas Exchange here are some answers and I will group them according to Independent and Collaborative Interventions:\nIndependent:\n> Assess respirations, noting quality, rate, depth, pattern and breathing effort.\n> Assess lung sounds and note changes\n> Assess for changes in behavior and orientation\n> Monitor activity level and provide rest period in between activities.\n\nCollaborative:\n> Monitor chest radiograph reports\n> Monitor closely ABG's, lab reports and note changes\n> Assess for s/sx of increasing hypoxia & hypercapnia\n> Assess PFT (pulmonary function test)\n> Assess hydration and electrolytes \n> Monitor O2 sat. with a pulse oximeter\n> Administer medication as prescribed and observe for therapeutic effect
281675
breath deeply 2-5 times