Ejection fraction increased from 41% to 52%. Aortic root size increased from 35 to 37mm. Please explain these test results.
208188
any increase in ef is usually good.It means your heart is pumping more efficiently. The main thing you should worry about is why(leaky heart valve, virus, alcohol consumption) your heart was dilated in the first place and try to fix it. The aortic size is harder to evaluate the place I work at now that would be large but other places use different measurements and that could be normal. Go to www.seemyheart.org its a web site by the American society of Echocardiography for patients to understand there results
127582
your O2 saturation rate is the % of oxygenated blood running through your body. Normal is around 98% to 99%
327351
This has been going on for a year so you are really flirting with disaster.  37 is an obscene level for the lower reading on your bp.  The pain is most likely due to blood not flowing through your system correctly.  Eyesight failing is another symptom as well as dizziness.  These can all be corrected-- usually with an angioplasty which is usually an outpatient procedure.  If it takes more, I hate to say it but you only have yourself to blame for letting it go this long.  Go to the emergency room now, especially if the pain on the right side is an aching pain as opposed to a sharp pain.  Good Luck.
622484
Treadmill stress electrocardiography is employed widely to detect underlying cardiovascular disease in asymptomatic persons. To determine the frequency and significance of an abnormal S-T segment response to exercise in young athletes with increased left ventricular (LV) mass induced by physical conditioning, 75 male isometrically trained athletes without evidence of heart disease (mean age 21 years) and 48 nonathletic young normal subjects were studied by M-mode echocardiography and exercise electrocardiography. Ventricular septal and posterior free wall thicknesses and calculated LV mass were significantly greater in athletes than in control subjects. An abnormal S-T segment response to exercise was present in 7 (9%) of the 75 athletes, all of whom had a LV mass greater than 275 g and in 5 the LV mass was above the 95th percentile of that of the control subjects. An abnormal treadmill exercise test result also was present in 3 (6%) of 48 control subjects. Seven of the 10 subjects with abnormal exercise test results had radionuclide angiograms at rest and with exercise, each of which was normal. It is concluded, therefore, that (1) abnormal exercise test results occur commonly in both athletes and non-athletes; (2) almost 10% of isometrically trained athletes have "false-positive" exercise electrocardiograms, suggesting that this test has major limitations in screening for underlying cardiovascular disease in athletes; and (3) in athletes, a relation was present between "physiologically" increased LV mass and false-positive exercise test results.
54677
The risks really aren't that bad, as long as you continue to have your yearly check-up. They increase after the age of thirty IF you are a smoker. There's an increase in the risk of strokes and blod clots I think. Hope this helps.
559654
that the heart rate goes up fast exercise then at rest comes back quickly to normal is ok.\nthe important thing to look for (after you said the coronaries were ok) is systolic dysfunction,\nthat can be cleared by doing an exercise echocardiogram.\n\nof course age has something to do with it too.
375764
This something would be aortic artery pressue (reflecting systemic circulation) is equal to pulmonary artery pressure and this would happen in pulmonary hypertension. Yet, in pulmonary hypertension, closure of the pulmonic valve would be rather delayed than increase in intensity.
406583
I don't think there is a known association in between dengue fever and aortic dissection, its mainly a risk of hemorrhagic death, if you are not a local of these areas then avoid going all these endemic places, good luck
521969
a normal pulse rate is 60-100. You may need a physical if your HR and BP are up and has never done this before. You may just be out of shape or pushing yourself more, but it may be something more serious like heart rhythm prob or defects, anemia, etc.
405413
If the thickness of the vessel wall is the same in both as well, then theorhetically you would get slightly  higher flow from the one 4mm vessel than the 2-2mm vessels since the one of the wall thicknesses is eliminated.
338193
Left ventricular contraction septal hypokinesia. This isn't a disease. That's radiologist speak for what he saw, which is the ventricular septum (lower inner wall of the heart) doesn't appear to be contracting as hard as normal. This is probably the result of a past infarct, which she may not have even noticed. This is just one part of the ventrical. As long as EF (ejection fraction) is still good, this isn't a big deal, in and of itself. What is of concern is the degree of occlusion of the coronary arteries, her blood pressure, and her lipid profile, as these will determine the likelihood of another infarct (heart attack). This area of septum will not recover, but unless it a very large area, the remaining heart muscle will easily make up for the loss.
396249
The pages listed under this information section  contain summaries of relevant topics written by many different experts in the field for the Montgomery Heart Foundation for Cardiomyopathy. The information contained in these summaries was originally collected in 1997 to put together a printed brochure for patients, families and health care professionals.  Although these summaries are now 6 or 7 years old, they have been re-reviewed and we believe  contain basic and general information that is still helpful to patients and families today.\n\n \nWhile the entity of the "athlete's heart" has been recognized for over 100 years, only in the last two decades has the application of echocardiography and other noninvasive imagine techniques permitted definition with some precision of the alterations in cardiac dimensions associated with athletic conditioning.  Echocardiography has demonstrated that long-term athletic training leads to an increase in left ventricular (LV) mass due to increases in LV diastolic cavity dimension, wall thickness, or both.  These changes in cardiac morphology are relatively mild in absolute terms, and the differences between athlete and non-athlete populations are statistically significant but generally small.  Furthermore, cardiac alterations associated with training differ somewhat depending on the particular sporting discipline in which the individual athlete participates.  In particular, the changes in LV wall thickness, cavity dimensions, or both associated with long-term athletic training may be more striking in certain sports such as distance running, swimming, cycling and rowing/canoeing.  It is in athletes raining in such sports that the differential diagnosis is more likely to be raised.\n\nDifferential Diagnosis\n \nDilated Cardiomyopathy\nIn an important minority of athletes, the increase in LV end-diastolic cavity dimension that occurs with training overlaps that which is characteristic of certain pathologic entities.  While LV cavity dimension is athletes is usually in the range of 53 to 58 mm, in some individuals it may extend into what is regarded as the pathological range of >60 mm (up to 70 mm), and thereby resemble dilated cardiomyopathy.  However, the absence of LV systolic dysfunction is usually sufficient to distinguish such physiologic ventricular enlargement induced by training from dilated cardiomyopathy. \n\nArrhythmogenic Right Ventricular Dysplasia (ARVD)\nBecause highly trained athletes may demonstrate right ventricular enlargement and a variety of depolarization, repolarization and conduction abnormalities on the ECG, the differential diagnosis between athlete's heart and ARVD may arise.  Identification of ARVD by echocardiography may be exceedingly difficult because of technical limitations in imaging right heart morphology (and assessing right ventricular function), and also because the spectrum of disease is broad.  Demonstration of right ventricular segmental or global dysfunction or substantial cavity enlargement supports the diagnosis.  Magnetic resonance imaging, however, affords a more reliable noninvasive diagnosis of this condition.  In ARVD, ECG's frequently show T wave inversion in V1-V3.\n\nHypertrophic Cardiomyopathy\nThe dilemma of distinguishing clinically between athlete's heart and structural heart disease most frequently arises with respect to hypetrophic cardiomyopathy (HCM).  While at present there is no single approach that will definitively resolve this question in all such athletes, several strategies are described here that alone or in combination offer a large measure of clarification in most instances for this often compelling differential diagnosis.  The definition of HCM employed here is that of a patient (or athlete) with evidence of a hypertrophied and nondilated LV in the absence of another cardiac of systemic disease that could itself produce hypetrophy of the magnitude present in that individual.\n\nWall Thickness\nIn
21501
I've never heard of this, so first of all my thoughts are with your family.  I knew strokes, heart attacks and those types of things didn't only happen to "older" people anymore, but that they were also happening to people as young as their 20's.  I had absolutely no idea, I am going to try to look this up tho, as I have two young children.  Were there any symptoms, something long coming that didn't get noticed because it was also considered a normal thing?  I hope he stays strong and has a great turn around back to good health.
649160
TMT is nothing but  taking your ECG while you are walking on a TREAD MILL at various speeds and resistant. If Echo has shown valve expansion TMT will help you to know the exact condition of your heart and the chances of attack during stress full situations. Better than MD consult A CARDIAC SURGEON. GOOD LUCK
333173
A positive tilt table test indicates (as you probably know) that your heart is beating inappropriately to maintain appropriate blood pressure.  Your blood pressure drops and you faint.  Normally your body would enlist a number of mechanisms to prevent fainting...the arteries should constrict, the heart should beat faster to compensate.  Something, however is going awry.  It might be that your heart is beating waaay too fast - hence POTS.  But that's far from a forgone conclusion.  And NMS is mostly a diagnosis of exclusion...if everything else checks out, then we are left with NMS.\n\nThe cardiologists role is to tease all of this out.  What is happening exactly with your heart when this phenomenon occurs?  They also will want to make sure the structures are working like they are supposed to...normal contractions, normal valvular function, etc.  They also can be helpful in monitoring your heart over a period of a couple days to determine if your heart goes too fast, too slow, or what.\n\nAs far as treatments are concerned, you first need a diagnosis in order to discuss treatment.  Try not to worry.  The plan is to get more information and get to the bottom of all of this.  Once there is more information then it will direct treatment.\n\nGood luck.
218494
Sorry - height has not been demonstrated to be associated with an increased risk of myocardial infarction (or heart attacks).  The Framingham Heart Study is the seminal study from where we get most of our current knowledge about risk factors for heart disease, and height was NOT a significant risk factor.  But some particularly tall individuals may have Marfan syndrome which is associated, as someone else mentioned, with ascending aortic aneurysms.  In Marfan patients, these aneurysms are at high risk of dissecting (which is what happened to John Ritter).  Dissections feel and present like heart attacks in many ways and are often mislabelled as "heart attacks".  Patients will typically complain of severe chest pain.  Dissection can lead to heart attack, tamponade, stroke, rupture, and death.  I think this is probably what you are talking about.
320356
It's the complete opposite. The stronger the heart the lower your BP and resting heart rate should be. When I used to run 30+ miles a week, my resting heart rate would be around 46 and the BP would be 115/70... You sound like you are doing the things that you need to do. Have you minimized salts from your diet as well?