sative for food poisoning induced by *B*. *cereus*, namely the haemolysin BL (Hbl) and the non-haemolytic enterotoxin (Nhe) complexes which causes diarrohea. Hbl consists of the protein components B, L1 and L2 \[[@pone.0214536.ref002], [@pone.0214536.ref003]\], encoded by *hblA* (B), *hblC* (L~2~) and *hblD* (L~1~) \[[@pone.0214536.ref002]\]. Nhe is composed of the protein constituents NheA, NheB and NheC encoded by *nheA*, *nheB* and *nheC* \[[@pone.0214536.ref004]\]. For both enterotoxin complexes, the combination of their respective components is necessary in order to develop their enterotoxigenic potential \[[@pone.0214536.ref005], [@pone.0214536.ref006]\].

The probiotic strain *B*. *toyonensis* BCT-7112^T^ (NCIMB 14858^T^) is the active ingredient of the feed additive preparation Toyocerin (product of Rubinum S.A.), designated as the type strain \[[@pone.0214536.ref007], [@pone.0214536.ref008]\]. The genome of *B*. *toyonensis* BCT-7112^T^ (NCBI Acc.-No.: CP006863.1) shows relevant differences from the genome of *B*. *cereus* food-poisoning strains regarding the presence of genes considered to be the causative agents of gastrointestinal diseases, e.g., haemolysin BL (Hbl), non-haemolytic enterotoxin (Nhe), cytotoxin K (CytK) and cereulide \[[@pone.0214536.ref009]\]. The Nhe (non-haemolytic enterotoxin) and Hbl (haemolysin BL) operons present in the genome of *B*. *toyonensis* may be nonfunctional, since some of their toxin components show differences in the corresponding amino acid sequences when compared to those present in *B*. *cereus* food-poisoning strains. Furthermore, only three of the seven genes comprising the operon encoding cereulide synthetase are present in the genome of *B*. *toyonensis*. Therefore, the cereulide synthetase is not expected to be functional. Additionally, the *cytK* gene is absent in the genome of *B*. *toyonensis* \[[@pone.0214536.ref007]\]. Although the presence of the Nhe and Hbl operons is not synonymous with their expression or functional properties of the gene products *p