he severe range (also noted in [@R24]).

Scores for labile crying did not correlate across patients with impulsiveness (attention and planning facets: ρ = 0.12, *p* = 0.60), depression (ρ = 0.24, *p* = 0.28), or any memory score in which patients had shown impaired performance as compared with HCs (all *p*s, *p*~corr~ ≥0.240), and were not associated with any demographic or clinical variables examined (all *p*s, *p*~corr~ ≥0.440).

### Self-report: patients with vs patients without pathologic tearfulness and HCs {#s2-1-2}

In a research-oriented clinical interview, 19 of 38 patients were identified as presenting with pathologic tearfulness. In particular, they reported being moved to tears easily by relatively minor stimuli in a manner at odds with their premorbid state ([table 1](#T1){ref-type="table"}). The other 19 reported never having experienced such instances. No patient reported experiencing episodes of labile laughter.

###### 

Patients\' self-reports

![](NEUROLOGY2019010967TT1)

The majority of patients and their family members reported specific triggers of such reactions, including sad stories on the news and witnessing other people crying ([table 2](#T2){ref-type="table"}).

###### 

Triggers of pathologic tearfulness as identified by patients and their family members

![](NEUROLOGY2019010967TT2)

Patients with pathologic tearfulness did not differ from the rest of the participants in any demographic or clinical details or in episodic memory impairment, depression, or impulsiveness. Moreover, they did not differ from the rest of the patients or HCs in premorbid intelligence, vocabulary, semantic knowledge, visuomotor function or executive function, anxiety, empathy, or irritability. Among all the tests and questionnaires administered, the only one in which they scored differently from both the rest of the patients and HCs was CNS-LS ([table 3](#T3){ref-type="table"}).

###### 

Neuropsychological, clinical, and demographic profile of patients with vs patients without pathologic tearfulness vs 